Mice lacking neutrophil elastase reveal impaired host defense against gram negative bacterial sepsis

542 indexed citations

Abstract

loading...

About

This paper, published in 1998, received 542 indexed citations. Written by Abderrazzaq Belaaouaj, Ronald McCarthy, Mary Baumann, Zhimin Gao, Timothy J. Ley, Soman N. Abraham and Steven D. Shapiro covering the research area of Immunology and Allergy, Immunology and Cancer Research. It is primarily cited by scholars working on Immunology (302 citations), Molecular Biology (133 citations) and Cancer Research (115 citations). Published in Nature Medicine.

Countries where authors are citing Mice lacking neutrophil elastase reveal impaired host defense against gram negative bacterial sepsis

Specialization
Citations

This map shows the geographic impact of Mice lacking neutrophil elastase reveal impaired host defense against gram negative bacterial sepsis. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mice lacking neutrophil elastase reveal impaired host defense against gram negative bacterial sepsis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mice lacking neutrophil elastase reveal impaired host defense against gram negative bacterial sepsis more than expected).

Fields of papers citing Mice lacking neutrophil elastase reveal impaired host defense against gram negative bacterial sepsis

Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of Mice lacking neutrophil elastase reveal impaired host defense against gram negative bacterial sepsis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the Mice lacking neutrophil elastase reveal impaired host defense against gram negative bacterial sepsis.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

This paper is also available at doi.org/10.1038/nm0598-615.

Explore hit-papers with similar magnitude of impact

Breakdown of academic impact, for the paper Functional specialization of gut CD103 + dendritic cells in the regulation of tissue-selective T cell homing Breakdown of academic impact, for the paper Conserved Fungal LysM Effector Ecp6 Prevents Chitin-Triggered Immunity in Plants Breakdown of academic impact, for the paper Distinct subsets of unmyelinated primary sensory fibers mediate behavioral responses to noxious thermal and mechanical stimuli Breakdown of academic impact, for the paper The roles of hydraulic and carbon stress in a widespread climate-induced forest die-off Breakdown of academic impact, for the paper Antitumor Activity of PR-171, a Novel Irreversible Inhibitor of the Proteasome Breakdown of academic impact, for the paper Accurate SHAPE-directed RNA structure determination Breakdown of academic impact, for the paper Loss of a Callose Synthase Results in Salicylic Acid-Dependent Disease Resistance Breakdown of academic impact, for the paper A Comparison of PAM50 Intrinsic Subtyping with Immunohistochemistry and Clinical Prognostic Factors in Tamoxifen-Treated Estrogen Receptor–Positive Breast Cancer Breakdown of academic impact, for the paper Differentiation of muscle, fat, cartilage, and bone from progenitor cells present in a bone-derived clonal cell population: effect of dexamethasone. Breakdown of academic impact, for the paper The Novel Histologic International Association for the Study of Lung Cancer/American Thoracic Society/European Respiratory Society Classification System of Lung Adenocarcinoma Is a Stage-Independent Predictor of Survival
Rankless by CCL
2026