Fetuin-A acts as an endogenous ligand of TLR4 to promote lipid-induced insulin resistance
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doi.org/10.1038/nm.2851 →Countries where authors are citing Fetuin-A acts as an endogenous ligand of TLR4 to promote lipid-induced insulin resistance
This map shows the geographic impact of Fetuin-A acts as an endogenous ligand of TLR4 to promote lipid-induced insulin resistance. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fetuin-A acts as an endogenous ligand of TLR4 to promote lipid-induced insulin resistance with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fetuin-A acts as an endogenous ligand of TLR4 to promote lipid-induced insulin resistance more than expected).
Fields of papers citing Fetuin-A acts as an endogenous ligand of TLR4 to promote lipid-induced insulin resistance
This network shows the impact of Fetuin-A acts as an endogenous ligand of TLR4 to promote lipid-induced insulin resistance. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the Fetuin-A acts as an endogenous ligand of TLR4 to promote lipid-induced insulin resistance.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
This paper is also available at doi.org/10.1038/nm.2851.