CXCR4-activated astrocyte glutamate release via TNFα: amplification by microglia triggers neurotoxicity
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doi.org/10.1038/89490 →Countries where authors are citing CXCR4-activated astrocyte glutamate release via TNFα: amplification by microglia triggers neurotoxicity
This map shows the geographic impact of CXCR4-activated astrocyte glutamate release via TNFα: amplification by microglia triggers neurotoxicity. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by CXCR4-activated astrocyte glutamate release via TNFα: amplification by microglia triggers neurotoxicity with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites CXCR4-activated astrocyte glutamate release via TNFα: amplification by microglia triggers neurotoxicity more than expected).
Fields of papers citing CXCR4-activated astrocyte glutamate release via TNFα: amplification by microglia triggers neurotoxicity
This network shows the impact of CXCR4-activated astrocyte glutamate release via TNFα: amplification by microglia triggers neurotoxicity. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the CXCR4-activated astrocyte glutamate release via TNFα: amplification by microglia triggers neurotoxicity.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
This paper is also available at doi.org/10.1038/89490.