Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-κb-inducing kinase
- Journal
- Nature Genetics
In The Last Decade
doi.org/10.1038/8780 →Countries where authors are citing Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-κb-inducing kinase
This map shows the geographic impact of Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-κb-inducing kinase. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-κb-inducing kinase with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-κb-inducing kinase more than expected).
Fields of papers citing Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-κb-inducing kinase
This network shows the impact of Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-κb-inducing kinase. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-κb-inducing kinase.
About Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-κb-inducing kinase
This paper, published in 1999, received 379 indexed citations . Written by Reiko Shinkura, Kazuhiro Kitada, Fumihiko Matsuda, Kei Tashiro, Koichi Ikuta, Misao Suzuki, K. Kogishi, Tadao Serikawa and Tasuku Honjo covering the research area of Cancer Research, Immunology and Immunology and Allergy. It is primarily cited by scholars working on Immunology (274 citations), Cancer Research (187 citations) and Molecular Biology (108 citations). Published in Nature Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
This paper is also available at doi.org/10.1038/8780.