Yuanlin Wu

736 total citations
24 papers, 469 citations indexed

About

Yuanlin Wu is a scholar working on Molecular Biology, Cancer Research and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Yuanlin Wu has authored 24 papers receiving a total of 469 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 10 papers in Cancer Research and 7 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Yuanlin Wu's work include Cancer-related molecular mechanisms research (9 papers), RNA modifications and cancer (8 papers) and Ferroptosis and cancer prognosis (4 papers). Yuanlin Wu is often cited by papers focused on Cancer-related molecular mechanisms research (9 papers), RNA modifications and cancer (8 papers) and Ferroptosis and cancer prognosis (4 papers). Yuanlin Wu collaborates with scholars based in China and Hong Kong. Yuanlin Wu's co-authors include Xu Wu, Jing Shen, Yueshui Zhao, Mingxing Li, Zhangang Xiao, Jianhua Yin, Chi Hin Cho, Lingling Zhang, Parham Jabbarzadeh Kaboli and Qijie Zhao and has published in prestigious journals such as PLoS ONE, Current Medicinal Chemistry and Pharmacological Research.

In The Last Decade

Yuanlin Wu

24 papers receiving 464 citations

Peers

Yuanlin Wu
Junya Yan China
Hanlin Mok United States
Wade M. Junker United States
Philip Hicks United States
Fui Teen Chong Singapore
Yuanlin Wu
Citations per year, relative to Yuanlin Wu Yuanlin Wu (= 1×) peers Tianxiao Xu

Countries citing papers authored by Yuanlin Wu

Since Specialization
Citations

This map shows the geographic impact of Yuanlin Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yuanlin Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yuanlin Wu more than expected).

Fields of papers citing papers by Yuanlin Wu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yuanlin Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yuanlin Wu. The network helps show where Yuanlin Wu may publish in the future.

Co-authorship network of co-authors of Yuanlin Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Yuanlin Wu. A scholar is included among the top collaborators of Yuanlin Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yuanlin Wu. Yuanlin Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhang, Lu, et al.. (2025). A method for detecting feeding fish in ponds based on FFishNet-YOLOv8. Computers and Electronics in Agriculture. 230. 109873–109873. 2 indexed citations
2.
Wu, Yuanlin, Jiandong Zhang, Junjun Zhao, & Bin Wang. (2024). Folate-modified liposomes mediate the co-delivery of cisplatin with miR-219a-5p for the targeted treatment of cisplatin-resistant lung cancer. BMC Pulmonary Medicine. 24(1). 159–159. 3 indexed citations
3.
Zhu, Ting, Yuanlin Wu, Jiandong Zhang, et al.. (2024). LINC00665 promotes glycolysis in lung adenocarcinoma cells via the let-7c-5p/HMMR axis. Journal of Bioenergetics and Biomembranes. 56(2). 181–191. 2 indexed citations
4.
Wu, Yuanlin, et al.. (2023). Hsa-miR-195-5p Inhibits Autophagy and Gemcitabine Resistance of Lung Adenocarcinoma Cells via E2F7/CEP55. Biochemical Genetics. 61(4). 1528–1547. 7 indexed citations
5.
Zhu, Ting, Yuanlin Wu, Jiandong Zhang, et al.. (2023). A five-collagen-based risk model in lung adenocarcinoma: prognostic significance and immune landscape. Frontiers in Oncology. 13. 1180723–1180723. 6 indexed citations
6.
Yue, Ming, Ruoqiu Fu, Chen Li, et al.. (2022). The pathogenesis, diagnosis, prevention, and treatment of CAR-T cell therapy-related adverse reactions. Frontiers in Pharmacology. 13. 950923–950923. 35 indexed citations
7.
Fu, Ruoqiu, Tingting Jiang, Dongyu Duan, et al.. (2022). Mechanism of Lethal Skin Toxicities Induced by Epidermal Growth Factor Receptor Inhibitors and Related Treatment Strategies. Frontiers in Oncology. 12. 804212–804212. 26 indexed citations
8.
Wu, Yuanlin, Bin Wang, Haiyong Wang, et al.. (2022). Construction of a prognostic risk assessment model for lung adenocarcinoma based on Integrin β family‐related genes. Journal of Clinical Laboratory Analysis. 36(6). e24419–e24419. 7 indexed citations
9.
Li, Li, Rui Ni, Ziwei Li, et al.. (2022). Insights into Regulatory Factors in Megakaryocyte Development and Function: Basic Mechanisms and Potential Targets. Frontiers in Bioscience-Landmark. 27(11). 313–313. 7 indexed citations
10.
Wu, Guomin, et al.. (2021). Identification and Validation of Immune-Related LncRNA Prognostic Signature for Lung Adenocarcinoma. Frontiers in Genetics. 12. 681277–681277. 23 indexed citations
11.
Wang, Haiyong, Bin Wang, Chu Zhang, et al.. (2020). MiR-29c-3p Suppresses the Migration, Invasion and Cell Cycle in Esophageal Carcinoma via CCNA2/p53 Axis. Frontiers in Bioengineering and Biotechnology. 8. 75–75. 25 indexed citations
12.
Wu, Yuanlin, Jing Li, Parham Jabbarzadeh Kaboli, et al.. (2020). Natural killer cells as a double-edged sword in cancer immunotherapy: A comprehensive review from cytokine therapy to adoptive cell immunotherapy. Pharmacological Research. 155. 104691–104691. 25 indexed citations
13.
Wang, Haiyong, Bin Wang, Chu Zhang, et al.. (2020). The value of CEP55 gene as a diagnostic biomarker and independent prognostic factor in LUAD and LUSC. PLoS ONE. 15(5). e0233283–e0233283. 21 indexed citations
14.
Zhu, Ting, Zhifeng Ma, Haiyong Wang, et al.. (2020). YAP/TAZ affects the development of pulmonary fibrosis by regulating multiple signaling pathways. Molecular and Cellular Biochemistry. 475(1-2). 137–149. 11 indexed citations
15.
Wu, Yuanlin, et al.. (2020). A three-gene signature and clinical outcome in pediatric acute myeloid leukemia. Clinical & Translational Oncology. 23(4). 866–873. 9 indexed citations
16.
Yin, Jianhua, Hanyu Zhang, Xu Wu, et al.. (2020). CD44 inhibition attenuates EGFR signaling and enhances cisplatin sensitivity in human EGFR wild‑type non‑small‑cell lung cancer cells. International Journal of Molecular Medicine. 45(6). 1783–1792. 35 indexed citations
17.
Kaboli, Parham Jabbarzadeh, Lingling Zhang, Shixin Xiang, et al.. (2019). Molecular Markers of Regulatory T Cells in Cancer Immunotherapy with Special Focus on Acute Myeloid Leukemia (AML) - A Systematic Review. Current Medicinal Chemistry. 27(28). 4673–4698. 23 indexed citations
18.
Zhao, Yueshui, Qijie Zhao, Parham Jabbarzadeh Kaboli, et al.. (2019). m1A Regulated Genes Modulate PI3K/AKT/mTOR and ErbB Pathways in Gastrointestinal Cancer. Translational Oncology. 12(10). 1323–1333. 121 indexed citations
19.
Zhao, Yueshui, Ling Lin, Zhangang Xiao, et al.. (2018). Protective Role ofγδT Cells in Different Pathogen Infections and Its Potential Clinical Application. Journal of Immunology Research. 2018. 1–15. 49 indexed citations
20.
Wang, Beilei, Bo Zhang, Hua Huo, et al.. (2014). Detection of microvasculature alterations by synchrotron radiation in murine with delayed jellyfish envenomation syndrome. Toxicon. 81. 48–53. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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