Young‐Saeng Jang

450 total citations
25 papers, 376 citations indexed

About

Young‐Saeng Jang is a scholar working on Immunology, Molecular Biology and Nutrition and Dietetics. According to data from OpenAlex, Young‐Saeng Jang has authored 25 papers receiving a total of 376 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Immunology, 6 papers in Molecular Biology and 6 papers in Nutrition and Dietetics. Recurrent topics in Young‐Saeng Jang's work include Immune Cell Function and Interaction (13 papers), T-cell and B-cell Immunology (8 papers) and Immunotherapy and Immune Responses (7 papers). Young‐Saeng Jang is often cited by papers focused on Immune Cell Function and Interaction (13 papers), T-cell and B-cell Immunology (8 papers) and Immunotherapy and Immune Responses (7 papers). Young‐Saeng Jang collaborates with scholars based in South Korea, United States and Croatia. Young‐Saeng Jang's co-authors include Pyeung-Hyeun Kim, Goo‐Young Seo, Eun‐Ju Chang, Bongkun Choi, Jeong Min Lee, Seok‐Rae Park, Eun Jin Lee, Da‐Hyun Song, Youngsup Song and Sang‐Wook Kang and has published in prestigious journals such as The Journal of Immunology, Journal of Cellular Physiology and Journal of Leukocyte Biology.

In The Last Decade

Young‐Saeng Jang

25 papers receiving 374 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Young‐Saeng Jang South Korea 11 172 128 44 40 38 25 376
Xinyue Li China 12 80 0.5× 163 1.3× 36 0.8× 30 0.8× 23 0.6× 38 449
Ellen Bleck Germany 13 117 0.7× 108 0.8× 36 0.8× 14 0.3× 36 0.9× 25 405
Nongyu Huang China 10 140 0.8× 206 1.6× 26 0.6× 67 1.7× 14 0.4× 17 418
Carla Álvarez Chile 19 280 1.6× 211 1.6× 41 0.9× 51 1.3× 48 1.3× 37 794
Marcelo Mattos United States 7 101 0.6× 231 1.8× 50 1.1× 44 1.1× 11 0.3× 8 594
Anastasia Stavropoulou Greece 11 84 0.5× 141 1.1× 47 1.1× 32 0.8× 7 0.2× 18 404
Hannah Coleman United States 13 148 0.9× 193 1.5× 211 4.8× 63 1.6× 22 0.6× 29 548
Libor Kolesár Czechia 14 195 1.1× 116 0.9× 78 1.8× 39 1.0× 19 0.5× 30 602
Ziwei Jiang China 5 165 1.0× 98 0.8× 41 0.9× 50 1.3× 10 0.3× 13 427
Y Tsuji Japan 14 192 1.1× 81 0.6× 30 0.7× 30 0.8× 63 1.7× 30 489

Countries citing papers authored by Young‐Saeng Jang

Since Specialization
Citations

This map shows the geographic impact of Young‐Saeng Jang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Young‐Saeng Jang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Young‐Saeng Jang more than expected).

Fields of papers citing papers by Young‐Saeng Jang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Young‐Saeng Jang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Young‐Saeng Jang. The network helps show where Young‐Saeng Jang may publish in the future.

Co-authorship network of co-authors of Young‐Saeng Jang

This figure shows the co-authorship network connecting the top 25 collaborators of Young‐Saeng Jang. A scholar is included among the top collaborators of Young‐Saeng Jang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Young‐Saeng Jang. Young‐Saeng Jang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kim, Dong-Hyun, Gi Hong Choi, Han Na Lee, et al.. (2025). Treatment of IL-18-binding protein biologics suppresses fibrotic progression in metabolic dysfunction-associated steatohepatitis. Cell Reports Medicine. 6(4). 102047–102047. 3 indexed citations
2.
Jang, Young‐Saeng, Jung Tak Park, Seung Goo Kang, et al.. (2023). Combined Treatment With TGF-β1, Retinoic Acid, and Lactoferrin Robustly Generate Inducible Tregs (iTregs) Against High Affinity Ligand. Immune Network. 23(5). e37–e37. 2 indexed citations
3.
Jang, Young‐Saeng, Kyungsun Lee, Mi‐Hyun Park, et al.. (2023). Albumin-binding recombinant human IL-18BP ameliorates macrophage activation syndrome and atopic dermatitis via direct IL-18 inactivation. Cytokine. 172. 156413–156413. 7 indexed citations
4.
Jang, Young‐Saeng, Goo‐Young Seo, Sunhee Park, et al.. (2021). Lactoferrin Potentiates Inducible Regulatory T Cell Differentiation through TGF-β Receptor III Binding and Activation of Membrane-Bound TGF-β. The Journal of Immunology. 207(10). 2456–2464. 3 indexed citations
5.
Park, Jung Tak, et al.. (2020). Lactoferrin Induces Tolerogenic Bone Marrow-Derived Dendritic Cells. Immune Network. 20(5). e38–e38. 3 indexed citations
7.
Kim, Pyeung-Hyeun, Young‐Saeng Jang, Goo‐Young Seo, et al.. (2018). Mechanism underlying the induction of Foxp3+ regulatory T cells by lactoferrin. The Journal of Immunology. 200(Supplement_1). 47.16–47.16. 1 indexed citations
8.
Jang, Young‐Saeng, Goo‐Young Seo, Sung‐Gyoo Park, et al.. (2018). Murine γδ T Cells Render B Cells Refractory to Commitment of IgA Isotype Switching. Immune Network. 18(4). e25–e25. 4 indexed citations
9.
Seo, Goo‐Young, Jeong Min Lee, Young‐Saeng Jang, et al.. (2017). Mechanism underlying the suppressor activity of retinoic acid on IL4-induced IgE synthesis and its physiological implication. Cellular Immunology. 322. 49–55. 18 indexed citations
10.
Lee, Eun Jin, Young‐Saeng Jang, Kwonyoon Kang, et al.. (2015). Atrazine induces endoplasmic reticulum stress‐mediated apoptosis of T lymphocytes via the caspase‐8‐dependent pathway. Environmental Toxicology. 31(8). 998–1008. 21 indexed citations
11.
Lee, Jeong Min, Young‐Saeng Jang, Sun‐Jin Kim, et al.. (2015). Retinoic acid enhances lactoferrin-induced IgA responses by increasing betaglycan expression. Cellular and Molecular Immunology. 13(6). 862–870. 18 indexed citations
12.
Kim, Hyeonjin, Goo‐Young Seo, Young‐Saeng Jang, et al.. (2015). Lactoferrin Combined with Retinoic Acid Stimulates B1 Cells to Express IgA Isotype and Gut-homing Molecules. Immune Network. 15(1). 37–37. 14 indexed citations
13.
Chung, Yeon‐Ho, Young‐Saeng Jang, Bongkun Choi, et al.. (2014). Beclin‐1 Is Required for RANKL‐Induced Osteoclast Differentiation. Journal of Cellular Physiology. 229(12). 1963–1971. 56 indexed citations
14.
Jang, Young‐Saeng & In‐Hong Choi. (2014). Contrasting Roles of Different Endoglin Forms in Atherosclerosis. Immune Network. 14(5). 237–237. 18 indexed citations
15.
Choi, Bongkun, Eun Jin Lee, Da‐Hyun Song, et al.. (2014). Elevated Pentraxin 3 in bone metastatic breast cancer is correlated with osteolytic function. Oncotarget. 5(2). 481–492. 54 indexed citations
16.
Chang, Hyun‐Kyung, et al.. (2012). Lactoferrin Stimulates Mouse Macrophage to Express BAFF via Smad3 Pathway. Immune Network. 12(3). 84–84. 3 indexed citations
17.
Jang, Young‐Saeng, Jaehee Kim, Goo‐Young Seo, & Pyeung-Hyeun Kim. (2011). TGF-β1 Stimulates Mouse Macrophages to Express APRIL through Smad and p38MAPK/CREB Pathways. Molecules and Cells. 32(3). 251–256. 10 indexed citations
18.
Jang, Young‐Saeng, et al.. (2009). IL-4 stimulates mouse macrophages to express APRIL through p38MAPK and two different downstream molecules, CREB and Stat6. Cytokine. 47(1). 43–47. 13 indexed citations
19.
Jang, Young‐Saeng, et al.. (2009). Development of the Rheocast Diesel Engine Block with ADC10 Alloy via ART (Advanced Rheocasting Technology) System. ISIJ International. 49(11). 1700–1709. 10 indexed citations
20.
Jang, Young‐Saeng, et al.. (2004). Allergen‐specific immunosuppression by ovalbumin fused with diphtheria toxin in mice sensitized with albumins of different origin. Clinical & Experimental Allergy. 34(10). 1642–1648. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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