Yongfang Ma
- Molecular Biology
- Cancer Research
- Pathology and Forensic Medicine
- Oncology
- Pulmonary and Respiratory Medicine
- Topics
- PI3K/AKT/mTOR signaling in cancer (6 papers)Cancer Mechanisms and Therapy (5 papers)MicroRNA in disease regulation (4 papers)
- Journals
- Environmental Science & TechnologyThe Science of The Total EnvironmentJournal of Hazardous Materials
- Partner nations
- ChinaGermanyUnited States
In The Last Decade
Yongfang Ma
23 papers receiving 371 citations
Peers
Comparison fields: 5 of 66
- Molecular Biology 223
- Cancer Research 109
- Pathology and Forensic Medicine 74
- Oncology 66
- Pulmonary and Respiratory Medicine 58
Countries citing papers authored by Yongfang Ma
This map shows the geographic impact of Yongfang Ma's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yongfang Ma with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yongfang Ma more than expected).
Fields of papers citing papers by Yongfang Ma
This network shows the impact of papers produced by Yongfang Ma. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yongfang Ma. The network helps show where Yongfang Ma may publish in the future.
Co-authorship network of co-authors of Yongfang Ma
This figure shows the co-authorship network connecting the top 25 collaborators of Yongfang Ma. A scholar is included among the top collaborators of Yongfang Ma based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yongfang Ma. Yongfang Ma is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 0 | |
| 3 | 22 | |
| 4 | 16 | |
| 5 | 1 | |
| 6 | 12 | |
| 7 | ETS-1/c-Met drives resistance to sorafenib in hepatocellular carcinoma. | 4 |
| 8 | 3 | |
| 9 | 18 | |
| 10 | 11 | |
| 11 | 19 | |
| 12 | 2 | |
| 13 | 22 | |
| 14 | 28 | |
| 15 | 70 | |
| 16 | 14 | |
| 17 | 17 | |
| 18 | 1 | |
| 19 | PKI-587 enhances chemosensitivity of oxaliplatin in hepatocellular carcinoma through suppressing DNA damage repair pathway (NHEJ and HR) and PI3K/AKT/mTOR pathway. | 36 |
| 20 | BEZ235 increases sorafenib inhibition of hepatocellular carcinoma cells by suppressing the PI3K/AKT/mTOR pathway. | 29 |
About Yongfang Ma
Yongfang Ma is a scholar working on Physiology, Cancer Research and Hepatology, having authored 25 papers that have together received 376 indexed citations. Recurring topics across this work include PI3K/AKT/mTOR signaling in cancer (6 papers), Cancer Mechanisms and Therapy (5 papers) and MicroRNA in disease regulation (4 papers). The work is most often cited by research in Cancer Research (109 citations), Hepatology (42 citations) and Pathology and Forensic Medicine (74 citations). Yongfang Ma has collaborated with scholars based in China, Germany and United States. Frequent co-authors include Ruyue Xu, Shuping Zhou, Weiya Cao, Amin Li, Yinci Zhang, Song Li, Xueke Liu, Lunxu Liu, Qiang Pu and Yuan Yue. Their work appears in journals such as Environmental Science & Technology, The Science of The Total Environment and Journal of Hazardous Materials.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.