Yoichiro Kaku

405 total citations
11 papers, 323 citations indexed

About

Yoichiro Kaku is a scholar working on Immunology, Physiology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Yoichiro Kaku has authored 11 papers receiving a total of 323 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Immunology, 6 papers in Physiology and 3 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Yoichiro Kaku's work include Asthma and respiratory diseases (5 papers), Immune Cell Function and Interaction (4 papers) and Immune cells in cancer (3 papers). Yoichiro Kaku is often cited by papers focused on Asthma and respiratory diseases (5 papers), Immune Cell Function and Interaction (4 papers) and Immune cells in cancer (3 papers). Yoichiro Kaku collaborates with scholars based in Japan and Canada. Yoichiro Kaku's co-authors include Tomoaki Hoshino, Tomotaka Kawayama, Shinichi Takenaka, Haruki Imaoka, Masanobu Matsuoka, Masaki Okamoto, Yuki Sakazaki, Motohiro Takeya, Yoshihiro Komohara and Yoshitaka Morimatsu and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Biochemical and Biophysical Research Communications.

In The Last Decade

Yoichiro Kaku

11 papers receiving 320 citations

Peers

Yoichiro Kaku

IMMUN

PHYSI

PRM

MB

DERMA

Shinichi Takenaka Japan

Andrea G. Bree United States

Pierre Camateros Canada

Luc Colas France

Christina H. Swasey United States

Julianty Angsana United States

Chiara Cardamone Italy

Nina Sopel Germany

Katherine A. Weissler United States

Luis Fernández-Pereira Spain

Shinichi Takenaka Japan

Yoichiro Kaku

192 ×1.0

IMMUN

90 ×0.9

PHYSI

94 ×1.1

PRM

113 ×1.3

MB

34 ×0.8

DERMA

Citations per year, relative to Yoichiro Kaku Yoichiro Kaku (= 1×) peers Shinichi Takenaka

Countries citing papers authored by Yoichiro Kaku

Since Specialization

Citations

This map shows the geographic impact of Yoichiro Kaku's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yoichiro Kaku with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yoichiro Kaku more than expected).

Fields of papers citing papers by Yoichiro Kaku

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yoichiro Kaku. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yoichiro Kaku. The network helps show where Yoichiro Kaku may publish in the future.

Co-authorship network of co-authors of Yoichiro Kaku

This figure shows the co-authorship network connecting the top 25 collaborators of Yoichiro Kaku. A scholar is included among the top collaborators of Yoichiro Kaku based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yoichiro Kaku. Yoichiro Kaku is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

1.

Tokunaga, Yoshihisa, Haruki Imaoka, Yoichiro Kaku, Tomotaka Kawayama, & Tomoaki Hoshino. (2019). The significance of CD163-expressing macrophages in asthma. Annals of Allergy Asthma & Immunology. 123(3). 263–270. 10 indexed citations

2.

Imaoka, Haruki, Yoshitaka Morimatsu, Yoichiro Kaku, et al.. (2018). Role of CD163 in human fatal asthma and murine asthma model.. PA648–PA648. 1 indexed citations

3.

Matsuoka, Masanobu, Tomotaka Kawayama, Masaki Tominaga, et al.. (2018). Attenuated Airway Eosinophilic Inflammations in IL-38 Knockout Mouse Model. The Kurume Medical Journal. 65(2). 37–46. 14 indexed citations

4.

Takenaka, Shinichi, Shinjiro Kaieda, Tomotaka Kawayama, et al.. (2015). IL-38: A new factor in rheumatoid arthritis. Biochemistry and Biophysics Reports. 4. 386–391. 59 indexed citations

5.

Kaku, Yoichiro, et al.. (2014). M2 macrophage marker CD163, CD204 and CD206 expression on alveolar macrophages in the lung of patients with chronic obstructive pulmonary. European Respiratory Journal. 44(Suppl 58). P1481–P1481. 2 indexed citations

6.

Kaku, Yoichiro, Haruki Imaoka, Yoshitaka Morimatsu, et al.. (2014). Overexpression of CD163, CD204 and CD206 on Alveolar Macrophages in the Lungs of Patients with Severe Chronic Obstructive Pulmonary Disease. PLoS ONE. 9(1). e87400–e87400. 116 indexed citations

7.

Takenaka, Shinichi, Tomotaka Kawayama, Haruki Imaoka, et al.. (2014). The progression of comorbidity in IL-18 transgenic chronic obstructive pulmonary disease mice model. Biochemical and Biophysical Research Communications. 445(3). 597–601. 10 indexed citations

8.

Kawayama, Tomotaka, Haruki Imaoka, Yuki Sakazaki, et al.. (2013). Interleukin-18 expression, CD8+ T cells, and eosinophils in lungs of nonsmokers with fatal asthma. Annals of Allergy Asthma & Immunology. 112(1). 23–28.e1. 34 indexed citations

9.

Kawayama, Tomotaka, Kazuko Matsunaga, Yoichiro Kaku, et al.. (2013). Decreased CTLA4+ and Foxp3+ CD25highCD4+ Cells in Induced Sputum from Patients with Mild Atopic Asthma. Allergology International. 62(2). 203–213. 22 indexed citations

10.

Kawayama, Tomotaka, Haruki Imaoka, Yuki Sakazaki, et al.. (2013). IL-18 Induces Airway Hyperresponsiveness and Pulmonary Inflammation via CD4+ T Cell and IL-13. PLoS ONE. 8(1). e54623–e54623. 51 indexed citations

11.

Yokoyama, Toshinobu, et al.. (2011). A necrotic lung ball caused by co-infection with Candida and Streptococcus pneumoniae. SHILAP Revista de lepidopterología. 221–221. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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