Yinxian Wen

895 total citations
49 papers, 595 citations indexed

About

Yinxian Wen is a scholar working on Molecular Biology, Pediatrics, Perinatology and Child Health and Rheumatology. According to data from OpenAlex, Yinxian Wen has authored 49 papers receiving a total of 595 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 15 papers in Pediatrics, Perinatology and Child Health and 14 papers in Rheumatology. Recurrent topics in Yinxian Wen's work include Birth, Development, and Health (14 papers), Osteoarthritis Treatment and Mechanisms (13 papers) and Orthopedic Infections and Treatments (7 papers). Yinxian Wen is often cited by papers focused on Birth, Development, and Health (14 papers), Osteoarthritis Treatment and Mechanisms (13 papers) and Orthopedic Infections and Treatments (7 papers). Yinxian Wen collaborates with scholars based in China, France and United States. Yinxian Wen's co-authors include Liaobin Chen, Hui Wang, Yangfan Shangguan, Jacques Magdalou, Hao Xiao, Jun Qin, Linlong Wang, Qingxian Li, Biao Chen and Yang Tan and has published in prestigious journals such as PLoS ONE, Scientific Reports and Chemical Engineering Journal.

In The Last Decade

Yinxian Wen

46 papers receiving 589 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yinxian Wen China 14 209 133 99 87 71 49 595
Hyun Jin Jung South Korea 20 272 1.3× 137 1.0× 54 0.5× 121 1.4× 25 0.4× 75 838
Zhe Su China 14 220 1.1× 98 0.7× 31 0.3× 92 1.1× 30 0.4× 70 566
Necat Yılmaz Türkiye 15 145 0.7× 52 0.4× 62 0.6× 169 1.9× 36 0.5× 77 740
Ying Chang China 13 156 0.7× 81 0.6× 16 0.2× 76 0.9× 47 0.7× 43 597
Robert Klepacz Poland 12 92 0.4× 41 0.3× 65 0.7× 109 1.3× 55 0.8× 39 469
Yu Seob Shin South Korea 18 152 0.7× 31 0.2× 122 1.2× 185 2.1× 48 0.7× 94 921
Hala T. El‐Bassyouni Egypt 12 188 0.9× 68 0.5× 84 0.8× 99 1.1× 10 0.1× 91 593
Sonja Barth Austria 12 95 0.5× 134 1.0× 28 0.3× 80 0.9× 27 0.4× 18 519

Countries citing papers authored by Yinxian Wen

Since Specialization
Citations

This map shows the geographic impact of Yinxian Wen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yinxian Wen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yinxian Wen more than expected).

Fields of papers citing papers by Yinxian Wen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yinxian Wen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yinxian Wen. The network helps show where Yinxian Wen may publish in the future.

Co-authorship network of co-authors of Yinxian Wen

This figure shows the co-authorship network connecting the top 25 collaborators of Yinxian Wen. A scholar is included among the top collaborators of Yinxian Wen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yinxian Wen. Yinxian Wen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Liu, Siyi, Haitao Chen, Yang Xu, Yinxian Wen, & Liaobin Chen. (2024). Identification and validation of up-regulated TNFAIP6 in osteoarthritis with type 2 diabetes mellitus. Scientific Reports. 14(1). 31450–31450.
2.
Chen, Haitao, Ming Tu, Siyi Liu, Yinxian Wen, & Liaobin Chen. (2023). Dendrobine Alleviates Cellular Senescence and Osteoarthritis via the ROS/NF-κB Axis. International Journal of Molecular Sciences. 24(3). 2365–2365. 36 indexed citations
3.
Gao, Weihang, et al.. (2023). Multifunctional gold nanoparticles for osteoporosis: synthesis, mechanism and therapeutic applications. Journal of Translational Medicine. 21(1). 889–889. 13 indexed citations
4.
Liu, Liang, Yinxian Wen, Qubo Ni, Liaobin Chen, & Hui Wang. (2023). Prenatal ethanol exposure and changes in fetal neuroendocrine metabolic programming. Biological Research. 56(1). 61–61. 4 indexed citations
5.
Shangguan, Yangfan, Xufeng Li, Jun Qin, et al.. (2022). Positive programming of the GC-IGF1 axis mediates adult osteoporosis susceptibility in male offspring rats induced by prenatal dexamethasone exposure. Biochemical Pharmacology. 206. 115264–115264. 9 indexed citations
6.
Li, Qingxian, Yinxian Wen, Linlong Wang, et al.. (2021). Hyperglycemia-induced accumulation of advanced glycosylation end products in fibroblast-like synoviocytes promotes knee osteoarthritis. Experimental & Molecular Medicine. 53(11). 1735–1747. 67 indexed citations
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Qi, Yongjian, Bin Li, Yinxian Wen, et al.. (2021). H3K9ac of TGFβRI in human umbilical cord: a potential biomarker for evaluating cartilage differentiation and susceptibility to osteoarthritis via a two-step strategy. Stem Cell Research & Therapy. 12(1). 163–163. 9 indexed citations
10.
Wu, Zhixin, et al.. (2021). HEMGN and SLC2A1 might be potential diagnostic biomarkers of steroid-induced osteonecrosis of femoral head: study based on WGCNA and DEGs screening. BMC Musculoskeletal Disorders. 22(1). 85–85. 11 indexed citations
11.
Xiao, Hao, Yinxian Wen, Yang Tan, et al.. (2020). Subchondral bone dysplasia partly participates in prenatal dexamethasone induced-osteoarthritis susceptibility in female offspring rats. Bone. 133. 115245–115245. 14 indexed citations
12.
Hu, Wen, Guihua Wang, Bo He, et al.. (2020). Effects of prenatal nicotine exposure on hepatic glucose and lipid metabolism in offspring rats and its hereditability. Toxicology. 432. 152378–152378. 11 indexed citations
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Wu, Zhixin, et al.. (2019). Egr1/p300/ACE signal mediates postnatal osteopenia in female rat offspring induced by prenatal ethanol exposure. Food and Chemical Toxicology. 136. 111083–111083. 15 indexed citations
15.
Wen, Yinxian, Yangfan Shangguan, Hang Hu, et al.. (2018). Activation of local bone RAS by maternal excessive glucocorticoid participated in the fetal programing of adult osteopenia induced by prenatal caffeine exposure. Toxicology and Applied Pharmacology. 363. 1–10. 9 indexed citations
16.
Shangguan, Yangfan, Yinxian Wen, Yang Tan, et al.. (2018). Intrauterine Programming of Glucocorticoid–Insulin-Like Growth Factor-1 Axis–Mediated Developmental Origin of Osteoporosis Susceptibility in Female Offspring Rats with Prenatal Caffeine Exposure. American Journal Of Pathology. 188(12). 2863–2876. 21 indexed citations
17.
Xiao, Hao, Yinxian Wen, Yangfan Shangguan, et al.. (2018). Increased H3K27ac level of ACE mediates the intergenerational effect of low peak bone mass induced by prenatal dexamethasone exposure in male offspring rats. Cell Death and Disease. 9(6). 638–638. 52 indexed citations
18.
Qi, Yongjian, et al.. (2016). Intravenous morphine titration as a rapid and efficient analgesia for adult patients with femoral shaft fractures after injury. The American Journal of Emergency Medicine. 34(11). 2107–2111. 3 indexed citations
19.
Luo, Hanwen, Hao Kou, Yinxian Wen, et al.. (2015). Prenatal caffeine exposure induced a lower level of fetal blood leptin mainly via placental mechanism. Toxicology and Applied Pharmacology. 289(1). 109–116. 39 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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