Yingqiang Du

479 total citations
22 papers, 383 citations indexed

About

Yingqiang Du is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Surgery. According to data from OpenAlex, Yingqiang Du has authored 22 papers receiving a total of 383 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 10 papers in Cardiology and Cardiovascular Medicine and 4 papers in Surgery. Recurrent topics in Yingqiang Du's work include Cardiac Fibrosis and Remodeling (6 papers), Signaling Pathways in Disease (5 papers) and Inflammasome and immune disorders (4 papers). Yingqiang Du is often cited by papers focused on Cardiac Fibrosis and Remodeling (6 papers), Signaling Pathways in Disease (5 papers) and Inflammasome and immune disorders (4 papers). Yingqiang Du collaborates with scholars based in China, United States and Poland. Yingqiang Du's co-authors include Zhijian Yang, Peng Wu, Yingbin Ge, Zhihui Xu, Xin Gu, Nan Aa, Jia Liu, Ningtian Zhou, Haoyu Meng and Chengyi Peng and has published in prestigious journals such as Journal of Hazardous Materials, Biochemical and Biophysical Research Communications and BMC Public Health.

In The Last Decade

Yingqiang Du

17 papers receiving 381 citations

Peers

Countries citing papers authored by Yingqiang Du

Since Specialization

Citations

This map shows the geographic impact of Yingqiang Du's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yingqiang Du with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yingqiang Du more than expected).

Fields of papers citing papers by Yingqiang Du

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yingqiang Du. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yingqiang Du. The network helps show where Yingqiang Du may publish in the future.

Co-authorship network of co-authors of Yingqiang Du

This figure shows the co-authorship network connecting the top 25 collaborators of Yingqiang Du. A scholar is included among the top collaborators of Yingqiang Du based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yingqiang Du. Yingqiang Du is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Title	Journal	Authors	Indexed citations
1	Baicalin reduced vandetanib induced myocardial injury by regulating redox balance and NLRP3 inflammasome pathway	Tissue and Cell	Fen Wang, Zhixuan Zhang et al.	0
2	Exposure to polyethylene terephthalate micro(nano)plastics exacerbates inflammation and fibrosis after myocardial infarction by reprogramming the gut and lung microbiota and metabolome	Journal of Hazardous Materials	Xin Gu, Zhixuan Zhang et al.	9
3	Knowledge, attitude, and practice of coronary heart disease patients towards antithrombotic therapy	BMC Public Health	Yingqiang Du, Hong Xin et al.	0
4	p16INK4a Aggravated Sepsis-associated Cardiac Injury by Inhibiting the PI3K/AKT Pathway and Inducing Redox Imbalance	Journal of Cardiovascular Translational Research	Baihong Li, Wei Wang et al.	0
5	Fibroblasts‐specific p16^INK4a exacerbates inflammageing‐mediated post‐infarction ventricular remodelling through interacting with STAT3 to regulate NLRP3 transcription	Clinical and Translational Medicine	Xin Gu, Yingqiang Du et al.	0
6	Rosmarinic acid attenuates TNF-α induced cardiomyocyte injury via regulatingmiR-344a-3p	Cellular and Molecular Biology	Jun Zhang, Yingqiang Du et al.	0
7	MAP4K4 exacerbates cardiac microvascular injury in diabetes by facilitating S-nitrosylation modification of Drp1	Cardiovascular Diabetology	Yuqiong Chen, Bo Guan et al.	20
8	P16INK4a deletion alleviates contrast-induced acute kidney injury by ameliorating renal cell apoptosis and suppressing inflammation and oxidative stress	Experimental Gerontology	Xiaodong Zhang, Guangyi Huang et al.	5
9	1,25(OH)₂D₃ ameliorates doxorubicin‑induced cardiomyopathy by inhibiting the NLRP3 inflammasome and oxidative stress	Experimental and Therapeutic Medicine	Xin Gu, Lin Zhao et al.	1
10	Inflammasome activation and metabolic remodelling in p16‐positive aging cells aggravates high‐fat diet‐induced lung fibrosis by inhibiting NEDD4L‐mediated K48‐polyubiquitin‐dependent degradation of SGK1	Clinical and Translational Medicine	Xin Gu, Haoyu Meng et al.	15
11	The crystallin alpha B (HSPB5)-tripartite motif containing 33 (TRIM33) axis mediates myocardial fibrosis induced by angiotensinogen II through transforming growth factor-β (TGF-β1)-Smad3/4 signaling	Bioengineered	Yingqiang Du, Tongtong Yang et al.	10
12	Roflumilast Attenuates Doxorubicin-Induced Cardiotoxicity by Targeting Inflammation and Cellular Senescence in Cardiomyocytes Mediated by SIRT1	Drug Design Development and Therapy	Sheng Zhang, Peng Wu et al.	25
13	Resveratrol improves cardiac function by promoting M2-like polarization of macrophages in mice with myocardial infarction.	PubMed	Yingqiang Du, Kexin Shi et al.	27
14	P16^INK4a played a critical role in exacerbating acute tubular necrosis in acute kidney injury.	PubMed	Xin Gu, Chengyi Peng et al.	16
15	Hypoxia-Inducible Factor 1 alpha (HIF-1α)/Vascular Endothelial Growth Factor (VEGF) Pathway Participates in Angiogenesis of Myocardial Infarction in Muscone-Treated Mice: Preliminary Study	Medical Science Monitor	Yingqiang Du, Yingbin Ge et al.	59
16	Muscone improves cardiac function in mice after myocardial infarction by alleviating cardiac macrophage-mediated chronic inflammation through inhibition of NF-κB and NLRP3 inflammasome.	PubMed	Yingqiang Du, Xin Gu et al.	49
17	Exogenous HGF Prevents Cardiomyocytes from Apoptosis after Hypoxia via Up-Regulating Cell Autophagy	Cellular Physiology and Biochemistry	Jia Liu, Zhiwen Tao et al.	25
18	Necroptosis Induced by Ad-HGF Activates Endogenous C-Kit+ Cardiac Stem Cells and Promotes Cardiomyocyte Proliferation and Angiogenesis in the Infarcted Aged Heart	Cellular Physiology and Biochemistry	Jia Liu, Peng Wu et al.	20
19	Ad-HGF improves the cardiac remodeling of rat following myocardial infarction by upregulating autophagy and necroptosis and inhibiting apoptosis.	PubMed	Jia Liu, Peng Wu et al.	38
20	Puerarin improves cardiac function through regulation of energy metabolism in Streptozotocin-Nicotinamide induced diabetic mice after myocardial infarction	Biochemical and Biophysical Research Communications	Peng Wu, Yingqiang Du et al.	40

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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