Yingqiang Du

479 total citations
22 papers, 383 citations indexed

About

Yingqiang Du is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Surgery. According to data from OpenAlex, Yingqiang Du has authored 22 papers receiving a total of 383 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 10 papers in Cardiology and Cardiovascular Medicine and 4 papers in Surgery. Recurrent topics in Yingqiang Du's work include Cardiac Fibrosis and Remodeling (6 papers), Signaling Pathways in Disease (5 papers) and Inflammasome and immune disorders (4 papers). Yingqiang Du is often cited by papers focused on Cardiac Fibrosis and Remodeling (6 papers), Signaling Pathways in Disease (5 papers) and Inflammasome and immune disorders (4 papers). Yingqiang Du collaborates with scholars based in China, United States and Poland. Yingqiang Du's co-authors include Zhijian Yang, Peng Wu, Yingbin Ge, Zhihui Xu, Xin Gu, Nan Aa, Jia Liu, Ningtian Zhou, Haoyu Meng and Chengyi Peng and has published in prestigious journals such as Journal of Hazardous Materials, Biochemical and Biophysical Research Communications and BMC Public Health.

In The Last Decade

Yingqiang Du

17 papers receiving 381 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yingqiang Du China 12 201 80 53 46 45 22 383
Ningtian Zhou China 11 219 1.1× 91 1.1× 50 0.9× 56 1.2× 45 1.0× 20 388
Dina Xie United States 11 203 1.0× 133 1.7× 49 0.9× 49 1.1× 59 1.3× 16 379
Jinlong Luo China 13 237 1.2× 42 0.5× 59 1.1× 28 0.6× 48 1.1× 32 533
Jiang Shi-sen China 11 140 0.7× 83 1.0× 77 1.5× 24 0.5× 59 1.3× 29 359
Xiyun Bian China 13 280 1.4× 73 0.9× 33 0.6× 47 1.0× 82 1.8× 29 472
Wan‐Ching Chou Taiwan 9 154 0.8× 75 0.9× 30 0.6× 32 0.7× 20 0.4× 15 343
Chuansheng Xu China 10 202 1.0× 126 1.6× 41 0.8× 41 0.9× 26 0.6× 16 401
Tongtong Cao China 11 293 1.5× 97 1.2× 73 1.4× 35 0.8× 66 1.5× 17 560
Junxian Cao China 14 209 1.0× 120 1.5× 29 0.5× 38 0.8× 28 0.6× 28 527
Xukun Bi China 13 198 1.0× 71 0.9× 51 1.0× 36 0.8× 65 1.4× 17 379

Countries citing papers authored by Yingqiang Du

Since Specialization
Citations

This map shows the geographic impact of Yingqiang Du's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yingqiang Du with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yingqiang Du more than expected).

Fields of papers citing papers by Yingqiang Du

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yingqiang Du. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yingqiang Du. The network helps show where Yingqiang Du may publish in the future.

Co-authorship network of co-authors of Yingqiang Du

This figure shows the co-authorship network connecting the top 25 collaborators of Yingqiang Du. A scholar is included among the top collaborators of Yingqiang Du based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yingqiang Du. Yingqiang Du is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wang, Fen, Zhixuan Zhang, Guangyi Huang, et al.. (2025). Baicalin reduced vandetanib induced myocardial injury by regulating redox balance and NLRP3 inflammasome pathway. Tissue and Cell. 94. 102795–102795.
2.
Gu, Xin, Zhixuan Zhang, Lin Zhao, et al.. (2025). Exposure to polyethylene terephthalate micro(nano)plastics exacerbates inflammation and fibrosis after myocardial infarction by reprogramming the gut and lung microbiota and metabolome. Journal of Hazardous Materials. 488. 137410–137410. 9 indexed citations
3.
Du, Yingqiang, Hong Xin, Yuqiong Chen, et al.. (2025). Knowledge, attitude, and practice of coronary heart disease patients towards antithrombotic therapy. BMC Public Health. 25(1). 549–549.
4.
Li, Baihong, Wei Wang, Xiaoyan Wang, et al.. (2025). p16INK4a Aggravated Sepsis-associated Cardiac Injury by Inhibiting the PI3K/AKT Pathway and Inducing Redox Imbalance. Journal of Cardiovascular Translational Research. 18(2). 375–391.
5.
6.
Zhang, Jun, Yingqiang Du, & Feili Li. (2024). Rosmarinic acid attenuates TNF-α induced cardiomyocyte injury via regulatingmiR-344a-3p. Cellular and Molecular Biology. 70(1). 207–212.
7.
Chen, Yuqiong, Bo Guan, Yingqiang Du, et al.. (2024). MAP4K4 exacerbates cardiac microvascular injury in diabetes by facilitating S-nitrosylation modification of Drp1. Cardiovascular Diabetology. 23(1). 164–164. 20 indexed citations
8.
Zhang, Xiaodong, Guangyi Huang, Zhixuan Zhang, et al.. (2024). P16INK4a deletion alleviates contrast-induced acute kidney injury by ameliorating renal cell apoptosis and suppressing inflammation and oxidative stress. Experimental Gerontology. 187. 112372–112372. 5 indexed citations
9.
Gu, Xin, Lin Zhao, Jiabao Ye, et al.. (2023). 1,25(OH)2D3 ameliorates doxorubicin‑induced cardiomyopathy by inhibiting the NLRP3 inflammasome and oxidative stress. Experimental and Therapeutic Medicine. 26(3). 413–413. 1 indexed citations
10.
11.
12.
Zhang, Sheng, Peng Wu, Jia Liu, Yingqiang Du, & Zhijian Yang. (2021). Roflumilast Attenuates Doxorubicin-Induced Cardiotoxicity by Targeting Inflammation and Cellular Senescence in Cardiomyocytes Mediated by SIRT1. Drug Design Development and Therapy. Volume 15. 87–97. 25 indexed citations
13.
Du, Yingqiang, et al.. (2019). Resveratrol improves cardiac function by promoting M2-like polarization of macrophages in mice with myocardial infarction.. PubMed. 11(8). 5212–5226. 27 indexed citations
14.
Gu, Xin, Chengyi Peng, Shiyu Lin, et al.. (2019). P16INK4a played a critical role in exacerbating acute tubular necrosis in acute kidney injury.. PubMed. 11(6). 3850–3861. 16 indexed citations
15.
16.
Du, Yingqiang, Xin Gu, Haoyu Meng, et al.. (2018). Muscone improves cardiac function in mice after myocardial infarction by alleviating cardiac macrophage-mediated chronic inflammation through inhibition of NF-κB and NLRP3 inflammasome.. PubMed. 10(12). 4235–4246. 49 indexed citations
17.
Liu, Jia, Zhiwen Tao, Peng Wu, et al.. (2016). Exogenous HGF Prevents Cardiomyocytes from Apoptosis after Hypoxia via Up-Regulating Cell Autophagy. Cellular Physiology and Biochemistry. 38(6). 2401–2413. 25 indexed citations
18.
Liu, Jia, Peng Wu, Hao Wang, et al.. (2016). Necroptosis Induced by Ad-HGF Activates Endogenous C-Kit+ Cardiac Stem Cells and Promotes Cardiomyocyte Proliferation and Angiogenesis in the Infarcted Aged Heart. Cellular Physiology and Biochemistry. 40(5). 847–860. 20 indexed citations
19.
Liu, Jia, Peng Wu, Yingqiang Du, et al.. (2016). Ad-HGF improves the cardiac remodeling of rat following myocardial infarction by upregulating autophagy and necroptosis and inhibiting apoptosis.. PubMed. 8(11). 4605–4627. 38 indexed citations
20.
Wu, Peng, et al.. (2015). Puerarin improves cardiac function through regulation of energy metabolism in Streptozotocin-Nicotinamide induced diabetic mice after myocardial infarction. Biochemical and Biophysical Research Communications. 463(4). 1108–1114. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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