Ying‐Qian Lu

482 total citations
12 papers, 240 citations indexed

About

Ying‐Qian Lu is a scholar working on Genetics, Molecular Biology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Ying‐Qian Lu has authored 12 papers receiving a total of 240 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Genetics, 4 papers in Molecular Biology and 4 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Ying‐Qian Lu's work include Neurogenetic and Muscular Disorders Research (6 papers), Medical Imaging and Pathology Studies (3 papers) and Genetics and Neurodevelopmental Disorders (2 papers). Ying‐Qian Lu is often cited by papers focused on Neurogenetic and Muscular Disorders Research (6 papers), Medical Imaging and Pathology Studies (3 papers) and Genetics and Neurodevelopmental Disorders (2 papers). Ying‐Qian Lu collaborates with scholars based in China and United States. Ying‐Qian Lu's co-authors include Wan‐Jin Chen, En‐Lin Dong, Miao Zhao, Ning Wang, Hui‐Zhen Su, Shuang Wu, Xinxin Guo, J. He, Xiang Lin and Xiang‐Ping Yao and has published in prestigious journals such as Neuron, Gene and Oncotarget.

In The Last Decade

Ying‐Qian Lu

11 papers receiving 239 citations

Peers

Ying‐Qian Lu

SURGE

CMN

PRM

NEPHR

Hui‐Zhen Su China

En‐Lin Dong China

Claudia Stancanelli Italy

Eliana Marisa Ramos United States

Bianca M. de Graaf Netherlands

Kristina Martens Canada

Yukiko Hirano Japan

Caterina Marconi Italy

Stayko Sarafov Bulgaria

Paola Rinchetti Italy

Hui‐Zhen Su China

Ying‐Qian Lu

53 ×0.6

95 ×1.2

SURGE

79 ×1.1

CMN

91 ×1.4

PRM

87 ×1.6

NEPHR

Citations per year, relative to Ying‐Qian Lu Ying‐Qian Lu (= 1×) peers Hui‐Zhen Su

Countries citing papers authored by Ying‐Qian Lu

Since Specialization

Citations

This map shows the geographic impact of Ying‐Qian Lu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ying‐Qian Lu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ying‐Qian Lu more than expected).

Fields of papers citing papers by Ying‐Qian Lu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ying‐Qian Lu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ying‐Qian Lu. The network helps show where Ying‐Qian Lu may publish in the future.

Co-authorship network of co-authors of Ying‐Qian Lu

This figure shows the co-authorship network connecting the top 25 collaborators of Ying‐Qian Lu. A scholar is included among the top collaborators of Ying‐Qian Lu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ying‐Qian Lu. Ying‐Qian Lu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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12 of 12 papers shown

Li, Jiaqi, et al.. (2024). Clinical and functional characterization of a novel STUB1 mutation in a Chinese spinocerebellar ataxia 48 pedigree. Orphanet Journal of Rare Diseases. 19(1). 471–471.

Lu, Ying‐Qian, Jian‐Min Chen, Lin Han, et al.. (2022). Novel Intronic Mutations of TBK1 Promote Aberrant Splicing Modes in Amyotrophic Lateral Sclerosis. Frontiers in Molecular Neuroscience. 15. 691534–691534. 6 indexed citations

Guo, Xinxin, Hui‐Zhen Su, Xiaohuan Zou, et al.. (2019). Identification of SLC20A2 deletions in patients with primary familial brain calcification. Clinical Genetics. 96(1). 53–60. 11 indexed citations

Yao, Xiang‐Ping, Xuewen Cheng, Chong Wang, et al.. (2018). Biallelic Mutations in MYORG Cause Autosomal Recessive Primary Familial Brain Calcification. Neuron. 98(6). 1116–1123.e5. 97 indexed citations

Lu, Ying‐Qian, En‐Lin Dong, Xiao-Hong Lin, et al.. (2018). Generation of an integration-free induced pluripotent stem cell line, FJMUi001-A, from a hereditary spastic paraplegia patient carrying compound heterozygous p.P498L and p.R618W mutations in CAPN1 (SPG76). Stem Cell Research. 34. 101354–101354. 4 indexed citations

Dong, En‐Lin, Chong Wang, Shuang Wu, et al.. (2018). Clinical spectrum and genetic landscape for hereditary spastic paraplegias in China. Molecular Neurodegeneration. 13(1). 36–36. 55 indexed citations

Wu, Shuang, Chong Wang, En‐Lin Dong, et al.. (2018). c.835-5T>G Variant in SMN1 Gene Causes Transcript Exclusion of Exon 7 and Spinal Muscular Atrophy. Journal of Molecular Neuroscience. 65(2). 196–202. 6 indexed citations

Zhang, Qi‐Jie, Jin-Jing Li, Xiang Lin, et al.. (2017). Modeling the phenotype of spinal muscular atrophy by the direct conversion of human fibroblasts to motor neurons. Oncotarget. 8(7). 10945–10953. 21 indexed citations

Zhang, Qi‐Jie, Xiang Lin, Jin-Jing Li, et al.. (2017). Application of urine cells in drug intervention for spinal muscular atrophy. Experimental and Therapeutic Medicine. 14(3). 1993–1998. 5 indexed citations

10.

Lin, Xiang, Jin-Jing Li, Qi‐Jie Zhang, et al.. (2017). Modeling the differential phenotypes of spinal muscular atrophy with high-yield generation of motor neurons from human induced pluripotent stem cells. Oncotarget. 8(26). 42030–42042. 20 indexed citations

11.

Yao, Xiang‐Ping, Hui‐Zhen Su, Xinxin Guo, et al.. (2016). Mutation screening of PDGFB gene in Chinese population with primary familial brain calcification. Gene. 597. 17–22. 11 indexed citations

12.

Huang, Xiao, Yujuan Wang, Ying‐Qian Lu, et al.. (2002). [Screening of novel genes differentially expressed in human renal cell carcinoma by suppression subtractive hybridization].. PubMed. 21(10). 1065–9. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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