Yingmin Kuang

627 total citations
24 papers, 436 citations indexed

About

Yingmin Kuang is a scholar working on Molecular Biology, Cancer Research and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Yingmin Kuang has authored 24 papers receiving a total of 436 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 10 papers in Cancer Research and 7 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Yingmin Kuang's work include Cancer, Hypoxia, and Metabolism (6 papers), Bladder and Urothelial Cancer Treatments (4 papers) and Ferroptosis and cancer prognosis (4 papers). Yingmin Kuang is often cited by papers focused on Cancer, Hypoxia, and Metabolism (6 papers), Bladder and Urothelial Cancer Treatments (4 papers) and Ferroptosis and cancer prognosis (4 papers). Yingmin Kuang collaborates with scholars based in China and Togo. Yingmin Kuang's co-authors include Zihan Yi, Yuechun Zhu, Zhe Yang, Qiaoqiao Han, Huixin Yang, Lu Jiang, Yuqian Li, Lijuan Yang, Qiao Zhang and Yanling Wang and has published in prestigious journals such as Cell Death and Differentiation, Frontiers in Immunology and Medicine.

In The Last Decade

Yingmin Kuang

22 papers receiving 434 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yingmin Kuang China 12 273 182 72 48 46 24 436
Guanghou Fu China 11 294 1.1× 122 0.7× 89 1.2× 87 1.8× 38 0.8× 26 575
Yanghe Zhang China 9 215 0.8× 151 0.8× 43 0.6× 40 0.8× 48 1.0× 16 385
Cong Niu China 12 330 1.2× 80 0.4× 63 0.9× 37 0.8× 57 1.2× 19 460
Ajay Palagani United States 12 243 0.9× 118 0.6× 30 0.4× 73 1.5× 89 1.9× 23 520
Louise King Australia 10 250 0.9× 50 0.3× 82 1.1× 38 0.8× 63 1.4× 21 462
Julie-Ann Hulin Australia 13 297 1.1× 107 0.6× 32 0.4× 88 1.8× 14 0.3× 24 464
Arti Verma United States 13 289 1.1× 96 0.5× 83 1.2× 72 1.5× 55 1.2× 29 519
Lucia Natarelli Germany 15 342 1.3× 273 1.5× 25 0.3× 17 0.4× 102 2.2× 21 609

Countries citing papers authored by Yingmin Kuang

Since Specialization
Citations

This map shows the geographic impact of Yingmin Kuang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yingmin Kuang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yingmin Kuang more than expected).

Fields of papers citing papers by Yingmin Kuang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yingmin Kuang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yingmin Kuang. The network helps show where Yingmin Kuang may publish in the future.

Co-authorship network of co-authors of Yingmin Kuang

This figure shows the co-authorship network connecting the top 25 collaborators of Yingmin Kuang. A scholar is included among the top collaborators of Yingmin Kuang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yingmin Kuang. Yingmin Kuang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kuang, Yingmin, Yu Huang, Jiaxin Niu, et al.. (2025). MTCH2 regulates NRF2-mediated RRM1 expression to promote melanoma proliferation and dacarbazine insensitivity. Cell Death and Disease. 16(1). 268–268.
2.
Fang, Yun, Lu Jiang, Zihan Yi, et al.. (2025). NDUFS3 promotes proliferation via glucose metabolism reprogramming inducing AMPK phosphorylating PRPS1 to increase the purine nucleotide synthesis in melanoma. Cell Death and Differentiation. 32(12). 2193–2209. 1 indexed citations
3.
Wu, Na, Yun Fang, Junlin Xie, et al.. (2024). Molecular mechanisms of MAZ targeting up-regulation of NDUFS3 expression to promote malignant progression in melanoma. Communications Biology. 7(1). 1491–1491.
4.
Fang, Yun, Na Wu, Zhe Yang, et al.. (2024). NOD2 reduces the chemoresistance of melanoma by inhibiting the TYMS/PLK1 signaling axis. Cell Death and Disease. 15(10). 720–720. 3 indexed citations
5.
Fang, Yun, Feng Yu, Na Wu, et al.. (2022). Palmitic Acid Promotes Lung Metastasis of Melanomas via the TLR4/TRIF-Peli1-pNF-κB Pathway. Metabolites. 12(11). 1132–1132. 10 indexed citations
6.
Yu, Feng, Lijuan Yang, Zihan Yi, et al.. (2022). NRF2-directed PRPS1 upregulation to promote the progression and metastasis of melanoma. Frontiers in Immunology. 13. 989263–989263. 6 indexed citations
7.
Wang, Shujie, Wenjing Liu, Lifeng Wang, et al.. (2021). Overexpression of ERCC3 is associated with poor prognosis in patients with pancreatic cancer. Journal of Cancer. 12(9). 2550–2559. 6 indexed citations
8.
Yang, Hui, et al.. (2021). Overexpression CPT1A reduces lipid accumulation via PPARα/CD36 axis to suppress the cell proliferation in ccRCC. Acta Biochimica et Biophysica Sinica. 54(2). 220–231. 29 indexed citations
10.
Zhang, Qiao, Shujie Wang, Qiaoqiao Han, et al.. (2021). G6PD upregulates Cyclin E1 and MMP9 to promote clear cell renal cell carcinoma progression. International Journal of Medical Sciences. 19(1). 47–64. 10 indexed citations
11.
Chen, Jing, Yun Fang, Zihan Yi, et al.. (2020). LINC00511 as a prognostic biomarker for human cancers: a systematic review and meta-analysis. BMC Cancer. 20(1). 682–682. 11 indexed citations
12.
Kuang, Yingmin, et al.. (2020). Effect of miR-210 on Proliferation and Migration of Pancreatic Cancer Cells Through Regulating Runx3 Level. Journal of Biomaterials and Tissue Engineering. 10(12). 1827–1831. 2 indexed citations
13.
Zhang, Qiao, Zhe Yang, Qiaoqiao Han, et al.. (2020). NF-κB and pSTAT3 synergistically drive G6PD overexpression and facilitate sensitivity to G6PD inhibition in ccRCC. Cancer Cell International. 20(1). 483–483. 16 indexed citations
14.
Zhou, Meiling, Qiao Zhang, Kokouvi Kassegne, et al.. (2019). Garlic-derived bioactive compound S-allylcysteine inhibits cancer progression through diverse molecular mechanisms. Nutrition Research. 73. 1–14. 26 indexed citations
15.
Yi, Zihan, Lu Jiang, Meiling Zhou, et al.. (2019). Glutathione peroxidase 3 (GPX3) suppresses the growth of melanoma cells through reactive oxygen species (ROS)‐dependent stabilization of hypoxia‐inducible factor 1‐α and 2‐α. Journal of Cellular Biochemistry. 120(11). 19124–19136. 45 indexed citations
16.
Chen, Long, Huixin Yang, Zihan Yi, et al.. (2018). LncRNA GAS5 regulates redox balance and dysregulates the cell cycle and apoptosis in malignant melanoma cells. Journal of Cancer Research and Clinical Oncology. 145(3). 637–652. 54 indexed citations
17.
Zhang, Qiao, Zhe Yang, Qiaoqiao Han, et al.. (2017). Overexpression of G6PD Represents a Potential Prognostic Factor in Clear Cell Renal Cell Carcinoma. Journal of Cancer. 8(4). 665–673. 35 indexed citations
18.
Zhang, Qiao, Zhe Yang, Qiaoqiao Han, et al.. (2017). G6PD promotes renal cell carcinoma proliferation through positive feedback regulation of p-STAT3. Oncotarget. 8(65). 109043–109060. 38 indexed citations
20.
Kuang, Yingmin, et al.. (2007). [Changes of the immune cells, cytokines and growth hormone in teenager drug addicts].. PubMed. 23(9). 821–3. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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