Ye Cheng

3.1k total citations · 2 hit papers
59 papers, 2.1k citations indexed

About

Ye Cheng is a scholar working on Molecular Biology, Cancer Research and Epidemiology. According to data from OpenAlex, Ye Cheng has authored 59 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 15 papers in Cancer Research and 11 papers in Epidemiology. Recurrent topics in Ye Cheng's work include Adipokines, Inflammation, and Metabolic Diseases (7 papers), Cancer-related molecular mechanisms research (6 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (6 papers). Ye Cheng is often cited by papers focused on Adipokines, Inflammation, and Metabolic Diseases (7 papers), Cancer-related molecular mechanisms research (6 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (6 papers). Ye Cheng collaborates with scholars based in China, Germany and Bangladesh. Ye Cheng's co-authors include Weiwei Tang, Fan Wu, Qian Wang, Dawei Rong, Wenling Zhang, Xuehao Wang, Betty Zhang, Ziyi Chen, Florian P. Reiter and Enrico N. De Toni and has published in prestigious journals such as Journal of Agricultural and Food Chemistry, International Journal of Molecular Sciences and PLoS Biology.

In The Last Decade

Ye Cheng

54 papers receiving 2.1k citations

Hit Papers

The mechanisms of sorafenib resistance in hepatocellular ... 2020 2026 2022 2024 2020 2020 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ye Cheng China 19 1.2k 650 283 275 262 59 2.1k
Zhaodong Li China 21 1.2k 0.9× 332 0.5× 322 1.1× 218 0.8× 327 1.2× 50 2.4k
Wei Gong China 22 854 0.7× 359 0.6× 227 0.8× 324 1.2× 177 0.7× 35 2.1k
Soumitro Pal United States 26 1.3k 1.0× 473 0.7× 646 2.3× 493 1.8× 277 1.1× 53 2.4k
Kaishan Tao China 31 1.5k 1.2× 921 1.4× 646 2.3× 304 1.1× 194 0.7× 108 2.9k
Yoshinori Otsuki Japan 34 1.6k 1.3× 492 0.8× 368 1.3× 360 1.3× 170 0.6× 110 3.0k
Wen‐Sheng Wu Taiwan 22 1.0k 0.9× 360 0.6× 273 1.0× 181 0.7× 84 0.3× 41 1.8k
Chuan Liu China 28 1.3k 1.0× 603 0.9× 436 1.5× 254 0.9× 253 1.0× 121 2.3k
Kelvin K. Tsai Taiwan 27 1.4k 1.1× 492 0.8× 907 3.2× 307 1.1× 275 1.0× 51 2.6k
Xudong Tang China 21 776 0.6× 520 0.8× 390 1.4× 148 0.5× 179 0.7× 46 1.7k
Oluwaseun Adebayo Bamodu Taiwan 28 1.1k 0.9× 569 0.9× 468 1.7× 196 0.7× 208 0.8× 67 2.0k

Countries citing papers authored by Ye Cheng

Since Specialization
Citations

This map shows the geographic impact of Ye Cheng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ye Cheng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ye Cheng more than expected).

Fields of papers citing papers by Ye Cheng

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ye Cheng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ye Cheng. The network helps show where Ye Cheng may publish in the future.

Co-authorship network of co-authors of Ye Cheng

This figure shows the co-authorship network connecting the top 25 collaborators of Ye Cheng. A scholar is included among the top collaborators of Ye Cheng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ye Cheng. Ye Cheng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Deng, Shurong, Yang Wang, Ching-Wen Huang, et al.. (2024). Extracellular ATP activates H2O2 signaling to mitigate cadmium toxicity by restricting Cd2 + entry and triggering the antioxidant system in Arabidopsis. Plant Science. 352. 112362–112362. 1 indexed citations
3.
Cheng, Ye, et al.. (2024). HDL regulates the risk of cardiometabolic and inflammatory-related diseases: Focusing on cholesterol efflux capacity. International Immunopharmacology. 138. 112622–112622. 9 indexed citations
4.
Wang, Jiahong, et al.. (2024). LAPTM4B enhances the stemness of CD133+ liver cancer stem-like cells via WNT/β-catenin signaling. JHEP Reports. 7(4). 101306–101306.
5.
Wang, Meifeng, et al.. (2024). HDAC2 promotes autophagy-associated HCC malignant progression by transcriptionally activating LAPTM4B. Cell Death and Disease. 15(8). 593–593. 10 indexed citations
6.
Lin, Kai‐Hsin, et al.. (2024). Diverse Cytokines Secreted by Adipocyte in Linking Cardio-Metabolic Disorder and SLE. Frontiers in Bioscience-Landmark. 29(11). 373–373.
9.
Li, Xiao, Liangliang Wu, Zhiying Zheng, et al.. (2022). Tegaserod Maleate Inhibits Breast Cancer Progression and Enhances the Sensitivity of Immunotherapy. Journal of Oncology. 2022. 1–12. 4 indexed citations
10.
Li, Xiao, Guangshun Sun, Liangliang Wu, et al.. (2021). Upregulation of ADAR Promotes Breast Cancer Progression and Serves as a Potential Therapeutic Target. Journal of Oncology. 2021. 1–18. 12 indexed citations
11.
Wu, Fan, Guoqiang Sun, Wubin Zheng, et al.. (2021). circCORO1C promotes the proliferation and metastasis of hepatocellular carcinoma by enhancing the expression of PD‐L1 through NF‐κB pathway. Journal of Clinical Laboratory Analysis. 35(12). e24003–e24003. 17 indexed citations
12.
Rong, Dawei, Guangshun Sun, Fan Wu, et al.. (2021). Epigenetics: Roles and therapeutic implications of non-coding RNA modifications in human cancers. Molecular Therapy — Nucleic Acids. 25. 67–82. 65 indexed citations
13.
Jiang, Lei, et al.. (2021). The Ratio of CD86+/CD163+ Macrophages Predicts Postoperative Recurrence in Stage II-III Colorectal Cancer. Frontiers in Immunology. 12. 724429–724429. 12 indexed citations
14.
Liu, Wei, Jinghuan Huang, Feng Chen, et al.. (2021). MSC-derived small extracellular vesicles overexpressing miR-20a promoted the osteointegration of porous titanium alloy by enhancing osteogenesis via targeting BAMBI. Stem Cell Research & Therapy. 12(1). 348–348. 30 indexed citations
15.
Su, Xin, Ye Cheng, & Dong Chang. (2021). The Important Role of Leptin in Modulating the Risk of Dermatological Diseases. Frontiers in Immunology. 11. 593564–593564. 16 indexed citations
16.
Sun, Guoqiang, Dawei Rong, Zhouxiao Li, et al.. (2021). Role of Small Molecule Targeted Compounds in Cancer: Progress, Opportunities, and Challenges. Frontiers in Cell and Developmental Biology. 9. 694363–694363. 62 indexed citations
17.
Su, Xin, Guoming Zhang, Ye Cheng, & Bin Wang. (2021). New insights into ANGPTL8 in modulating the development of cardio-metabolic disorder diseases. Molecular Biology Reports. 48(4). 3761–3771. 2 indexed citations
18.
Su, Xin, Ye Cheng, & Bin Wang. (2021). ANGPTL8 in cardio-metabolic diseases. Clinica Chimica Acta. 519. 260–266. 5 indexed citations
19.
Tang, Weiwei, Ziyi Chen, Wenling Zhang, et al.. (2020). The mechanisms of sorafenib resistance in hepatocellular carcinoma: theoretical basis and therapeutic aspects. Signal Transduction and Targeted Therapy. 5(1). 87–87. 752 indexed citations breakdown →
20.
Tai, Yang, et al.. (2019). <p>Suppressing growth and invasion of human hepatocellular carcinoma cells by celecoxib through inhibition of cyclooxygenase-2</p>. Cancer Management and Research. Volume 11. 2831–2848. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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