Wen‐Sheng Wu

2.2k total citations · 1 hit paper
41 papers, 1.8k citations indexed

About

Wen‐Sheng Wu is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Wen‐Sheng Wu has authored 41 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 10 papers in Oncology and 7 papers in Cell Biology. Recurrent topics in Wen‐Sheng Wu's work include Protein Kinase Regulation and GTPase Signaling (6 papers), Liver physiology and pathology (5 papers) and PI3K/AKT/mTOR signaling in cancer (5 papers). Wen‐Sheng Wu is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (6 papers), Liver physiology and pathology (5 papers) and PI3K/AKT/mTOR signaling in cancer (5 papers). Wen‐Sheng Wu collaborates with scholars based in Taiwan, United States and Sudan. Wen‐Sheng Wu's co-authors include Jia‐Ru Wu, Chi‐Tan Hu, Chuan-Chu Cheng, Feng‐Sheng Wang, Yur‐Ren Kuo, Ming‐Che Lee, Ren-In You, Chao‐Cheng Huang, Kuender D. Yang and Chi‐Tan Hu and has published in prestigious journals such as PLoS ONE, Oncogene and Scientific Reports.

In The Last Decade

Wen‐Sheng Wu

40 papers receiving 1.8k citations

Hit Papers

The signaling mechanism of ROS in tumor progression 2006 2026 2012 2019 2006 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wen‐Sheng Wu Taiwan 22 1.0k 360 273 188 181 41 1.8k
Baskaran Govindarajan United States 21 1000 1.0× 221 0.6× 412 1.5× 159 0.8× 276 1.5× 26 1.8k
Mahmoud Aghaei Iran 26 1.1k 1.0× 296 0.8× 249 0.9× 184 1.0× 186 1.0× 116 2.0k
Katiuscia Dallaglio Italy 21 1.0k 1.0× 395 1.1× 644 2.4× 157 0.8× 300 1.7× 31 1.8k
Hyun Ju Kang South Korea 27 757 0.7× 271 0.8× 382 1.4× 90 0.5× 248 1.4× 96 2.1k
Huijuan Liu China 26 1.4k 1.3× 633 1.8× 329 1.2× 126 0.7× 220 1.2× 93 2.2k
Seok‐Il Hong South Korea 27 1.3k 1.3× 401 1.1× 354 1.3× 214 1.1× 186 1.0× 54 2.0k
Renata Colavitti Italy 17 1.3k 1.3× 324 0.9× 206 0.8× 181 1.0× 431 2.4× 21 2.3k
Siwanon Jirawatnotai Thailand 21 1.0k 1.0× 189 0.5× 494 1.8× 257 1.4× 139 0.8× 49 2.4k
Katarzyna Piwocka Poland 29 1.4k 1.3× 202 0.6× 370 1.4× 263 1.4× 288 1.6× 80 2.4k
Arkadiusz Surażyński Poland 26 951 0.9× 606 1.7× 715 2.6× 141 0.8× 143 0.8× 105 2.2k

Countries citing papers authored by Wen‐Sheng Wu

Since Specialization
Citations

This map shows the geographic impact of Wen‐Sheng Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wen‐Sheng Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wen‐Sheng Wu more than expected).

Fields of papers citing papers by Wen‐Sheng Wu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wen‐Sheng Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wen‐Sheng Wu. The network helps show where Wen‐Sheng Wu may publish in the future.

Co-authorship network of co-authors of Wen‐Sheng Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Wen‐Sheng Wu. A scholar is included among the top collaborators of Wen‐Sheng Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wen‐Sheng Wu. Wen‐Sheng Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lien, Te-Sheng, Der‐Shan Sun, Wen‐Sheng Wu, & Hsin‐Hou Chang. (2024). Dengue Envelope Protein as a Cytotoxic Factor Inducing Hemorrhage and Endothelial Cell Death in Mice. International Journal of Molecular Sciences. 25(19). 10858–10858. 2 indexed citations
2.
Li, Chi-Cheng, Songlin Liu, Der‐Shan Sun, et al.. (2024). Therapeutic Potential of Salvia miltiorrhiza Root Extract in Alleviating Cold-Induced Immunosuppression. International Journal of Molecular Sciences. 25(17). 9432–9432.
3.
Lien, Te-Sheng, Der‐Shan Sun, Wen‐Sheng Wu, & Hsin‐Hou Chang. (2023). Simulation of Hemorrhage Pathogenesis in Mice through Dual Stimulation with Dengue Envelope Protein Domain III-Coated Nanoparticles and Antiplatelet Antibody. International Journal of Molecular Sciences. 24(11). 9270–9270. 4 indexed citations
4.
Wu, Wen‐Sheng, Ruifang Chen, Chuan-Chu Cheng, et al.. (2022). Suppressing of Src–Hic-5–JNK–AKT Signaling Reduced GAPDH Expression for Preventing the Progression of HuCCT1 Cholangiocarcinoma. Pharmaceutics. 14(12). 2698–2698. 2 indexed citations
5.
Shiue, Chiou-Nan, Yen-Cheng Chen, Ming‐Che Lee, et al.. (2020). PKCδ mediates mitochondrial ROS generation and oxidation of HSP60 to relieve RKIP inhibition on MAPK pathway for HCC progression. Free Radical Biology and Medicine. 163. 69–87. 44 indexed citations
6.
Chang, Yo‐Chen, Chia-Wei Lin, Ming-Chu Hsieh, et al.. (2017). High mobility group B1 up-regulates angiogenic and fibrogenic factors in human retinal pigment epithelial ARPE-19 cells. Cellular Signalling. 40. 248–257. 27 indexed citations
7.
Wu, Jia‐Ru, Chi‐Tan Hu, Ren-In You, et al.. (2015). Hydrogen peroxide inducible clone-5 mediates reactive oxygen species signaling for hepatocellular carcinoma progression. Oncotarget. 6(32). 32526–32544. 23 indexed citations
8.
Hu, Chi‐Tan, et al.. (2015). PKCε-mediated c-Met endosomal processing directs fluctuant c-Met-JNK-paxillin signaling for tumor progression of HepG2. Cellular Signalling. 27(7). 1544–1555. 21 indexed citations
9.
10.
Wu, Wen‐Chuan, Yo‐Chen Chang, Kwou‐Yeung Wu, et al.. (2013). Pharmacological implications from the adhesion‐induced signaling profiles in cultured human retinal pigment epithelial cells. The Kaohsiung Journal of Medical Sciences. 30(1). 1–11. 8 indexed citations
11.
Hu, Chi‐Tan, et al.. (2010). Snail associates with EGR‐1 and SP‐1 to upregulate transcriptional activation of p15INK4b. FEBS Journal. 277(5). 1202–1218. 33 indexed citations
12.
Hu, Chi‐Tan, et al.. (2008). The transcriptional factor Snail simultaneously triggers cell cycle arrest and migration of human hepatoma HepG2. Journal of Biomedical Science. 15(3). 343–355. 28 indexed citations
13.
Wu, Wen‐Sheng, Jia‐Ru Wu, & Chi‐Tan Hu. (2008). Signal cross talks for sustained MAPK activation and cell migration: the potential role of reactive oxygen species. Cancer and Metastasis Reviews. 27(2). 303–314. 136 indexed citations
14.
Kuo, Yur‐Ren, Wen‐Sheng Wu, Feng‐Sheng Wang, et al.. (2007). Extracorporeal Shock Wave Enhanced Extended Skin Flap Tissue Survival via Increase of Topical Blood Perfusion and Associated with Suppression of Tissue Pro-Inflammation. Journal of Surgical Research. 143(2). 385–392. 76 indexed citations
16.
Wu, Wen‐Sheng, et al.. (2005). Activation of protein kinase C alpha is required for TPA-triggered ERK (MAPK) signaling and growth inhibition of human hepatoma cell HepG2. Journal of Biomedical Science. 12(2). 289–296. 30 indexed citations
18.
Wu, Wen‐Sheng & Hsue-Yin Hsu. (2001). Involvement of p-15INK4b and p-16INK4a Gene Expression in Saikosaponin a and TPA-Induced Growth Inhibition of HepG2 Cells. Biochemical and Biophysical Research Communications. 285(2). 183–187. 25 indexed citations
19.
Hsu, Shih‐Lan, Wen‐Sheng Wu, Yeu‐Sheng Tyan, & Chen‐Kung Chou. (1998). Retinoic acid-induced apoptosis is prevented by serum albumin and enhanced by Lipiodol in human hepatoma Hep3B cells. Cancer Letters. 129(2). 205–214. 19 indexed citations
20.
Kuo, Min‐Liang, et al.. (1993). EFfects of tannic acid on 12-O-tetradecanoylphorbol-13-acetate-induced protein kinase C activation in NIH 3T3 cells. Biochemical Pharmacology. 46(8). 1327–1332. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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