Yasuhiro Indo
About
Yasuhiro Indo is a scholar working on Clinical Biochemistry, Molecular Biology and Cellular and Molecular Neuroscience.
According to data from OpenAlex, Yasuhiro Indo has authored 55 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Clinical Biochemistry, 24 papers in Molecular Biology and 21 papers in Cellular and Molecular Neuroscience. Recurrent topics in Yasuhiro Indo's work include Metabolism and Genetic Disorders (25 papers), Hereditary Neurological Disorders (17 papers) and Biochemical and Molecular Research (12 papers). Yasuhiro Indo is often cited by papers focused on Metabolism and Genetic Disorders (25 papers), Hereditary Neurological Disorders (17 papers) and Biochemical and Molecular Research (12 papers). Yasuhiro Indo collaborates with scholars based in Japan, United States and Italy. Yasuhiro Indo's co-authors include Kay Tanaka, Ichiro Matsuda, Hiroshi Mitsubuchi, Yutaka Awaya, F. Endo, Kohji Ohta, Tomoyasu Kawano, Paul M. Coates, Mohammad A. Karim and Etsuo Naito and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Nature Genetics.
In The Last Decade
Yasuhiro Indo
55 papers receiving 2.1k citations
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late) cites · hero ref
| Name | h | Career | Trend | Papers | Cites | |||||
|---|---|---|---|---|---|---|---|---|---|---|
| Yasuhiro Indo Japan | 25 | 1.1k | 853 | 616 | 523 | 231 | 55 | 2.2k | ||
| A E Harding United Kingdom | 31 | 1.7k 1.5× | 1.3k 1.5× | 666 1.1× | 150 0.3× | 172 0.7× | 61 | 3.1k | ||
| Albena Jordanova Belgium | 32 | 1.9k 1.7× | 1.7k 2.0× | 247 0.4× | 296 0.6× | 570 2.5× | 87 | 3.6k | ||
| Hidefumi Ito Japan | 38 | 1.5k 1.3× | 838 1.0× | 114 0.2× | 785 1.5× | 441 1.9× | 145 | 3.9k | ||
| C. Hindelang France | 22 | 1.8k 1.6× | 1.1k 1.3× | 151 0.2× | 194 0.4× | 347 1.5× | 43 | 2.5k | ||
| Bart Dermaut Belgium | 31 | 1.9k 1.7× | 540 0.6× | 575 0.9× | 1.3k 2.5× | 232 1.0× | 64 | 3.3k | ||
| Cinzia Gellera Italy | 41 | 3.7k 3.3× | 2.3k 2.7× | 1.2k 1.9× | 439 0.8× | 260 1.1× | 155 | 5.5k | ||
| Benoît Funalot France | 26 | 1.1k 1.0× | 813 1.0× | 189 0.3× | 239 0.5× | 185 0.8× | 87 | 2.3k | ||
| Tommy L. Lewis United States | 20 | 1.8k 1.6× | 756 0.9× | 207 0.3× | 470 0.9× | 382 1.7× | 28 | 2.6k | ||
| Roman Chrast Switzerland | 35 | 1.8k 1.6× | 1.1k 1.3× | 132 0.2× | 560 1.1× | 538 2.3× | 70 | 3.3k | ||
| Alessandro Simonati Italy | 28 | 959 0.9× | 488 0.6× | 182 0.3× | 864 1.7× | 562 2.4× | 94 | 2.2k |
Countries citing papers authored by Yasuhiro Indo
Since
Specialization
Citations
This map shows the geographic impact of Yasuhiro Indo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yasuhiro Indo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yasuhiro Indo more than expected).
Fields of papers citing papers by Yasuhiro Indo
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Yasuhiro Indo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yasuhiro Indo. The network helps show where Yasuhiro Indo may publish in the future.
Co-authorship network of co-authors of Yasuhiro Indo
This figure shows the co-authorship network connecting the top 25 collaborators of Yasuhiro Indo. A scholar is included among the top collaborators of Yasuhiro Indo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yasuhiro Indo. Yasuhiro Indo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Indo, Yasuhiro. (2018). NGF-dependent neurons and neurobiology of emotions and feelings: Lessons from congenital insensitivity to pain with anhidrosis. Neuroscience & Biobehavioral Reviews. 87. 1–16.
2.
Indo, Yasuhiro. (2012). Nerve growth factor and the physiology of pain: lessons from congenital insensitivity to pain with anhidrosis. Clinical Genetics. 82(4). 341–350.
3.
Indo, Yasuhiro. (2010). Nerve growth factor, pain, itch and inflammation: lessons from congenital insensitivity to pain with anhidrosis. Expert Review of Neurotherapeutics. 10(11). 1707–1724.
4.
Indo, Yasuhiro. (2009). Nerve growth factor, interoception, and sympathetic neuron: Lesson from congenital insensitivity to pain with anhidrosis. Autonomic Neuroscience. 147(1-2). 3–8.
5.
Simonati, Alessandro, et al.. (2002). No mutation in the TRKA (NTRK1) gene encoding a receptor tyrosine kinase for nerve growth factor in a patient with hereditary sensory and autonomic neuropathy type V. Annals of Neurology. 52(2). 224–227.
6.
Indo, Yasuhiro. (2002). Genetics of congenital insensitivity to pain with anhidrosis (CIPA) or hereditary sensory and autonomic neuropathy type IV. Clinical, biological and molecular aspects of mutations in TRKA(NTRK1) gene encoding the receptor tyrosine kinase for nerve growth factor.. Clinical Autonomic Research. 12(7). I20–I32.
7.
Indo, Yasuhiro, Sek Mardy, Yuichi Miura, et al.. (2001). Congenital insensitivity to pain with anhidrosis (CIPA): Novel mutations of theTRKA (NTRK1) gene, a putative uniparental disomy, and a linkage of the mutantTRKA andPKLR genes in a family with CIPA and pyruvate kinase deficiency. Human Mutation. 18(4). 308–318.
8.
Indo, Yasuhiro. (2001). Molecular basis of congenital insensitivity to pain with anhidrosis (CIPA): Mutations and polymorphisms inTRKA (NTRK1) gene encoding the receptor tyrosine kinase for nerve growth factor. Human Mutation. 18(6). 462–471.
9.
Toscano, Evelina, Roberto Della Casa, Sek Mardy, et al.. (2000). Multisystem Involvement in Congenital Insensitivity to Pain with Anhidrosis (CIPA), a Nerve Growth Factor Receptor(Trk A)-Related Disorder. Neuropediatrics. 31(1). 39–41.
10.
Hiura, Makoto, Katsumi Torigoe, Osamu Numata, et al.. (2000). Complete paternal uniparental isodisomy for chromosome 1 revealed by mutation analyses of the TRKA (NTRK1) gene encoding a receptor tyrosine kinase for nerve growth factor in a patient with congenital insensitivity to pain with anhidrosis. Human Genetics. 107(3). 205–209.
11.
Miura, Yoshiko, Sek Mardy, Yutaka Awaya, et al.. (2000). Mutation and polymorphism analysis of the TRKA (NTRK1) gene encoding a high-affinity receptor for nerve growth factor in congenital insensitivity to pain with anhidrosis (CIPA) families. Human Genetics. 106(1). 116–124.
12.
Mitsubuchi, Hiroshi, Sek Mardy, Yuichi Miura, et al.. (1998). Molecular basis of intermittent maple syrup urine disease: novel mutations in the E2 gene of the branched-chain α-keto acid dehydrogenase complex. Journal of Human Genetics. 43(2). 91–100.
13.
Karim, Mohammad A., Kohji Ohta, Yoshihiro Jinno, et al.. (1996). Human ESP1/CRP2, a Member of the LIM Domain Protein Family: Characterization of the cDNA and Assignment of the Gene Locus to Chromosome 14q32.3. Genomics. 31(2). 167–176.
14.
Mitsubuchi, Hiroshi, Yasuhiro Indo, Kohji Ohta, et al.. (1994). Deficiency of the E1β subunit in the branched-chain α-keto acid dehydrogenase complex due to a single base substitution to the intron 5, resulting in two alternatively spliced mRNAs in patient with maple syrup urine disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1225(3). 317–325.
15.
Yamaguchi, Seiji, Yasuhiro Indo, Paul M. Coates, Takashi Hashimoto, & Kay Tanaka. (1993). Identification of Very-Long-Chain Acyl-CoA Dehydrogenase Deficiency in Three Patients Previously Diagnosed with Long-Chain Acyl-CoA Dehydrogenase Deficiency. Pediatric Research. 34(1). 111–113.
16.
Coates, Paul M., Yasuhiro Indo, D S Young, Daniel E. Hale, & Kay Tanaka. (1992). Immunochemical Characterization of Variant Medium-Chain Acyl-CoA Dehydrogenase in Fibroblasts from Patients with Medium-Chain Acyl-CoA Dehydrogenase Deficiency. Pediatric Research. 31(1). 34–38.
17.
Mitsubuchi, Hiroshi, Yoshitaka Nobukuni, Izumi Akaboshi, et al.. (1991). Maple syrup urine disease caused by a partial deletion in the inner E2 core domain of the branched chain alpha-keto acid dehydrogenase complex due to aberrant splicing. A single base deletion at a 5'-splice donor site of an intron of the E2 gene disrupts the consensus sequence in this region.. Journal of Clinical Investigation. 87(4). 1207–1211.
18.
Indo, Yasuhiro, Paul M. Coates, Daniel E. Hale, & Kay Tanaka. (1991). Immunochemical Characterization of Variant Long-Chain Acyl-CoA Dehydrogenase in Cultured Fibroblasts from Nine Patients with Long-Chain Acyl-CoA Dehydrogenase Deficiency. Pediatric Research. 30(3). 211–215.
19.
Naito, Etsuo, Yasuhiro Indo, & Kay Tanaka. (1990). Identification of two variant short chain acyl-coenzyme A dehydrogenase alleles, each containing a different point mutation in a patient with short chain acyl-coenzyme A dehydrogenase deficiency.. Journal of Clinical Investigation. 85(5). 1575–1582.
20.
Endo, Fumio, Kunihiko Motohara, Yasuhiro Indo, & Ichiro Matsuda. (1987). Immunochemical Studies of Human Prolidase with Monoclonal and Polyclonal Antibodies:Absence of the Subunit of Prolidase in Erythrocytes from a Patient with Prolidase Deficiency. Pediatric Research. 22(6). 627–633.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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