Yangyan Cui

538 total citations
9 papers, 305 citations indexed

About

Yangyan Cui is a scholar working on Molecular Biology, Surgery and Cancer Research. According to data from OpenAlex, Yangyan Cui has authored 9 papers receiving a total of 305 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 4 papers in Surgery and 4 papers in Cancer Research. Recurrent topics in Yangyan Cui's work include Bladder and Urothelial Cancer Treatments (3 papers), Epigenetics and DNA Methylation (3 papers) and Cancer, Hypoxia, and Metabolism (2 papers). Yangyan Cui is often cited by papers focused on Bladder and Urothelial Cancer Treatments (3 papers), Epigenetics and DNA Methylation (3 papers) and Cancer, Hypoxia, and Metabolism (2 papers). Yangyan Cui collaborates with scholars based in China and United States. Yangyan Cui's co-authors include Ruimin Huang, Jun Yan, Hongqian Guo, Xiaojing Huang, Dianzheng Zhang, Lan Shen, Junlong Zhuang, Xiaozhi Zhao, Qun Lu and Lin Yang and has published in prestigious journals such as Journal of Biological Chemistry, Cancer Research and Theranostics.

In The Last Decade

Yangyan Cui

9 papers receiving 303 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yangyan Cui China 7 214 157 49 49 46 9 305
Wen-Yu Lyu China 7 329 1.5× 165 1.1× 44 0.9× 57 1.2× 17 0.4× 13 416
Surong Zhao China 11 247 1.2× 198 1.3× 30 0.6× 80 1.6× 14 0.3× 25 364
Zongli Ding China 11 321 1.5× 214 1.4× 43 0.9× 59 1.2× 20 0.4× 18 386
Hailong Xia China 9 169 0.8× 113 0.7× 25 0.5× 59 1.2× 27 0.6× 27 335
Juan Xie China 9 175 0.8× 96 0.6× 23 0.5× 67 1.4× 18 0.4× 21 298
Yao Xiong China 8 292 1.4× 217 1.4× 23 0.5× 28 0.6× 13 0.3× 15 378
Yonghui Li China 10 222 1.0× 103 0.7× 27 0.6× 18 0.4× 42 0.9× 13 343
Erfei Chen China 12 265 1.2× 188 1.2× 46 0.9× 90 1.8× 24 0.5× 25 404

Countries citing papers authored by Yangyan Cui

Since Specialization
Citations

This map shows the geographic impact of Yangyan Cui's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yangyan Cui with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yangyan Cui more than expected).

Fields of papers citing papers by Yangyan Cui

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yangyan Cui. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yangyan Cui. The network helps show where Yangyan Cui may publish in the future.

Co-authorship network of co-authors of Yangyan Cui

This figure shows the co-authorship network connecting the top 25 collaborators of Yangyan Cui. A scholar is included among the top collaborators of Yangyan Cui based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yangyan Cui. Yangyan Cui is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Li, Meiqian, Yangyan Cui, Jiaxuan Li, et al.. (2024). SPOP downregulation promotes bladder cancer progression based on cancer cell-macrophage crosstalk via STAT3/CCL2/IL-6 axis and is regulated by VEZF1. Theranostics. 14(17). 6543–6559. 7 indexed citations
2.
Cui, Yangyan, et al.. (2022). Identification of hexosamine biosynthesis pathway as a novel prognostic signature and its correlation with immune infiltration in bladder cancer. Frontiers in Molecular Biosciences. 9. 1009168–1009168. 1 indexed citations
3.
Yan, Yi‐Lin, Zhen Zhu, Yangyan Cui, et al.. (2020). Downregulation of TET1 Promotes Bladder Cancer Cell Proliferation and Invasion by Reducing DNA Hydroxymethylation of AJAP1. Frontiers in Oncology. 10. 667–667. 23 indexed citations
4.
Zhuang, Junlong, Lan Shen, Lin Yang, et al.. (2017). TGFβ1 Promotes Gemcitabine Resistance through Regulating the LncRNA-LET/NF90/miR-145 Signaling Axis in Bladder Cancer. Theranostics. 7(12). 3053–3067. 125 indexed citations
5.
Han, Yangyang, Weiwei Huang, Dandan Liu, et al.. (2017). Triptolide Inhibits the AR Signaling Pathway to Suppress the Proliferation of Enzalutamide Resistant Prostate Cancer Cells. Theranostics. 7(7). 1914–1927. 49 indexed citations
6.
Yan, Jun, Qing Zhang, Wei Zhao, et al.. (2015). Abstract 3517: Honokiol inhibits bladder tumor growth by suppressing EZH2/miR-143 axis. Cancer Research. 75(15_Supplement). 3517–3517. 1 indexed citations
7.
Zhang, Qing, Wei Zhao, Junlong Zhuang, et al.. (2015). Honokiol inhibits bladder tumor growth by suppressing EZH2/miR-143 axis. Oncotarget. 6(35). 37335–37348. 39 indexed citations
8.
Zhao, Wei, Cunjie Chang, Yangyan Cui, et al.. (2014). Steroid Receptor Coactivator-3 Regulates Glucose Metabolism in Bladder Cancer Cells through Coactivation of Hypoxia Inducible Factor 1α. Journal of Biological Chemistry. 289(16). 11219–11229. 48 indexed citations
9.
Chang, Cunjie, Wei Zhao, Bingxian Xie, et al.. (2013). Pao Pereira Extract Suppresses Castration-Resistant Prostate Cancer Cell Growth, Survival, and Invasion Through Inhibition of NFκB Signaling. Integrative Cancer Therapies. 13(3). 249–258. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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