Y. Matsuzaki

660 citations
23 papers · 538 indexed · h-index 12
Topics
Cancer-related Molecular Pathways (11 papers)Histone Deacetylase Inhibitors Research (7 papers)Epigenetics and DNA Methylation (6 papers)
Partner nations
JapanUnited States

In The Last Decade

Y. Matsuzaki

23 papers receiving 525 citations

Peers

Y. Matsuzaki
Comparison fields: 5 of 72
  • Molecular Biology 336
  • Oncology 146
  • Plant Science 99
  • Cancer Research 60
  • Pathology and Forensic Medicine 53
Replace Munju Cho with:
Munju Cho South Korea
Roberta R. Ruela-de-Sousa Brazil
Xiaocheng Cao China
Yunlong Shan China
Rodolphe Filomenko France
Eiko Shibata Japan
Weili Min China
Kangdong Liu China
Ma'in Y. Maitah United States
Mariano Cárdenas Argentina
Y. Matsuzaki relative to Munju Cho South Korea Munju Cho's profile →
Citations per field
00.5×1.5×2.0×
Munju Cho · 1×
Citations per year

Countries citing papers authored by Y. Matsuzaki

Since Specialization
Citations

This map shows the geographic impact of Y. Matsuzaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Y. Matsuzaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Y. Matsuzaki more than expected).

Fields of papers citing papers by Y. Matsuzaki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Y. Matsuzaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Y. Matsuzaki. The network helps show where Y. Matsuzaki may publish in the future.

Co-authorship network of co-authors of Y. Matsuzaki

This figure shows the co-authorship network connecting the top 25 collaborators of Y. Matsuzaki. A scholar is included among the top collaborators of Y. Matsuzaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Y. Matsuzaki. Y. Matsuzaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 1
2
ABNORMALITY OF SERUM LIPIDS IN THE HCV CARRIERS IN EPIDEMIOLOGICAL SURVEY OF JAPANESE LARGE COHORT
3
3 46
4 118
5 20
6 35
7 26
8 18
9 11
10 3
11 4
12 24
13 79
14 14
15 20
16 6
17 62
18 9
19 27
20 9

About Y. Matsuzaki

Y. Matsuzaki is a scholar working on Oncology, Hepatology and Biotechnology, having authored 23 papers that have together received 538 indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (11 papers), Histone Deacetylase Inhibitors Research (7 papers) and Epigenetics and DNA Methylation (6 papers). The work is most often cited by research in Oncology (146 citations), Molecular Biology (336 citations) and Cancer Research (60 citations). Y. Matsuzaki has collaborated with scholars based in Japan and United States. Frequent co-authors include Toshiyuki Sakai, Tomoya Yokota, Toshiaki Hitomi, Makoto Koyama, Akiyoshi Nishikawa, Hoyoku Nishino, Frédérique Zindy, Martine F. Roussel, Yusuke Okuyama and Toshihiko Osawa. Their work appears in journals such as Journal of Biological Chemistry, Gastroenterology and Hepatology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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