Xuxiao He

991 total citations · 2 hit papers
11 papers, 532 citations indexed

About

Xuxiao He is a scholar working on Molecular Biology, Cancer Research and Epidemiology. According to data from OpenAlex, Xuxiao He has authored 11 papers receiving a total of 532 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 7 papers in Cancer Research and 3 papers in Epidemiology. Recurrent topics in Xuxiao He's work include Cancer, Hypoxia, and Metabolism (6 papers), Autophagy in Disease and Therapy (2 papers) and Mitochondrial Function and Pathology (2 papers). Xuxiao He is often cited by papers focused on Cancer, Hypoxia, and Metabolism (6 papers), Autophagy in Disease and Therapy (2 papers) and Mitochondrial Function and Pathology (2 papers). Xuxiao He collaborates with scholars based in China and United States. Xuxiao He's co-authors include Zhimin Lu, Hongfei Jiang, Shudi Luo, Guijun Liu, Xiaoming Jiang, Dong Guo, Haiyan He, Shan Li, Yongzhen Tao and Qingang Wu and has published in prestigious journals such as Molecular Cell, Hepatology and Cell Metabolism.

In The Last Decade

Xuxiao He

10 papers receiving 529 citations

Hit Papers

Aerobic glycolysis promotes tumor immune evasion by hexok... 2022 2026 2023 2024 2022 2024 50 100 150 200

Peers

Xuxiao He
Ji-Hak Jeong South Korea
Zunqi Hu China
Omar Rahal United States
Yuting He China
Xuxiao He
Citations per year, relative to Xuxiao He Xuxiao He (= 1×) peers Weihua Zhan

Countries citing papers authored by Xuxiao He

Since Specialization
Citations

This map shows the geographic impact of Xuxiao He's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xuxiao He with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xuxiao He more than expected).

Fields of papers citing papers by Xuxiao He

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xuxiao He. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xuxiao He. The network helps show where Xuxiao He may publish in the future.

Co-authorship network of co-authors of Xuxiao He

This figure shows the co-authorship network connecting the top 25 collaborators of Xuxiao He. A scholar is included among the top collaborators of Xuxiao He based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xuxiao He. Xuxiao He is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Luo, Shudi, Xiaoming Jiang, Zheng Wang, et al.. (2025). Inhibition of Tumor Lipogenesis and Growth by Peptide‐Based Targeting of SREBP Activation. Advanced Science. 12(45). e08111–e08111.
2.
He, Xuxiao, Deyu Chen, Guijun Liu, et al.. (2025). PI3Kβ functions as a protein kinase to promote cellular protein O-GlcNAcylation and acetyl-CoA production for tumor growth. Molecular Cell. 85(7). 1411–1425.e8. 4 indexed citations
3.
Liu, Guijun, Xuxiao He, Gaoxiang Zhao, & Zhimin Lu. (2024). Complement regulation in tumor immune evasion. Seminars in Immunology. 76. 101912–101912. 8 indexed citations
4.
Meng, Ying, Dong Guo, Liming Lin, et al.. (2024). Glycolytic enzyme PFKL governs lipolysis by promoting lipid droplet–mitochondria tethering to enhance β-oxidation and tumor cell proliferation. Nature Metabolism. 6(6). 1092–1107. 49 indexed citations breakdown →
5.
Li, Min, Zheng Wang, Jingjing Tao, et al.. (2024). Fructose-1,6-bisphosphatase 1 dephosphorylates and inhibits TERT for tumor suppression. Nature Chemical Biology. 20(11). 1505–1513. 17 indexed citations
6.
Guo, Dong, Yingying Tong, Xiaoming Jiang, et al.. (2022). Aerobic glycolysis promotes tumor immune evasion by hexokinase2-mediated phosphorylation of IκBα. Cell Metabolism. 34(9). 1312–1324.e6. 246 indexed citations breakdown →
7.
Liu, Guijun, Ningning Wang, Cunzhen Zhang, et al.. (2021). Fructose‐1,6‐Bisphosphate Aldolase B Depletion Promotes Hepatocellular Carcinogenesis Through Activating Insulin Receptor Signaling and Lipogenesis. Hepatology. 74(6). 3037–3055. 35 indexed citations
8.
He, Xuxiao, Li Min, Hongming Yu, et al.. (2020). Loss of hepatic aldolase B activates Akt and promotes hepatocellular carcinogenesis by destabilizing the Aldob/Akt/PP2A protein complex. PLoS Biology. 18(12). e3000803–e3000803. 47 indexed citations
9.
Liu, Guijun, Aimin Shi, Ningning Wang, et al.. (2020). Polyphenolic Proanthocyanidin-B2 suppresses proliferation of liver cancer cells and hepatocellular carcinogenesis through directly binding and inhibiting AKT activity. Redox Biology. 37. 101701–101701. 51 indexed citations
10.
Wang, Ningning, Hongzhi Liu, Guijun Liu, et al.. (2020). Yeast β-D-glucan exerts antitumour activity in liver cancer through impairing autophagy and lysosomal function, promoting reactive oxygen species production and apoptosis. Redox Biology. 32. 101495–101495. 63 indexed citations
11.
Jiang, Yabo, Yongzhen Tao, Xiuping Zhang, et al.. (2020). Loss of STAT5A promotes glucose metabolism and tumor growth through miRNA‐23a‐AKT signaling in hepatocellular carcinoma. Molecular Oncology. 15(2). 710–724. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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