Xuan Shang

1.2k total citations
41 papers, 615 citations indexed

About

Xuan Shang is a scholar working on Genetics, Hematology and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Xuan Shang has authored 41 papers receiving a total of 615 indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Genetics, 16 papers in Hematology and 11 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Xuan Shang's work include Hemoglobinopathies and Related Disorders (26 papers), Iron Metabolism and Disorders (13 papers) and Prenatal Screening and Diagnostics (9 papers). Xuan Shang is often cited by papers focused on Hemoglobinopathies and Related Disorders (26 papers), Iron Metabolism and Disorders (13 papers) and Prenatal Screening and Diagnostics (9 papers). Xuan Shang collaborates with scholars based in China and United States. Xuan Shang's co-authors include Xiangmin Xu, Xiangmin Xu, Xiaofeng Wei, Fu Xiong, Ren Cai, Manna Sun, Xinhua Zhang, Jiwu Lou, Yuqiu Zhou and Lingwen Zeng and has published in prestigious journals such as The Journal of Organic Chemistry, Gene and Sensors and Actuators B Chemical.

In The Last Decade

Xuan Shang

39 papers receiving 598 citations

Peers

Xuan Shang
Duantida Songdej Thailand
C Altay United States
Josiane Bardakdjian‐Michau France
P. Pravatmuang Thailand
G. V. Sciarratta Italy
Guillermo Drelichman Argentina
G. Mérault France
Amy Y. Y. Chan Hong Kong
Olufolake Adisa United States
VK Kalra India
Duantida Songdej Thailand
Xuan Shang
Citations per year, relative to Xuan Shang Xuan Shang (= 1×) peers Duantida Songdej

Countries citing papers authored by Xuan Shang

Since Specialization
Citations

This map shows the geographic impact of Xuan Shang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xuan Shang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xuan Shang more than expected).

Fields of papers citing papers by Xuan Shang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xuan Shang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xuan Shang. The network helps show where Xuan Shang may publish in the future.

Co-authorship network of co-authors of Xuan Shang

This figure shows the co-authorship network connecting the top 25 collaborators of Xuan Shang. A scholar is included among the top collaborators of Xuan Shang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xuan Shang. Xuan Shang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhu, Zihan, Yan Zhang, Lisha Luo, et al.. (2025). Salivary acid-modified carbon dots loaded with paclitaxel for imaging-guided combination treatment of breast cancer. Biomaterials Science. 13(15). 4168–4179.
2.
Wang, Ke, Yu Han, Haifeng Lv, et al.. (2024). Environment-friendly glass with high refractive index and radiation resistance. Ceramics International. 51(2). 1978–1987. 3 indexed citations
3.
Wei, Xiaofeng, et al.. (2024). SUPT5H mutations associated with elevation of Hb A2 level: Identification of two novel variants and literature review. Gene. 908. 148294–148294. 3 indexed citations
4.
Zhong, Long‐Jin, Xuan Shang, Hui Chen, et al.. (2024). Three‐Component Alkylsulfonylation of 1‐Acryloyl‐2‐Cyanoindoles with Hantzsch Esters through Sulfur Dioxide Insertion. European Journal of Organic Chemistry. 28(6). 1 indexed citations
5.
Guan, Yuqing, Wei Li, Wanying Lin, et al.. (2023). TRIM32 biallelic defects cause limb-girdle muscular dystrophy R8: identification of two novel mutations and investigation of genotype–phenotype correlation. Skeletal Muscle. 13(1). 10–10. 4 indexed citations
6.
Zhang, Yanxia, et al.. (2023). A novel gain‐of‐function PIP4K2A mutation elevates the expression of β‐globin and aggravates the severity of α‐thalassemia. British Journal of Haematology. 202(5). 1018–1023. 1 indexed citations
7.
Zhang, Qianqian, Peng Lin, Aiping Mao, et al.. (2023). An unusual case of thalassemia intermedia with inheritable complex repeats detected by single-molecule optical mapping. Haematologica. 109(3). 1000–1006. 2 indexed citations
8.
Wei, Xiaofeng, et al.. (2022). A novel splicing mutation of ANK1 is associated with phenotypic heterogeneity of hereditary spherocytosis in a Chinese family. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1869(1). 166595–166595. 3 indexed citations
9.
Ye, Yuhua, et al.. (2021). TEA domain transcription factor 4 modulates repression of fetal haemoglobin by direct binding to the γ‐globin gene promoters. British Journal of Haematology. 195(5). 764–769. 2 indexed citations
10.
11.
Guan, Yuqing, Yanxia Zhang, Xin‐Ming Shen, et al.. (2021). Charcot–Marie–Tooth Disease With Episodic Rhabdomyolysis Due to Two Novel Mutations in the β Subunit of Mitochondrial Trifunctional Protein and Effective Response to Modified Diet Therapy. Frontiers in Neurology. 12. 694966–694966. 1 indexed citations
12.
Shang, Xuan & Xiangmin Xu. (2016). Update in the genetics of thalassemia: What clinicians need to know. Best Practice & Research Clinical Obstetrics & Gynaecology. 39. 3–15. 66 indexed citations
13.
Hu, Lingling, Xuan Shang, Ren Cai, et al.. (2016). Two novel copy number variations involving the α-globin gene cluster on chromosome 16 cause thalassemia in two Chinese families. Molecular Genetics and Genomics. 291(3). 1443–1450. 22 indexed citations
14.
Huang, Jiwei, Xuan Shang, Ying Zhao, et al.. (2013). A novel fusion gene and a common α0-thalassemia deletion cause hemoglobin H disease in a Chinese family. Blood Cells Molecules and Diseases. 51(1). 31–34. 13 indexed citations
15.
Xiong, Fu, Qian Li, Cuimei Zhang, et al.. (2012). Analyses of GATA4, NKX2.5, and TFAP2B genes in subjects from southern China with sporadic congenital heart disease. Cardiovascular Pathology. 22(2). 141–145. 33 indexed citations
16.
Huang, Yeqing, Xuan Shang, Fu Xiong, et al.. (2011). Mutation analysis of 73 southern Chinese Wilson's disease patients: identification of 10 novel mutations and its clinical correlation. Journal of Human Genetics. 56(9). 660–665. 32 indexed citations
17.
Wei, Xiaofeng, et al.. (2010). Molecular characterization of a novel 27.6-kb deletion causing α+ thalassemia in a Chinese family. Annals of Hematology. 90(1). 17–22. 18 indexed citations
18.
Chen, Wanqun, Xinhua Zhang, Xuan Shang, et al.. (2010). The molecular basis of beta-thalassemia intermedia in southern China: genotypic heterogeneity and phenotypic diversity. BMC Medical Genetics. 11(1). 31–31. 54 indexed citations
19.
Wang, Zhipeng, Ying Li, Xuan Shang, et al.. (2010). Analysis of α-hemoglobin-stabilizing protein (AHSP) gene as a genetic modifier to the phenotype of β-thalassemia in Southern China. Blood Cells Molecules and Diseases. 45(2). 128–132. 14 indexed citations
20.
Xie, Jiansheng, et al.. (2010). Hemoglobin H disease due to a de novo mutation at the α2-globin gene and an inherited common α-thalassemia deletion found in a Chinese boy. Blood Cells Molecules and Diseases. 45(3). 223–226. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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