Xiong‐Jian Luo

3.4k total citations
89 papers, 2.0k citations indexed

About

Xiong‐Jian Luo is a scholar working on Genetics, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, Xiong‐Jian Luo has authored 89 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 61 papers in Genetics, 50 papers in Molecular Biology and 10 papers in Cognitive Neuroscience. Recurrent topics in Xiong‐Jian Luo's work include Genetic Associations and Epidemiology (53 papers), Genetics and Neurodevelopmental Disorders (21 papers) and Epigenetics and DNA Methylation (20 papers). Xiong‐Jian Luo is often cited by papers focused on Genetic Associations and Epidemiology (53 papers), Genetics and Neurodevelopmental Disorders (21 papers) and Epigenetics and DNA Methylation (20 papers). Xiong‐Jian Luo collaborates with scholars based in China, Hong Kong and United States. Xiong‐Jian Luo's co-authors include Jiewei Liu, Ming Li, Yong‐Gang Yao, Yongxia Huo, Yong Wu, Xiao Xiao, Bing Su, Shiwu Li, Xiaoyan Li and Xingyu Wang and has published in prestigious journals such as Nucleic Acids Research, Nature Communications and Journal of Neuroscience.

In The Last Decade

Xiong‐Jian Luo

86 papers receiving 1.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Xiong‐Jian Luo China 27 1.1k 785 257 199 174 89 2.0k
Guang He China 28 1.2k 1.1× 499 0.6× 106 0.4× 145 0.7× 282 1.6× 138 2.5k
Shea J. Andrews United States 21 892 0.8× 331 0.4× 46 0.2× 70 0.4× 99 0.6× 47 1.9k
Barbara Robertson United States 23 783 0.7× 152 0.2× 75 0.3× 75 0.4× 67 0.4× 53 1.8k
Edward B. Holson United States 29 1.9k 1.7× 313 0.4× 34 0.1× 241 1.2× 196 1.1× 45 3.1k
Marc B. Cox United States 27 1.8k 1.6× 248 0.3× 100 0.4× 20 0.1× 136 0.8× 50 2.8k
Kathleen Van Craenenbroeck Belgium 23 1.1k 1.0× 198 0.3× 39 0.2× 86 0.4× 51 0.3× 50 1.8k
Miklós Sántha Hungary 24 941 0.9× 291 0.4× 29 0.1× 278 1.4× 106 0.6× 57 2.4k
Douglas M. Ruderfer United States 24 2.5k 2.3× 2.5k 3.2× 30 0.1× 585 2.9× 110 0.6× 62 4.7k
Dominik Feuerbach Switzerland 31 1.4k 1.3× 111 0.1× 92 0.4× 95 0.5× 223 1.3× 75 2.4k

Countries citing papers authored by Xiong‐Jian Luo

Since Specialization
Citations

This map shows the geographic impact of Xiong‐Jian Luo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xiong‐Jian Luo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xiong‐Jian Luo more than expected).

Fields of papers citing papers by Xiong‐Jian Luo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xiong‐Jian Luo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xiong‐Jian Luo. The network helps show where Xiong‐Jian Luo may publish in the future.

Co-authorship network of co-authors of Xiong‐Jian Luo

This figure shows the co-authorship network connecting the top 25 collaborators of Xiong‐Jian Luo. A scholar is included among the top collaborators of Xiong‐Jian Luo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xiong‐Jian Luo. Xiong‐Jian Luo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Chen, Rui, Zhaowei Teng, Junyang Wang, et al.. (2025). Cross-ancestry genome-wide association study and systems-level integrative analyses implicate new risk genes and therapeutic targets for depression. Nature Human Behaviour. 9(4). 806–823. 3 indexed citations
4.
Luo, Ming‐Cheng, et al.. (2024). METTL5 enhances the mRNA stability of TPRKB through m6A modification to facilitate the aggressive phenotypes of hepatocellular carcinoma cell. Experimental Cell Research. 442(1). 114219–114219. 5 indexed citations
5.
Fan, Fengfeng, Mingxing Cheng, Nannan Li, et al.. (2023). A transposon-derived gene family regulates heading date in rice. Plant Science. 337. 111871–111871. 1 indexed citations
6.
Song, Meng, et al.. (2023). Proteome-wide Mendelian randomization reveals the causal effects of immune-related plasma proteins on psychiatric disorders. Human Genetics. 142(6). 809–818. 5 indexed citations
7.
Li, Wenqiang, Rui Chen, Jiewei Liu, et al.. (2023). Genome-wide meta-analysis, functional genomics and integrative analyses implicate new risk genes and therapeutic targets for anxiety disorders. Nature Human Behaviour. 8(2). 361–379. 21 indexed citations
8.
Zhang, Chengcheng, Xiaojing Li, Liansheng Zhao, et al.. (2023). Brain transcriptome-wide association study implicates novel risk genes underlying schizophrenia risk. Psychological Medicine. 53(14). 6867–6877. 7 indexed citations
9.
Wang, Junyang, Shiwu Li, Xiaoyan Li, et al.. (2022). Functional variant rs2270363 on 16p13.3 confers schizophrenia risk by regulating NMRAL1. Brain. 145(7). 2569–2585. 7 indexed citations
10.
Zhang, Chengcheng, Xiaojing Li, Liansheng Zhao, et al.. (2022). Comprehensive and integrative analyses identify TYW5 as a schizophrenia risk gene. BMC Medicine. 20(1). 169–169. 7 indexed citations
11.
Li, Shiwu, Yifan Li, Rui Chen, et al.. (2022). The schizophrenia-associated missense variant rs13107325 regulates dendritic spine density. Translational Psychiatry. 12(1). 361–361. 7 indexed citations
12.
Liu, Jiewei, Wenqiang Li, Yongfeng Yang, et al.. (2021). Genome wide association study identifies four loci for early onset schizophrenia. Translational Psychiatry. 11(1). 248–248. 16 indexed citations
13.
Luo, Zhi-Feng, et al.. (2021). miR-23b Attenuates LPS-Induced Inflammatory Responses in Acute Lung Injury via Inhibition of HDAC2. Biochemical Genetics. 59(2). 604–616. 5 indexed citations
14.
Li, Yifan, Wenqiang Li, Yongfeng Yang, et al.. (2021). A missense variant in NDUFA6 confers schizophrenia risk by affecting YY1 binding and NAGA expression. Molecular Psychiatry. 26(11). 6896–6911. 17 indexed citations
15.
Song, Meng, Jiewei Liu, Yongfeng Yang, et al.. (2021). Genome-Wide Meta-Analysis Identifies Two Novel Risk Loci for Epilepsy. Frontiers in Neuroscience. 15. 15 indexed citations
16.
Li, Shiwu, Yifan Li, Xiaoyan Li, et al.. (2020). Regulatory mechanisms of major depressive disorder risk variants. Molecular Psychiatry. 25(9). 1926–1945. 42 indexed citations
17.
Li, Shiwu, Wanli Zeng, Xiaoyan Li, et al.. (2019). Integration of GWAS and brain eQTL identifies FLOT1 as a risk gene for major depressive disorder. Neuropsychopharmacology. 44(9). 1542–1551. 30 indexed citations
18.
Liu, Jiewei, Yin Mo, Tian Ge, et al.. (2015). Allelic variation at 5-HTTLPR is associated with brain morphology in a Chinese population. Psychiatry Research. 226(1). 399–402. 3 indexed citations
19.
Huang, Liang, Liang Feng, Xiong‐Jian Luo, et al.. (2013). Bhlhb5 is required for the subtype development of retinal amacrine and bipolar cells in mice. Developmental Dynamics. 243(2). 279–289. 23 indexed citations
20.
Li, Hongyang, Zengqiang Gao, Ling Kang, et al.. (2006). TarFisDock: a web server for identifying drug targets with docking approach. Nucleic Acids Research. 34(Web Server). W219–W224. 301 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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