Xinliang Ming
Impact in
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- Cancer-related molecular mechanisms research
- MicroRNA in disease regulation
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- Hepatocellular Carcinoma Treatment and Prognosis
Papers in
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- Circular RNAs in diseases 2
- Inflammasome and immune disorders 1
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- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 2
- Immunotherapy and Immune Responses 1
- Co-authors
- Jiancheng Tu (5 shared papers)Shuo Li (2 shared papers)Sanyun Wu (1 shared paper)Yalan Yu (1 shared paper)Ping Luo (1 shared paper)Xuelan Zuo (1 shared paper)Xuefang Liu (4 shared papers)Nandi Li (3 shared papers)
- Journals
- Technology in Cancer Research & Treatment (2 papers)Epigenomics (1 paper)ACS Nano (1 paper)Analytical Cellular Pathology (1 paper)Pathology & Oncology Research (1 paper)
- Partner nations
- ChinaNew ZealandUnited States
In The Last Decade
Xinliang Ming
11 papers receiving 296 citations
Peers
Comparison fields: 5 of 53
- Cancer Research 96
- Hepatology 43
- Immunology 38
- Molecular Biology 122
- Oncology 40
Countries citing papers authored by Xinliang Ming
This map shows the geographic impact of Xinliang Ming's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xinliang Ming with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xinliang Ming more than expected).
Fields of papers citing papers by Xinliang Ming
This network shows the impact of papers produced by Xinliang Ming. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xinliang Ming. The network helps show where Xinliang Ming may publish in the future.
Co-authors
The 25 scholars most cited alongside Xinliang Ming, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 163 | |
| 2 | 2018 | 23 | |
| 3 | 2024 | 21 | |
| 4 | 2023 | 19 | |
| 5 | 2019 | 18 | |
| 6 | 2019 | 17 | |
| 7 | 2018 | 16 | |
| 8 | 2020 | 12 | |
| 9 | 2019 | 8 | |
| 10 | 2025 | 2 | |
| 11 | 2019 | 1 | |
| 12 | 2026 | 0 | |
| 13 | 2025 | 0 |
About Xinliang Ming
Xinliang Ming is a scholar working on Molecular Biology, Immunology, Cancer Research, Oncology and Epidemiology, having authored 13 papers that have together received 300 indexed citations. Recurring topics across this work include Cancer-related molecular mechanisms research (3 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (2 papers), Circular RNAs in diseases (2 papers), Hepatocellular Carcinoma Treatment and Prognosis (1 paper), Ferroptosis and cancer prognosis (1 paper), Telomeres, Telomerase, and Senescence (1 paper), Immunotherapy and Immune Responses (1 paper) and Inflammasome and immune disorders (1 paper). The work is most often cited by research in Cancer Research (96 citations), Hepatology (43 citations), Immunology (38 citations), Molecular Biology (122 citations) and Oncology (40 citations). Xinliang Ming has collaborated with scholars based in China, New Zealand and United States. Frequent co-authors include Jiancheng Tu, Shuo Li, Sanyun Wu, Yalan Yu, Ping Luo, Xuelan Zuo, Xuefang Liu, Nandi Li, Jicheng Wu and Ben Wang. Their work appears in journals such as Technology in Cancer Research & Treatment, Epigenomics, ACS Nano, Analytical Cellular Pathology and Pathology & Oncology Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.