Xiaolei Shi
- Molecular Biology
- Cancer Research top 5%
- Genetics top 10%
- Hepatology top 5%
- Surgery
- Co-authors
- Haozhen RenJun LiLongcheng ShangYuan ZhouJianfei HuangBo DaiJinglin WangYitao Ding
- Topics
- Ubiquitin and proteasome pathways (4 papers)Autophagy in Disease and Therapy (3 papers)Liver physiology and pathology (2 papers)
- Cited by
- Cancer ResearchHepatologyGenetics
- Journals
- Biochemical and Biophysical Research CommunicationsAntioxidants and Redox SignalingFrontiers in Physiology
- Partner nations
- ChinaUnited States
In The Last Decade
Xiaolei Shi
14 papers receiving 800 citations
Hit Papers
Peers
Comparison fields: 5 of 76
- Molecular Biology 502
- Cancer Research 317
- Genetics 147
- Hepatology 135
- Surgery 129
Countries citing papers authored by Xiaolei Shi
This map shows the geographic impact of Xiaolei Shi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xiaolei Shi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xiaolei Shi more than expected).
Fields of papers citing papers by Xiaolei Shi
This network shows the impact of papers produced by Xiaolei Shi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xiaolei Shi. The network helps show where Xiaolei Shi may publish in the future.
Co-authorship network of co-authors of Xiaolei Shi
This figure shows the co-authorship network connecting the top 25 collaborators of Xiaolei Shi. A scholar is included among the top collaborators of Xiaolei Shi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xiaolei Shi. Xiaolei Shi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 3 | |
| 3 | 74 | |
| 4 | 37 | |
| 5 | The up-regulation of P62 levels is associated with resistance of sorafenib in hepatocarcinoma cells. | 8 |
| 6 | 25 | |
| 7 | Hepatocellular carcinoma-derived exosomal miRNA-21 contributes to tumor progression by converting hepatocyte stellate cells to cancer-associated fibroblastsbreakdown → | 370 |
| 8 | 20 | |
| 9 | 32 | |
| 10 | 13 | |
| 11 | 72 | |
| 12 | 79 | |
| 13 | 34 | |
| 14 | 34 |
About Xiaolei Shi
Xiaolei Shi is a scholar working on Biological Psychiatry, Hepatology and Critical Care and Intensive Care Medicine, having authored 14 papers that have together received 802 indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (4 papers), Autophagy in Disease and Therapy (3 papers) and Liver physiology and pathology (2 papers). The work is most often cited by research in Cancer Research (317 citations), Hepatology (135 citations) and Genetics (147 citations). Xiaolei Shi has collaborated with scholars based in China and United States. Frequent co-authors include Haozhen Ren, Jun Li, Longcheng Shang, Yuan Zhou, Jianfei Huang, Bo Dai, Jinglin Wang, Yitao Ding, Hucheng Ma and Haoran Ding. Their work appears in journals such as Biochemical and Biophysical Research Communications, Antioxidants and Redox Signaling and Frontiers in Physiology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.