Xiangyan Wu

529 total citations
19 papers, 342 citations indexed

About

Xiangyan Wu is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Xiangyan Wu has authored 19 papers receiving a total of 342 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 8 papers in Oncology and 6 papers in Immunology. Recurrent topics in Xiangyan Wu's work include RNA modifications and cancer (5 papers), Cancer Immunotherapy and Biomarkers (3 papers) and Renin-Angiotensin System Studies (3 papers). Xiangyan Wu is often cited by papers focused on RNA modifications and cancer (5 papers), Cancer Immunotherapy and Biomarkers (3 papers) and Renin-Angiotensin System Studies (3 papers). Xiangyan Wu collaborates with scholars based in China and United States. Xiangyan Wu's co-authors include Jun Peng, Meizhu Wu, Zhiqing Shen, Youqin Chen, Nathaniel Weygant, Liya Liu, Courtney W. Houchen, Dongfeng Qu, Aling Shen and Ying Cheng and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Cancer Research.

In The Last Decade

Xiangyan Wu

18 papers receiving 341 citations

Peers

Xiangyan Wu

ONCOL

IMMUN

CCM

Zhiqing Shen China

Zifan Zeng China

Rui Yan China

Xuelei Cao China

Shengqi Huo China

Mélanie Bousquenaud France

Xingwen Da China

Xiaochun Lin China

Kenjiro Yamakawa Japan

Juhyung Woo United States

Zhiqing Shen China

Xiangyan Wu

240 ×1.2

63 ×0.8

ONCOL

101 ×1.3

23 ×0.4

IMMUN

69 ×1.3

CCM

Citations per year, relative to Xiangyan Wu Xiangyan Wu (= 1×) peers Zhiqing Shen

Countries citing papers authored by Xiangyan Wu

Since Specialization

Citations

This map shows the geographic impact of Xiangyan Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xiangyan Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xiangyan Wu more than expected).

Fields of papers citing papers by Xiangyan Wu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xiangyan Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xiangyan Wu. The network helps show where Xiangyan Wu may publish in the future.

Co-authorship network of co-authors of Xiangyan Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Xiangyan Wu. A scholar is included among the top collaborators of Xiangyan Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xiangyan Wu. Xiangyan Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

Wu, Xiangyan, et al.. (2023). A TP53-associated metabolic gene signature for the prediction of overall survival and therapeutic responses in hepatocellular carcinoma. SHILAP Revista de lepidopterología. 16(2). 100552–100552. 1 indexed citations

Cao, Yawen, et al.. (2023). Neutrophil extracellular traps-mediated molecular subtypes characterize the hallmarks of tumor microenvironment and guide precision medicine in hepatocellular carcinoma. SHILAP Revista de lepidopterología. 16(2). 100577–100577. 2 indexed citations

Gong, Ling, et al.. (2022). Case series of progressive familial intrahepatic cholestasis type 3: Characterization of variants in ABCB4 in China. Frontiers in Medicine. 9. 962408–962408. 2 indexed citations

Shen, Aling, Liya Liu, Yue Huang, et al.. (2022). Down-Regulating HAUS6 Suppresses Cell Proliferation by Activating the p53/p21 Pathway in Colorectal Cancer. Frontiers in Cell and Developmental Biology. 9. 16 indexed citations

Li, Jie, Liya Liu, Youqin Chen, et al.. (2022). Ribosome assembly factor PNO1 is associated with progression and promotes tumorigenesis in triple‑negative breast cancer. Oncology Reports. 47(6). 10 indexed citations

Wu, Xiangyan, Yuhan Ye, Kenneth J. Vega, & Jiannan Yao. (2022). Consensus Molecular Subtypes Efficiently Classify Gastric Adenocarcinomas and Predict the Response to Anti-PD-1 Immunotherapy. Cancers. 14(15). 3740–3740. 1 indexed citations

Wu, Xiangyan, et al.. (2021). Colony-stimulating factor 3 signaling in colon and rectal cancers: Immune response and CMS classification in TCGA data. PLoS ONE. 16(2). e0247233–e0247233. 14 indexed citations

Wu, Meizhu, Xiangyan Wu, Ying Cheng, et al.. (2021). Qingda Granule Attenuates Angiotensin II-Induced Blood Pressure and Inhibits Ca2+/ERK Signaling Pathway. Frontiers in Pharmacology. 12. 688877–688877. 18 indexed citations

Zhuang, Qunchuan, Aling Shen, Liya Liu, et al.. (2021). Prognostic and immunological roles of Fc fragment of IgG binding protein in colorectal cancer. Oncology Letters. 22(1). 526–526. 11 indexed citations

10.

Chen, Chu, Qingfeng Yi, Xiaoyan Zhang, et al.. (2020). N-terminal region of Helicobacter pylori CagA induces IL-8 production in gastric epithelial cells via the β1 integrin receptor. Journal of Medical Microbiology. 69(3). 457–464. 12 indexed citations

11.

Shen, Zhiqing, Youqin Chen, Li Li, et al.. (2020). Transcription Factor EBF1 Over-Expression Suppresses Tumor Growth in vivo and in vitro via Modulation of the PNO1/p53 Pathway in Colorectal Cancer. Frontiers in Oncology. 10. 1035–1035. 15 indexed citations

12.

Shen, Zhiqing, Xiangyan Wu, Jianfeng Chu, et al.. (2020). Huoxin pill attenuates myocardial infarction-induced apoptosis and fibrosis via suppression of p53 and TGF-β1/Smad2/3 pathways. Biomedicine & Pharmacotherapy. 130. 110618–110618. 18 indexed citations

13.

Wu, Xiangyan, Aling Shen, Meizhu Wu, et al.. (2020). Qingda granules attenuate hypertensive cardiac remodeling and inflammation in spontaneously hypertensive rats. Biomedicine & Pharmacotherapy. 129. 110367–110367. 20 indexed citations

14.

Cheng, Ying, Xiangyan Wu, Zhiqing Shen, et al.. (2020). Qingda granule attenuates angiotensin II-induced cardiac hypertrophy and apoptosis and modulates the PI3K/AKT pathway. Biomedicine & Pharmacotherapy. 133. 111022–111022. 56 indexed citations

15.

Nofchissey, Robert A., Maaz Khan, Megan R. Lerner, et al.. (2020). The role of sex in the innate and adaptive immune environment of metastatic colorectal cancer. British Journal of Cancer. 123(4). 624–632. 22 indexed citations

16.

Wu, Xiangyan, Dongfeng Qu, Nathaniel Weygant, Jun Peng, & Courtney W. Houchen. (2020). Cancer Stem Cell Marker DCLK1 Correlates with Tumorigenic Immune Infiltrates in the Colon and Gastric Adenocarcinoma Microenvironments. Cancers. 12(2). 274–274. 55 indexed citations

17.

Shen, Aling, Youqin Chen, Liya Liu, et al.. (2019). EBF1-Mediated Upregulation of Ribosome Assembly Factor PNO1 Contributes to Cancer Progression by Negatively Regulating the p53 Signaling Pathway. Cancer Research. 79(9). 2257–2270. 56 indexed citations

18.

Huang, Yue, Xiangyan Wu, Meizhu Wu, et al.. (2019). Antihypertensive and Vasodilatory Effects of Qingda Granules by Suppression of Calcium Influx and the AKT Pathway. Journal of Cardiovascular Pharmacology. 74(6). 549–557. 13 indexed citations

19.

Liu, Juan, et al.. (2015). [Epithelioid sarcoma of tongue:report of a case].. PubMed. 44(5). 346–7.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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