Xiangyan Liang

542 total citations
24 papers, 459 citations indexed

About

Xiangyan Liang is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Xiangyan Liang has authored 24 papers receiving a total of 459 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 4 papers in Pathology and Forensic Medicine and 4 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Xiangyan Liang's work include Cardiac Ischemia and Reperfusion (4 papers), Fibroblast Growth Factor Research (3 papers) and Epigenetics and DNA Methylation (3 papers). Xiangyan Liang is often cited by papers focused on Cardiac Ischemia and Reperfusion (4 papers), Fibroblast Growth Factor Research (3 papers) and Epigenetics and DNA Methylation (3 papers). Xiangyan Liang collaborates with scholars based in China, Australia and United States. Xiangyan Liang's co-authors include Haifeng Zhang, Fange Liu, Wenjuan Xing, Lei Zhang, Huqin Zhang, Ting Zhang, Lele Ji, Feng Fu, Yufeng Zhao and Fei Tian and has published in prestigious journals such as Journal of the American College of Cardiology, PLoS ONE and International Journal of Molecular Sciences.

In The Last Decade

Xiangyan Liang

22 papers receiving 451 citations

Peers

Xiangyan Liang
Gerald H. Wilken United States
Xiangyan Liang
Citations per year, relative to Xiangyan Liang Xiangyan Liang (= 1×) peers Gerald H. Wilken

Countries citing papers authored by Xiangyan Liang

Since Specialization
Citations

This map shows the geographic impact of Xiangyan Liang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xiangyan Liang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xiangyan Liang more than expected).

Fields of papers citing papers by Xiangyan Liang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xiangyan Liang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xiangyan Liang. The network helps show where Xiangyan Liang may publish in the future.

Co-authorship network of co-authors of Xiangyan Liang

This figure shows the co-authorship network connecting the top 25 collaborators of Xiangyan Liang. A scholar is included among the top collaborators of Xiangyan Liang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Xiangyan Liang. Xiangyan Liang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhang, Xiaochun, Xiangyan Liang, Lijun Zhang, et al.. (2022). α‐Lipoic acid up‐regulates gene expression but reduces protein levels of fibroblast growth factor 21 in HepG2 cells. Basic & Clinical Pharmacology & Toxicology. 131(4). 270–281.
2.
Zhang, Lijun, Xiangyan Liang, Xiaochun Zhang, et al.. (2022). β-Hydroxybutyrate upregulates FGF21 expression through inhibition of histone deacetylases in hepatocytes. Open Life Sciences. 17(1). 856–864.
3.
Liang, Xiangyan, et al.. (2022). Effect of family integrated care on physical growth and language development of premature infants: a retrospective study. Translational Pediatrics. 11(6). 965–977. 9 indexed citations
4.
Zhang, Lijun, Xiangyan Liang, Xiaochun Zhang, et al.. (2022). Pyruvate Upregulates Hepatic FGF21 Expression by Activating PDE and Inhibiting cAMP–Epac–CREB Signaling Pathway. International Journal of Molecular Sciences. 23(10). 5490–5490. 2 indexed citations
5.
Liu, Yingguang, et al.. (2020). The GPR120 Agonist TUG‐891 Inhibits the Motility and Phagocytosis of Mouse Alveolar Macrophages. BioMed Research International. 2020(1). 1706168–1706168. 8 indexed citations
6.
Zhao, Yufeng, Xiaocheng Li, Xiangyan Liang, et al.. (2020). GPR120 Regulates Pancreatic Polypeptide Secretion From Male Mouse Islets via PLC-Mediated Calcium Mobilization. Endocrinology. 161(10). 16 indexed citations
7.
Fu, Hui, et al.. (2019). Lipopolysaccharide inhibits GPR120 expression in macrophages via Toll‐like receptor 4 and p38 MAPK activation. Cell Biology International. 44(1). 89–97. 8 indexed citations
8.
Wang, Li, Xuefei Tian, Xiaoli Cheng, et al.. (2019). Protective effects of GPR120 agonist-programmed macrophages on renal interstitial fibrosis in unilateral ureteral obstruction (UUO) rats. Biomedicine & Pharmacotherapy. 117. 109172–109172. 13 indexed citations
9.
Zhao, Yufeng, Feng Jiang, Xiangyan Liang, et al.. (2018). Grifolic acid causes osteosarcoma cell death in vitro and in tumor-bearing mice. Biomedicine & Pharmacotherapy. 103. 1035–1042. 3 indexed citations
10.
Zhao, Yufeng, Lei Zhang, Di Chen, et al.. (2018). Grifolic acid induces GH3 adenoma cell death by inhibiting ATP production through a GPR120-independent mechanism. BMC Pharmacology and Toxicology. 19(1). 26–26. 9 indexed citations
11.
Liu, Yingguang, Yanyan Zhao, Jin Yang, et al.. (2018). [Differential expression and correlation to cytokine expressions of FFAR4 in peritoneal and alveolar macrophages].. PubMed. 34(1). 22–27. 2 indexed citations
12.
Dong, Qianqian, Wenjuan Xing, Xiangyan Liang, et al.. (2017). Tetrahydroxystilbene Glycoside Improves Microvascular Endothelial Dysfunction and Ameliorates Obesity-Associated Hypertension in Obese ZDF Rats Via Inhibition of Endothelial Autophagy. Cellular Physiology and Biochemistry. 43(1). 293–307. 20 indexed citations
14.
Wang, Wenqing, Xiangyan Liang, Wenjuan Xing, et al.. (2015). Apocynum venetum Leaf Attenuates Myocardial Ischemia/Reperfusion Injury by Inhibiting Oxidative Stress. The American Journal of Chinese Medicine. 43(1). 71–85. 15 indexed citations
15.
Liang, Xiangyan, et al.. (2014). GW25-e4408 Magnolol administration in prehypertension postpones the development of hypertension and the underlying mechanisms. Journal of the American College of Cardiology. 64(16). C25–C25. 2 indexed citations
16.
Fu, Feng, Liming Yu, Wenjuan Xing, et al.. (2014). Vasonatrin peptide attenuates myocardial ischemia-reperfusion injury in diabetic rats and underlying mechanisms. American Journal of Physiology-Heart and Circulatory Physiology. 308(4). H281–H290. 46 indexed citations
17.
Zhang, Lei, Huqin Zhang, Xiangyan Liang, et al.. (2013). Melatonin ameliorates cognitive impairment induced by sleep deprivation in rats: Role of oxidative stress, BDNF and CaMKII. Behavioural Brain Research. 256. 72–81. 122 indexed citations
18.
Zhang, Wei, Rong Li, Jia Li, et al.. (2013). Alpha-Linolenic Acid Exerts an Endothelial Protective Effect against High Glucose Injury via PI3K/Akt Pathway. PLoS ONE. 8(7). e68489–e68489. 26 indexed citations
19.
Ji, Lele, Ying Nan, Wenqing Wang, et al.. (2013). Achyranthes bidentata Polypeptides Reduces Oxidative Stress and Exerts Protective Effects against Myocardial Ischemic/Reperfusion Injury in Rats. International Journal of Molecular Sciences. 14(10). 19792–19804. 23 indexed citations
20.
Liang, Xiangyan, Jian‐Kang Chen, Yufeng Zhao, et al.. (2011). Effect of Feining on bleomycin-induced pulmonary injuries in rats. Journal of Ethnopharmacology. 134(3). 971–976. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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