Lele Ji

1.7k total citations
31 papers, 1.3k citations indexed

About

Lele Ji is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Lele Ji has authored 31 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 9 papers in Pathology and Forensic Medicine and 9 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Lele Ji's work include Cardiac Ischemia and Reperfusion (9 papers), Mitochondrial Function and Pathology (8 papers) and Cardiovascular Function and Risk Factors (5 papers). Lele Ji is often cited by papers focused on Cardiac Ischemia and Reperfusion (9 papers), Mitochondrial Function and Pathology (8 papers) and Cardiovascular Function and Risk Factors (5 papers). Lele Ji collaborates with scholars based in China, United States and Indonesia. Lele Ji's co-authors include Qichao Huang, Jinliang Xing, Haifeng Zhang, Tingting Ren, Xu Guo, Feng Fu, Haiyan Cao, Wenjuan Xing, Jibin Li and Jing Zhang and has published in prestigious journals such as Nature Communications, SHILAP Revista de lepidopterología and PLoS ONE.

In The Last Decade

Lele Ji

28 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Lele Ji China 18 784 215 199 198 185 31 1.3k
Xiangning Cui China 18 537 0.7× 145 0.7× 142 0.7× 119 0.6× 88 0.5× 34 1.2k
Jingxue Ye China 20 640 0.8× 112 0.5× 133 0.7× 101 0.5× 171 0.9× 39 1.2k
Chunhua Zhu China 26 793 1.0× 211 1.0× 125 0.6× 180 0.9× 195 1.1× 41 1.9k
Sehwan Jang Puerto Rico 21 880 1.1× 153 0.7× 373 1.9× 236 1.2× 432 2.3× 30 1.6k
Mirco Zucchelli Italy 20 681 0.9× 312 1.5× 129 0.6× 166 0.8× 137 0.7× 38 1.5k
Shen‐Jiang Hu China 20 685 0.9× 251 1.2× 99 0.5× 124 0.6× 126 0.7× 84 1.3k
Qiren Huang China 24 678 0.9× 80 0.4× 229 1.2× 220 1.1× 191 1.0× 71 1.3k
Xue Dong China 18 509 0.6× 140 0.7× 105 0.5× 115 0.6× 122 0.7× 47 994
Hai-Di Li China 25 736 0.9× 220 1.0× 317 1.6× 108 0.5× 323 1.7× 50 1.6k

Countries citing papers authored by Lele Ji

Since Specialization
Citations

This map shows the geographic impact of Lele Ji's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lele Ji with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lele Ji more than expected).

Fields of papers citing papers by Lele Ji

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lele Ji. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lele Ji. The network helps show where Lele Ji may publish in the future.

Co-authorship network of co-authors of Lele Ji

This figure shows the co-authorship network connecting the top 25 collaborators of Lele Ji. A scholar is included among the top collaborators of Lele Ji based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lele Ji. Lele Ji is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhang, Xiaoliang, Chenchen Xu, Lele Ji, & Haiwei Zhang. (2025). Endoplasmic reticulum stress in acute pancreatitis: Exploring the molecular mechanisms and therapeutic targets. Cell Stress and Chaperones. 30(3). 119–129. 3 indexed citations
2.
3.
He, Linjie, Lifei Guo, Man Gao, et al.. (2025). Hepatic AKAP1 deficiency exacerbates diet-induced MASLD by enhancing GPAT1-mediated lysophosphatidic acid synthesis. Nature Communications. 16(1). 4286–4286. 1 indexed citations
4.
Li, Yaya, Lele Ji, Chang Long, et al.. (2025). 4D Printable Shape Memory Polyamide Elastomer by Reactive Extrusion. ACS Applied Polymer Materials. 7(14). 8999–9009.
5.
6.
Yang, Ailin, Lifei Guo, Yanfang Zhang, et al.. (2023). MFN2-mediated mitochondrial fusion facilitates acute hypobaric hypoxia-induced cardiac dysfunction by increasing glucose catabolism and ROS production. Biochimica et Biophysica Acta (BBA) - General Subjects. 1867(9). 130413–130413. 3 indexed citations
7.
Yang, Ailin, Zhang Li, Linjie He, et al.. (2023). MFN2 deficiency promotes cardiac response to hypobaric hypoxia by reprogramming cardiomyocyte metabolism. Acta Physiologica. 239(1). e14018–e14018. 3 indexed citations
8.
Qi, Bingchao, Linjie He, Ya Zhao, et al.. (2020). Akap1 deficiency exacerbates diabetic cardiomyopathy in mice by NDUFS1-mediated mitochondrial dysfunction and apoptosis. Diabetologia. 63(5). 1072–1087. 91 indexed citations
9.
Wang, Qian, et al.. (2019). The Relationship between Mitochondria Ca2+ Intake Mediated by Mitochondria-associated Endoplasmic Reticulum Membranes and Tumor Genesis. 4(1). 1–5. 1 indexed citations
10.
Ren, Tingting, H Zhang, J Wang, et al.. (2017). MCU-dependent mitochondrial Ca2+ inhibits NAD+/SIRT3/SOD2 pathway to promote ROS production and metastasis of HCC cells. Oncogene. 36(42). 5897–5909. 152 indexed citations
11.
Ren, Tingting, Jiaojiao Wang, Hui Zhang, et al.. (2017). MCUR1-Mediated Mitochondrial Calcium Signaling Facilitates Cell Survival of Hepatocellular Carcinoma via Reactive Oxygen Species-Dependent P53 Degradation. Antioxidants and Redox Signaling. 28(12). 1120–1136. 57 indexed citations
12.
13.
Wang, Wenqing, Xiangyan Liang, Wenjuan Xing, et al.. (2015). Apocynum venetum Leaf Attenuates Myocardial Ischemia/Reperfusion Injury by Inhibiting Oxidative Stress. The American Journal of Chinese Medicine. 43(1). 71–85. 15 indexed citations
14.
Zhang, Xing, Jie Xu, Xiaoqing Cai, et al.. (2014). Acute Insulin Resistance Mediated by Advanced Glycation Endproducts in Severely Burned Rats. Critical Care Medicine. 42(6). e472–e480. 12 indexed citations
15.
Cui, Yongliang, Lele Ji, Wei Zhang, et al.. (2013). Catalpol decreases peroxynitrite formation and consequently exerts cardioprotective effects against ischemia/reperfusion insult. Pharmaceutical Biology. 51(4). 463–473. 45 indexed citations
16.
Ji, Lele, Xing Zhang, Qichao Huang, et al.. (2013). AMPK-Regulated and Akt-Dependent Enhancement of Glucose Uptake Is Essential in Ischemic Preconditioning-Alleviated Reperfusion Injury. PLoS ONE. 8(7). e69910–e69910. 69 indexed citations
17.
Ji, Lele, Wenjuan Xing, Ling Dong, & Feng Gao. (2013). Searching for Phytoinsulins as Cardiovascular Protector in Metabolic Syndrome. Current Pharmaceutical Design. 19(27). 4848–4858. 1 indexed citations
18.
Ji, Lele, Haibo Wang, Haifeng Zhang, et al.. (2009). [Synthesis and cardioprotective effect of a novel anti-ischemic/reperfused injury compound].. PubMed. 44(3). 321–6. 2 indexed citations
19.
Xing, Wenjuan, Wenjun Yan, Feng Fu, et al.. (2009). Insulin inhibits myocardial ischemia-induced apoptosis and alleviates chronic adverse changes in post-ischemic cardiac structure and function. APOPTOSIS. 14(9). 1050–1060. 27 indexed citations
20.
Li, Jia, Feng Wu, Haifeng Zhang, et al.. (2009). Insulin inhibits leukocyte–endothelium adherence via an Akt-NO-dependent mechanism in myocardial ischemia/reperfusion. Journal of Molecular and Cellular Cardiology. 47(4). 512–519. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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