Witaya Thamavit

1.3k total citations · 1 hit paper
49 papers, 1.0k citations indexed

About

Witaya Thamavit is a scholar working on Parasitology, Molecular Biology and Small Animals. According to data from OpenAlex, Witaya Thamavit has authored 49 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Parasitology, 11 papers in Molecular Biology and 10 papers in Small Animals. Recurrent topics in Witaya Thamavit's work include Parasites and Host Interactions (17 papers), Helminth infection and control (10 papers) and Drug-Induced Hepatotoxicity and Protection (6 papers). Witaya Thamavit is often cited by papers focused on Parasites and Host Interactions (17 papers), Helminth infection and control (10 papers) and Drug-Induced Hepatotoxicity and Protection (6 papers). Witaya Thamavit collaborates with scholars based in Thailand, Japan and Australia. Witaya Thamavit's co-authors include Nobuyuki Ito, Natth Bhamarapravati, Malcolm A Moore, Somphong Sahaphong, Subhkij Angsubhakorn, S Vajrasthira, Danai Tiwawech, Nobuyuki Ito, Tomoyuki Shirai and Chawalit Pairojkul and has published in prestigious journals such as JNCI Journal of the National Cancer Institute, British Journal of Cancer and International Journal of Cancer.

In The Last Decade

Witaya Thamavit

48 papers receiving 971 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Effects of dimethylnitrosamine on induction of cholangiocarcinoma in Opisthorchis viverrini-infected Syrian golden hamsters.

1978 246 citations Witaya Thamavit, Natth Bhamarapravati et al. PubMed profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Witaya Thamavit Thailand	17	283	275	266	206	168	49	1.0k
George R. Lankas United States	11	106 0.4×	26 0.1×	273 1.0×	349 1.7×	136 0.8×	24	932
R Pathmanathan Malaysia	12	154 0.5×	104 0.4×	121 0.5×	265 1.3×	88 0.5×	52	816
Chul Ho Lee South Korea	17	138 0.5×	40 0.1×	320 1.2×	134 0.7×	51 0.3×	53	910
Nigel B. Rendell United Kingdom	18	171 0.6×	38 0.1×	432 1.6×	97 0.5×	62 0.4×	36	929
S Bunyaratvej Thailand	13	98 0.3×	106 0.4×	97 0.4×	64 0.3×	17 0.1×	30	436
Anass Haimeur Canada	17	122 0.4×	212 0.8×	465 1.7×	694 3.4×	31 0.2×	19	1.7k
Yukinobu Goso Japan	15	285 1.0×	47 0.2×	393 1.5×	63 0.3×	17 0.1×	41	712
H. Frohberg Germany	13	31 0.1×	93 0.3×	100 0.4×	46 0.2×	151 0.9×	65	531
Tomoyuki Shirai Japan	12	64 0.2×	40 0.1×	215 0.8×	80 0.4×	110 0.7×	22	569
Regina J. Brown United States	13	48 0.2×	53 0.2×	386 1.5×	107 0.5×	134 0.8×	18	717

Countries citing papers authored by Witaya Thamavit

Since Specialization

Citations

This map shows the geographic impact of Witaya Thamavit's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Witaya Thamavit with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Witaya Thamavit more than expected).

Fields of papers citing papers by Witaya Thamavit

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Witaya Thamavit. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Witaya Thamavit. The network helps show where Witaya Thamavit may publish in the future.

Co-authorship network of co-authors of Witaya Thamavit

This figure shows the co-authorship network connecting the top 25 collaborators of Witaya Thamavit. A scholar is included among the top collaborators of Witaya Thamavit based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Witaya Thamavit. Witaya Thamavit is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Moore, Malcolm A, Witaya Thamavit, Danai Tiwawech, Nobuyuki Ito, & Hiroyuki Tsuda. (1998). Modulation of Dihydroxy‐di‐n‐propylnitrosamine‐induced Liver Lesion Development in Opisthorchis‐infected Syrian Hamsters by Praziquantel Treatment in Association with Butylated Hydroxyanisole or Dehydroepiandrosterone Administration. Japanese Journal of Cancer Research. 89(11). 1113–1117. 1 indexed citations

Thamavit, Witaya, Chawalit Pairojkul, Danai Tiwawech, Tomoyuki Shirai, & Nobuyuki Ito. (1994). Lack of promoting effect of proline on bile duct cancer development in dimethylnitrosamine‐initiated hamster livers. Teratogenesis Carcinogenesis and Mutagenesis. 14(4). 169–174. 3 indexed citations

Chawengkirttikul, Runglawan, et al.. (1994). Identification and potential use of a soluble tumor antigen for the detection of liver‐fluke‐associated cholangiocarcinoma induced in a hamster model. International Journal of Cancer. 57(5). 691–695. 10 indexed citations

Thamavit, Witaya, Chawalit Pairojkul, Danai Tiwawech, Tomoyuki Shirai, & Nobuyuki Ito. (1994). Strong promoting effect of Opisthorchis viverrini infection on dimethylnitrosamine-initiated hamster liver. Cancer Letters. 78(1-3). 121–125. 41 indexed citations

Thamavit, Witaya, Chawalit Pairojkul, Danai Tiwawech, et al.. (1993). SHORT COMMUNICATION: Promotion of cholangiocarcinogenesis in the hamster liver by bile duct ligation after dimethylnitrosamine initiation. Carcinogenesis. 14(11). 2415–2417. 56 indexed citations

Thamavit, Witaya, Malcolm A Moore, Stitaya Sirisinha, Tomoyuki Shirai, & Nobuyuki Ito. (1993). Time‐dependent Modulation of Liver Lesion Development in Opisthorchis‐infected Syrian Hamster by an Antihelminthic Drug, Praziquantel. Japanese Journal of Cancer Research. 84(2). 135–138. 12 indexed citations

Shirai, Tomoyuki, et al.. (1992). Histomorphological Characteristics of Cholangiocellular Carcinomas in Northeast Thailand, Where a Region Infection with the Liver Fluke, Opisthorchis viverrini is Endemic. Acta Pathologica Japonica. 42(10). 734–739. 6 indexed citations

Thamavit, Witaya, Malcolm A Moore, Somsak Ruchirawat, & Nobuyuki Ito. (1992). Repeated exposure to Opisthorchis viverrini and treatment with the antihelminthic Praziquantel lacks carcinogenic potential. Carcinogenesis. 13(2). 309–311. 16 indexed citations

Poonswad, Pilai, et al.. (1992). Chaunocephalosis in a Wild Population of Asian Open-billed Storks in Thailand. Journal of Wildlife Diseases. 28(3). 460–466. 8 indexed citations

10.

Thamavit, Witaya, Malcolm A Moore, Somsak Ruchirawat, & N. Ito. (1992). Lack of modulation effects of praziquantel on DMN-induced lesion development in the Syrian hamster liver. Cancer Letters. 61(3). 229–232. 4 indexed citations

11.

Shirai, Tomoyuki, Kazuo Hakoi, Witaya Thamavit, et al.. (1991). Promotion of Rat Hepatocarcinogenesis by Praziquantel. Japanese Journal of Cancer Research. 82(10). 1085–1088. 6 indexed citations

12.

Kitahori, Yoshiteru, M Ohshima, N Konishi, et al.. (1989). Promoting effect of 2,4-diaminoanisole sulfate on rat thyroid carcinogenesis. Cancer Letters. 45(2). 115–121. 12 indexed citations

13.

Thamavit, Witaya, Shoji Fukushima, Y. Kurata, M. Asamoto, & N. Ito. (1989). Modification by sodium l-ascorbate, butylated hydroxytoluene, phenobarbital and pepleomycin of lesion development in a wide-spectrum initiation rat model. Cancer Letters. 45(2). 93–101. 19 indexed citations

14.

Thamavit, Witaya, Malcolm A Moore, Yoshio Hiasa, & Nobuyuki Ito. (1988). Generation of High Yields of Syrian Hamster Cholangiocellular Carcinomas and Hepatocellular Nodules by Combined Nitrite and Aminopyrine Administration and Opisthorchis viverrini Infection. Japanese Journal of Cancer Research. 79(8). 909–916. 23 indexed citations

15.

Thamavit, Witaya, et al.. (1988). Enhancement of DHPN induced hepatocellular, cholangiocellular and pancreatic carcinogenesis by Opisthorchis viverrini infestation in Syrian golden hamsters. Carcinogenesis. 9(6). 1095–1098. 22 indexed citations

16.

Thamavit, Witaya, et al.. (1987). Level ofOpisthorchis infestation and carcinogen dose-dependence of cholangiocarcinoma induction in Syrian golden hamsters. Virchows Archiv B Cell Pathology Including Molecular Pathology. 54(1). 52–58. 70 indexed citations

17.

Moore, Malcolm A, Witaya Thamavit, Hiroyuki Tsuda, et al.. (1986). Modifying influence of dehydroepiandrosterone on the development of dihydroxy-di-n-propylnitrosamine-initiated lesions in the thyroid, lung and liver of F344 rats. Carcinogenesis. 7(2). 311–316. 50 indexed citations

18.

Ruenwongsa, Pintip, et al.. (1983). Liver collagen in Opisthorchis viverrini infected hamsters following praziquantel treatment.. PubMed. 14(3). 290–3. 6 indexed citations

19.

Ruenwongsa, Pintip & Witaya Thamavit. (1983). Increased efficacy of liposomes-encapsulated praziquantel in treatment of opisthorchiasis in hamsters.. PubMed. 14(4). 501–4. 3 indexed citations

20.

Bhamarapravati, Natth, et al.. (1981). Further study of α benzene hexachloride inhibition of aflatoxin B1 hepatocarcinogenesis in rats. British Journal of Cancer. 43(6). 881–883. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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