Wing‐Keung Chu

413 total citations
17 papers, 351 citations indexed

About

Wing‐Keung Chu is a scholar working on Molecular Biology, Surgery and Oncology. According to data from OpenAlex, Wing‐Keung Chu has authored 17 papers receiving a total of 351 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 3 papers in Surgery and 3 papers in Oncology. Recurrent topics in Wing‐Keung Chu's work include RNA Research and Splicing (5 papers), Cancer-related Molecular Pathways (3 papers) and Cancer-related gene regulation (3 papers). Wing‐Keung Chu is often cited by papers focused on RNA Research and Splicing (5 papers), Cancer-related Molecular Pathways (3 papers) and Cancer-related gene regulation (3 papers). Wing‐Keung Chu collaborates with scholars based in Taiwan and United States. Wing‐Keung Chu's co-authors include Jan‐Kan Chen, Shu‐Er Chow, Li‐Man Hung, Ming‐Jai Su, Shiang‐Fu Huang, Chih‐Chin Hsu, Cheng‐Ta Yang, Wei‐Shan Chen, Yi‐Jen Hsueh and Jong‐Shyan Wang and has published in prestigious journals such as Journal of Biological Chemistry, Biochemical and Biophysical Research Communications and International Journal of Molecular Sciences.

In The Last Decade

Wing‐Keung Chu

16 papers receiving 349 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wing‐Keung Chu Taiwan 10 162 89 55 53 40 17 351
Lyndsay Willmott United States 10 136 0.8× 129 1.4× 58 1.1× 32 0.6× 49 1.2× 36 381
V. Bumbaširević Serbia 8 186 1.1× 59 0.7× 59 1.1× 21 0.4× 30 0.8× 11 363
Sheng-Hong Tseng Taiwan 8 192 1.2× 81 0.9× 50 0.9× 100 1.9× 32 0.8× 12 362
Jenna D. Lovaas United States 5 215 1.3× 111 1.2× 96 1.7× 134 2.5× 24 0.6× 6 454
Zhang Yj China 11 220 1.4× 113 1.3× 102 1.9× 11 0.2× 21 0.5× 39 379
Weiqi Tan China 9 347 2.1× 39 0.4× 86 1.6× 30 0.6× 34 0.8× 12 488
Hala Elnakat Thomas United States 9 312 1.9× 68 0.8× 110 2.0× 12 0.2× 41 1.0× 9 460
Olivia Adams Switzerland 7 325 2.0× 83 0.9× 66 1.2× 11 0.2× 11 0.3× 10 508
Maylin Almonte‐Becerril Mexico 8 174 1.1× 43 0.5× 77 1.4× 22 0.4× 15 0.4× 20 440

Countries citing papers authored by Wing‐Keung Chu

Since Specialization
Citations

This map shows the geographic impact of Wing‐Keung Chu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wing‐Keung Chu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wing‐Keung Chu more than expected).

Fields of papers citing papers by Wing‐Keung Chu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wing‐Keung Chu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wing‐Keung Chu. The network helps show where Wing‐Keung Chu may publish in the future.

Co-authorship network of co-authors of Wing‐Keung Chu

This figure shows the co-authorship network connecting the top 25 collaborators of Wing‐Keung Chu. A scholar is included among the top collaborators of Wing‐Keung Chu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wing‐Keung Chu. Wing‐Keung Chu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Chu, Wing‐Keung, et al.. (2021). PKC Regulates YAP Expression through Alternative Splicing of YAP 3′UTR Pre-mRNA by hnRNP F. International Journal of Molecular Sciences. 22(2). 694–694. 3 indexed citations
2.
Chow, Shu‐Er, et al.. (2021). Human Caspase 12 Enhances NF-κB Activity through Activation of IKK in Nasopharyngeal Carcinoma Cells. International Journal of Molecular Sciences. 22(9). 4610–4610. 5 indexed citations
3.
Chu, Wing‐Keung, et al.. (2021). MicroRNA 630 Represses NANOG Expression through Transcriptional and Post-Transcriptional Regulation in Human Embryonal Carcinoma Cells. International Journal of Molecular Sciences. 23(1). 46–46. 6 indexed citations
4.
Chu, Wing‐Keung, et al.. (2018). Heterogeneous ribonucleoprotein F regulates YAP expression via a G-tract in 3′UTR. Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1862(1). 12–24. 8 indexed citations
5.
Yang, Cheng‐Ta, et al.. (2018). Downregulation of lumican accelerates lung cancer cell invasion through p120 catenin. Cell Death and Disease. 9(4). 414–414. 29 indexed citations
6.
Chu, Wing‐Keung, Chih‐Chin Hsu, Shiang‐Fu Huang, Chia‐Chi Hsu, & Shu‐Er Chow. (2017). Caspase 12 degrades IκBα protein and enhances MMP-9 expression in human nasopharyngeal carcinoma cell invasion. Oncotarget. 8(20). 33515–33526. 9 indexed citations
7.
Hsu, Chih‐Chin, Shiang‐Fu Huang, Jong‐Shyan Wang, et al.. (2016). Interplay of N-Cadherin and matrix metalloproteinase 9 enhances human nasopharyngeal carcinoma cell invasion. BMC Cancer. 16(1). 800–800. 39 indexed citations
8.
Hui‐Kang, David, Hung‐Chi Chen, Kevin Sheng-Kai, et al.. (2015). Preservation of human limbal epithelial progenitor cells on carbodiimide cross-linked amniotic membrane via integrin-linked kinase-mediated Wnt activation. Acta Biomaterialia. 31. 144–155. 37 indexed citations
9.
Chu, Wing‐Keung, et al.. (2013). Nanog expression is negatively regulated by protein kinase C activities in human cancer cell lines. Carcinogenesis. 34(7). 1497–1509. 8 indexed citations
10.
Chu, Wing‐Keung, et al.. (2008). Overexpression of ΔNp63 in a human nasopharyngeal carcinoma cell line downregulates CKIs and enhances cell proliferation. Journal of Cellular Physiology. 219(1). 117–122. 26 indexed citations
11.
Chu, Wing‐Keung, et al.. (2008). Glycogen synthase kinase‐3β regulates ΔNp63 gene transcription through the β‐catenin signaling pathway. Journal of Cellular Biochemistry. 105(2). 447–453. 14 indexed citations
12.
Chu, Wing‐Keung, et al.. (2008). Transcriptional Activity of the ΔNp63 Promoter Is Regulated by STAT3. Journal of Biological Chemistry. 283(12). 7328–7337. 30 indexed citations
13.
Chu, Wing‐Keung, et al.. (2006). Dual regulation of the ΔNp63 transcriptional activity by ΔNp63 in human nasopharyngeal carcinoma cell. Biochemical and Biophysical Research Communications. 342(4). 1356–1360. 20 indexed citations
14.
Hung, Li‐Man, WI Wei, Yi‐Jen Hsueh, Wing‐Keung Chu, & Fu‐Chan Wei. (2004). Ischemic preconditioning ameliorates microcirculatory disturbance through downregulation of TNF-alpha production in a rat cremaster muscle model. Journal of Biomedical Science. 11(6). 773–780. 9 indexed citations
15.
Hung, Li‐Man, WI Wei, Yi‐Jen Hsueh, Wing‐Keung Chu, & Fu‐Chan Wei. (2004). Ischemic Preconditioning Ameliorates Microcirculatory Disturbance through Downregulation of TNF-Alpha Production in a Rat Cremaster Muscle Model. Journal of Biomedical Science. 11(6). 773–780.
16.
Hung, Li‐Man, et al.. (2002). The protective effect of resveratrols on ischaemia‐reperfusion injuries of rat hearts is correlated with antioxidant efficacy. British Journal of Pharmacology. 135(7). 1627–1633. 106 indexed citations
17.
Chow, Shu‐Er, et al.. (2002). EXPOSURE TO OXIDIZED LOW-DENSITY LIPOPROTEIN REDUCES ACTIVABLE RAS PROTEIN IN VASCULAR ENDOTHELIAL CELLS. In Vitro Cellular & Developmental Biology - Animal. 38(6). 320–320. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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