William L. Willis

504 total citations
16 papers, 392 citations indexed

About

William L. Willis is a scholar working on Molecular Biology, Immunology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, William L. Willis has authored 16 papers receiving a total of 392 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Immunology and 4 papers in Pulmonary and Respiratory Medicine. Recurrent topics in William L. Willis's work include Blood properties and coagulation (3 papers), Extracellular vesicles in disease (2 papers) and Regulation of Appetite and Obesity (2 papers). William L. Willis is often cited by papers focused on Blood properties and coagulation (3 papers), Extracellular vesicles in disease (2 papers) and Regulation of Appetite and Obesity (2 papers). William L. Willis collaborates with scholars based in United States, Italy and Argentina. William L. Willis's co-authors include Douglas R. Seals, K. P. Davy, Wael N. Jarjour, Lai‐Chu Wu, Nicholas A. Young, Brian Hutzen, Stephanie Deangelis, Jiayuh Lin, Ke Ding and Mark Gardner and has published in prestigious journals such as Journal of Biological Chemistry, Blood and Gastroenterology.

In The Last Decade

William L. Willis

16 papers receiving 381 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
William L. Willis United States 13 186 69 61 55 51 16 392
Haizhao Yan China 12 265 1.4× 82 1.2× 63 1.0× 66 1.2× 18 0.4× 28 551
Zakia Shinwari Saudi Arabia 15 293 1.6× 48 0.7× 16 0.3× 73 1.3× 36 0.7× 31 552
Weiwei Liu China 11 185 1.0× 79 1.1× 20 0.3× 82 1.5× 73 1.4× 24 476
Yaoyao Zhou China 10 134 0.7× 81 1.2× 73 1.2× 53 1.0× 12 0.2× 16 583
Ritwik Datta India 11 235 1.3× 38 0.6× 161 2.6× 28 0.5× 11 0.2× 17 448
Xiaoju Li China 15 240 1.3× 45 0.7× 33 0.5× 30 0.5× 13 0.3× 46 546
Jinti Lin China 13 247 1.3× 68 1.0× 10 0.2× 40 0.7× 33 0.6× 17 565
Zewei Zhuo China 12 235 1.3× 44 0.6× 16 0.3× 56 1.0× 40 0.8× 36 462
Linjie He China 7 223 1.2× 31 0.4× 29 0.5× 18 0.3× 31 0.6× 12 341
Jianjie Wang China 10 140 0.8× 24 0.3× 30 0.5× 23 0.4× 15 0.3× 20 370

Countries citing papers authored by William L. Willis

Since Specialization
Citations

This map shows the geographic impact of William L. Willis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William L. Willis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William L. Willis more than expected).

Fields of papers citing papers by William L. Willis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William L. Willis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William L. Willis. The network helps show where William L. Willis may publish in the future.

Co-authorship network of co-authors of William L. Willis

This figure shows the co-authorship network connecting the top 25 collaborators of William L. Willis. A scholar is included among the top collaborators of William L. Willis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William L. Willis. William L. Willis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Bruckner, Shane, Binnaz Leblebicioğlu, Kanu S. Goyal, et al.. (2023). The therapeutic effects of gingival mesenchymal stem cells and their exosomes in a chimeric model of rheumatoid arthritis. Arthritis Research & Therapy. 25(1). 211–211. 13 indexed citations
2.
Hart, Marcia L., Takuma Tsuzuki Wada, William L. Willis, et al.. (2022). Gut dysbiosis is associated with acceleration of lupus nephritis. Scientific Reports. 12(1). 152–152. 31 indexed citations
3.
Willis, William L., et al.. (2020). In vitro Crosslinking Reactions and Substrate Incorporation Assays for The Identification of Transglutaminase-2 Protein Substrates. BIO-PROTOCOL. 10(12). e3657–e3657. 1 indexed citations
4.
Willis, William L., Linan Wang, Takuma Tsuzuki Wada, et al.. (2018). The proinflammatory protein HMGB1 is a substrate of transglutaminase-2 and forms high-molecular weight complexes with autoantigens. Journal of Biological Chemistry. 293(22). 8394–8409. 14 indexed citations
5.
Willis, William L., Liang Wang, Takuma Tsuzuki Wada, et al.. (2018). SAT0015 Identification and characterisationof high molecular weight hmgb1 protein complexes: implications for stress response, innate immunity and autoimmune disease. Annals of the Rheumatic Diseases. 77. 875–876. 1 indexed citations
6.
Wu, Lai‐Chu, William L. Willis, Stacy P. Ardoin, et al.. (2017). Daily Moderate Exercise Is Beneficial and Social Stress Is Detrimental to Disease Pathology in Murine Lupus Nephritis. Frontiers in Physiology. 8. 236–236. 17 indexed citations
7.
Young, Nicholas A., Lai‐Chu Wu, Craig J. Burd, et al.. (2016). Estrogen-regulated STAT1 activation promotes TLR8 expression to facilitate signaling via microRNA-21 in systemic lupus erythematosus. Clinical Immunology. 176. 12–22. 48 indexed citations
8.
9.
Young, Nicholas A., Mark Gardner, William L. Willis, et al.. (2014). Oral Administration of Nano-Emulsion Curcumin in Mice Suppresses Inflammatory-Induced NFκB Signaling and Macrophage Migration. PLoS ONE. 9(11). e111559–e111559. 49 indexed citations
10.
Willis, William L., et al.. (2013). Transglutaminase‐2 mediates calcium‐regulated crosslinking of the Y‐Box 1 (YB‐1) translation‐regulatory protein in TGFβ1‐activated myofibroblasts. Journal of Cellular Biochemistry. 114(12). 2753–2769. 12 indexed citations
11.
Subramanian, Sukanya, Aiwen Zhang, William L. Willis, et al.. (2012). Y-box binding protein-1 implicated in translational control of fetal myocardial gene expression after cardiac transplant. Experimental Biology and Medicine. 237(5). 593–607. 15 indexed citations
12.
13.
Sobo, Matthew, Sarah Ball, Brian Hutzen, et al.. (2009). MI-63: A novel small-molecule inhibitor targets MDM2 and induces apoptosis in embryonal and alveolar rhabdomyosarcoma cells with wild-type p53. British Journal of Cancer. 101(5). 774–781. 64 indexed citations
14.
Eiring, Anna M., Paolo Neviani, Ramasamy Santhanam, et al.. (2007). Identification of novel posttranscriptional targets of the BCR/ABL oncoprotein by ribonomics: requirement of E2F3 for BCR/ABL leukemogenesis. Blood. 111(2). 816–828. 33 indexed citations
15.
Hoogerwerf, Willemijntje A., Zhijun He, Shu‐Yuan Xiao, et al.. (2003). Mice lacking mast cells fail to develop nociceptive sensitization in response to chronic pancreatitis. Gastroenterology. 124(4). A32–A32. 1 indexed citations
16.
Davy, K. P., William L. Willis, & Douglas R. Seals. (1997). Influence of exercise training on heart rate variability in post‐menopausal women with elevated arterial blood pressure. Clinical Physiology. 17(1). 31–40. 53 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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