William H. Griffith

2.9k total citations
61 papers, 2.4k citations indexed

About

William H. Griffith is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, William H. Griffith has authored 61 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 49 papers in Cellular and Molecular Neuroscience, 38 papers in Molecular Biology and 18 papers in Cognitive Neuroscience. Recurrent topics in William H. Griffith's work include Neuroscience and Neuropharmacology Research (44 papers), Ion channel regulation and function (24 papers) and Neuroscience and Neural Engineering (14 papers). William H. Griffith is often cited by papers focused on Neuroscience and Neuropharmacology Research (44 papers), Ion channel regulation and function (24 papers) and Neuroscience and Neural Engineering (14 papers). William H. Griffith collaborates with scholars based in United States, Australia and United Kingdom. William H. Griffith's co-authors include David A. Brown, David Murchison, Louise C. Abbott, Lathrop Taylor, Leonard S. Dove, Robert T. Matthews, Joel P. Gallagher, Patricia Shinnick‐Gallagher, Sang‐Soep Nahm and Gerald D. Frye and has published in prestigious journals such as Journal of Neuroscience, The Journal of Physiology and Journal of Neurophysiology.

In The Last Decade

William H. Griffith

60 papers receiving 2.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
William H. Griffith United States 29 1.8k 1.2k 663 294 240 61 2.4k
Shui‐Wang Ying United States 20 1.7k 1.0× 1.1k 0.9× 539 0.8× 317 1.1× 237 1.0× 25 2.8k
Hideho Higashi Japan 33 2.3k 1.3× 1.8k 1.4× 414 0.6× 630 2.1× 232 1.0× 93 3.3k
Hylan C. Moises United States 31 1.8k 1.0× 994 0.8× 897 1.4× 379 1.3× 289 1.2× 50 2.5k
Saobo Lei United States 29 1.8k 1.0× 1.3k 1.0× 640 1.0× 277 0.9× 194 0.8× 63 2.6k
Hugh E. Criswell United States 27 1.5k 0.8× 926 0.7× 421 0.6× 191 0.6× 283 1.2× 65 2.4k
Thomas Munsch Germany 26 1.2k 0.7× 954 0.8× 474 0.7× 119 0.4× 134 0.6× 48 1.7k
Michael W. Swank United States 15 1.3k 0.7× 875 0.7× 705 1.1× 164 0.6× 211 0.9× 22 2.1k
James R. Unnerstall United States 22 1.8k 1.0× 1.4k 1.1× 312 0.5× 446 1.5× 142 0.6× 35 3.0k
Thomas McMahon United States 31 1.6k 0.9× 1.5k 1.2× 480 0.7× 640 2.2× 188 0.8× 45 3.0k
Thomas C. Foster United States 26 1.3k 0.7× 824 0.7× 937 1.4× 361 1.2× 372 1.6× 43 2.6k

Countries citing papers authored by William H. Griffith

Since Specialization
Citations

This map shows the geographic impact of William H. Griffith's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William H. Griffith with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William H. Griffith more than expected).

Fields of papers citing papers by William H. Griffith

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William H. Griffith. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William H. Griffith. The network helps show where William H. Griffith may publish in the future.

Co-authorship network of co-authors of William H. Griffith

This figure shows the co-authorship network connecting the top 25 collaborators of William H. Griffith. A scholar is included among the top collaborators of William H. Griffith based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William H. Griffith. William H. Griffith is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Bang, Eunyoung, et al.. (2021). Amitriptyline Decreases GABAergic Transmission in Basal Forebrain Neurons Using an Optogenetic Model of Aging. Frontiers in Aging Neuroscience. 13. 673155–673155. 8 indexed citations
3.
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Janer, Marta, Jinko Graham, Brad McNeney, et al.. (2007). Glutamate Cysteine Ligase Catalytic Subunit Promoter Polymorphisms and Associations with Type 1 Diabetes Age-at-onset and GAD65 Autoantibody Levels. Experimental and Clinical Endocrinology & Diabetes. 115(4). 221–228. 20 indexed citations
5.
Huang, Luping Z., et al.. (2007). Chronic neonatal nicotine increases anxiety but does not impair cognition in adult rats.. Behavioral Neuroscience. 121(6). 1342–1352. 37 indexed citations
6.
LaSarge, Candi L., Karienn S. Montgomery, Catherine Tucker, et al.. (2006). Deficits across multiple cognitive domains in a subset of aged Fischer 344 rats. Neurobiology of Aging. 28(6). 928–936. 58 indexed citations
7.
Murchison, David, et al.. (2005). Functional compensation by other voltage-gated Ca2+ channels in mouse basal forebrain neurons with CaV2.1 mutations. Brain Research. 1140. 105–119. 19 indexed citations
8.
Han, Sun‐Ho, David Murchison, & William H. Griffith. (2005). Low voltage-activated calcium and fast tetrodotoxin-resistant sodium currents define subtypes of cholinergic and noncholinergic neurons in rat basal forebrain. Molecular Brain Research. 134(2). 226–238. 11 indexed citations
9.
Griffith, William H.. (2002). Quest for ion channel modulation by free radicals during brain aging. Neurobiology of Aging. 23(5). 835–836. 1 indexed citations
10.
Murchison, David, Leonard S. Dove, Louise C. Abbott, & William H. Griffith. (2002). Homeostatic compensation maintains Ca 2+ signaling functions in Purkinje neurons in the leaner mutant mouse. The Cerebellum. 1(2). 119–127. 22 indexed citations
11.
Han, Sun‐Ho, Brian A. McCool, David Murchison, et al.. (2002). Single-cell RT-PCR detects shifts in mRNA expression profiles of basal forebrain neurons during aging. Molecular Brain Research. 98(1-2). 67–80. 20 indexed citations
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Griffith, William H., et al.. (1997). Calcium channel types mediating synaptic transmission during aging. Brain Research. 745(1-2). 339–342. 2 indexed citations
14.
Griffith, William H., et al.. (1997). Pharmacological characterization of ionotropic excitatory amino acid receptors in young and aged rat basal forebrain. Neuroscience. 82(4). 1179–1194. 26 indexed citations
15.
Frye, Gerald D., et al.. (1995). Acute ethanol dependence or long-term ethanol treatment and abstinence do not reduce hippocampal responses to carbachol. Alcohol. 12(1). 29–36. 7 indexed citations
16.
Griffith, William H., Joan A. Sim, & Robert T. Matthews. (1991). Electrophysiologic Characteristics of Basal Forebrain Neurons in vitro. Advances in experimental medicine and biology. 295. 143–155. 24 indexed citations
17.
Griffith, William H. & J. A. Sim. (1990). Comparison of 4-aminopyridine and tetrahydroaminoacridine on basal forebrain neurons.. Journal of Pharmacology and Experimental Therapeutics. 255(3). 986–993. 18 indexed citations
18.
Griffith, William H. & Lathrop Taylor. (1988). Phenytoin reduces excitatory synaptic transmission and post-tetanic potentiation in the in vitro hippocampus.. Journal of Pharmacology and Experimental Therapeutics. 246(3). 851–858. 26 indexed citations
19.
Griffith, William H., Judith M. Hills, & David A. Brown. (1988). Substance P‐mediated membrane currents in voltage‐clamped guinea pig inferior mesenteric ganglion cells. Synapse. 2(4). 432–441. 6 indexed citations
20.
Griffith, William H. & Lathrop Taylor. (1988). Sodium valproate decreases synaptic potentiation and epileptiform activity in hippocampus. Brain Research. 474(1). 155–164. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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