William F. Athas
- Cancer Research top 10%
- Oncology
- Molecular Biology
- Pathology and Forensic Medicine top 10%
- Radiation top 5%
- Co-authors
- William C. HuntCharles R. KeyMeg Adams-CameronEvan R. FarmerMohammad HedayatiLawrence I. GrossmanGenevieve M. MatanoskiH. Kubiński
- Topics
- Global Cancer Incidence and Screening (2 papers)Breast Cancer Treatment Studies (2 papers)Nonmelanoma Skin Cancer Studies (1 paper)
- Cited by
- Cancer ResearchRadiationDermatology
- Journals
- JNCI Journal of the National Cancer InstituteEcotoxicology and Environmental SafetyGeospatial health
- Partner nations
- United States
In The Last Decade
William F. Athas
5 papers receiving 614 citations
Peers
Comparison fields: 5 of 76
- Cancer Research 306
- Oncology 222
- Molecular Biology 153
- Pathology and Forensic Medicine 122
- Radiation 120
Countries citing papers authored by William F. Athas
This map shows the geographic impact of William F. Athas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William F. Athas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William F. Athas more than expected).
Fields of papers citing papers by William F. Athas
This network shows the impact of papers produced by William F. Athas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William F. Athas. The network helps show where William F. Athas may publish in the future.
Co-authorship network of co-authors of William F. Athas
This figure shows the co-authorship network connecting the top 25 collaborators of William F. Athas. A scholar is included among the top collaborators of William F. Athas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William F. Athas. William F. Athas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 29 | |
| 2 | Changes in nonmelanoma skin cancer incidence between 1977-1978 and 1998-1999 in Northcentral New Mexico. | 67 |
| 3 | 342 | |
| 4 | Development and field-test validation of an assay for DNA repair in circulating human lymphocytes. | 168 |
| 5 | 24 |
About William F. Athas
William F. Athas is a scholar working on Cancer Research, Dermatology and Oncology, having authored 5 papers that have together received 630 indexed citations. Recurring topics across this work include Global Cancer Incidence and Screening (2 papers), Breast Cancer Treatment Studies (2 papers) and Nonmelanoma Skin Cancer Studies (1 paper). The work is most often cited by research in Cancer Research (306 citations), Radiation (120 citations) and Dermatology (77 citations). William F. Athas has collaborated with scholars based in United States. Frequent co-authors include William C. Hunt, Charles R. Key, Meg Adams-Cameron, Evan R. Farmer, Mohammad Hedayati, Lawrence I. Grossman, Genevieve M. Matanoski, H. Kubiński, Yan Lin and Michael C. Wimberly. Their work appears in journals such as JNCI Journal of the National Cancer Institute, Ecotoxicology and Environmental Safety and Geospatial health.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.