William D. Andrews

2.4k total citations
41 papers, 1.5k citations indexed

About

William D. Andrews is a scholar working on Cellular and Molecular Neuroscience, Developmental Neuroscience and Molecular Biology. According to data from OpenAlex, William D. Andrews has authored 41 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Cellular and Molecular Neuroscience, 15 papers in Developmental Neuroscience and 14 papers in Molecular Biology. Recurrent topics in William D. Andrews's work include Axon Guidance and Neuronal Signaling (20 papers), Neurogenesis and neuroplasticity mechanisms (15 papers) and Corporate Taxation and Avoidance (8 papers). William D. Andrews is often cited by papers focused on Axon Guidance and Neuronal Signaling (20 papers), Neurogenesis and neuroplasticity mechanisms (15 papers) and Corporate Taxation and Avoidance (8 papers). William D. Andrews collaborates with scholars based in United Kingdom, United States and Japan. William D. Andrews's co-authors include John G. Parnavelas, Melissa Barber, Vasi Sundaresan, Sonja Rakić, Clare Faux, Anna Cariboni, Joanne M. Britto, Athéna R. Ypsilanti, Sarah Guthrie and Alain Chédotal and has published in prestigious journals such as Nature, Nature Communications and Journal of Neuroscience.

In The Last Decade

William D. Andrews

39 papers receiving 1.4k citations

Peers

William D. Andrews
W. Distler Germany
Abigail M. Garner United States
Raymond M. Esper United States
Michael J. McConnell United States
Stacy L. Donovan United States
W. Distler Germany
William D. Andrews
Citations per year, relative to William D. Andrews William D. Andrews (= 1×) peers W. Distler

Countries citing papers authored by William D. Andrews

Since Specialization
Citations

This map shows the geographic impact of William D. Andrews's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William D. Andrews with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William D. Andrews more than expected).

Fields of papers citing papers by William D. Andrews

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William D. Andrews. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William D. Andrews. The network helps show where William D. Andrews may publish in the future.

Co-authorship network of co-authors of William D. Andrews

This figure shows the co-authorship network connecting the top 25 collaborators of William D. Andrews. A scholar is included among the top collaborators of William D. Andrews based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William D. Andrews. William D. Andrews is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Liu, Wenfei, Rui Wang, Sevinç Bayram, et al.. (2020). Trem2 promotes anti-inflammatory responses in microglia and is suppressed under pro-inflammatory conditions. Human Molecular Genetics. 29(19). 3224–3248. 122 indexed citations
2.
Romanov, Roman A., Evgenii O. Tretiakov, Maria Eleni Kastriti, et al.. (2020). Molecular design of hypothalamus development. Nature. 582(7811). 246–252. 102 indexed citations
3.
Yeh, Mason L., Mathilda T.M. Mommersteeg, Melissa Barber, et al.. (2014). Robo1 Modulates Proliferation and Neurogenesis in the Developing Neocortex. Journal of Neuroscience. 34(16). 5717–5731. 41 indexed citations
4.
Alpár, Alán, Giuseppe Tortoriello, Daniela Calvigioni, et al.. (2014). Endocannabinoids modulate cortical development by configuring Slit2/Robo1 signalling. Nature Communications. 5(1). 4421–4421. 56 indexed citations
5.
Andrews, William D., Hidenori Tabata, Takashi Namba, et al.. (2012). Robo1 Regulates the Migration and Laminar Distribution of Upper-Layer Pyramidal Neurons of the Cerebral Cortex. Cerebral Cortex. 23(6). 1495–1508. 37 indexed citations
6.
Geutskens, Sacha B., et al.. (2012). Control of human hematopoietic stem/progenitor cell migration by the extracellular matrix protein Slit3. Laboratory Investigation. 92(8). 1129–1139. 26 indexed citations
7.
Cartelli, Daniele, Graziella Cappelletti, Anna Cariboni, et al.. (2012). Neuritin 1 promotes neuronal migration. Brain Structure and Function. 219(1). 105–118. 30 indexed citations
8.
Faux, Clare, Sonja Rakić, William D. Andrews, & Joanne M. Britto. (2012). Neurons on the Move: Migration and Lamination of Cortical Interneurons. Neurosignals. 20(3). 168–189. 58 indexed citations
9.
Faux, Clare, et al.. (2011). Differential gene expression in migratory streams of cortical interneurons. European Journal of Neuroscience. 34(10). 1584–1594. 36 indexed citations
10.
Faux, Clare, Sonja Rakić, William D. Andrews, et al.. (2009). Differential gene expression in migrating cortical interneurons during mouse forebrain development. The Journal of Comparative Neurology. 518(8). 1232–1248. 34 indexed citations
11.
Barber, Melissa, Thomas Di Meglio, William D. Andrews, et al.. (2009). The Role of Robo3 in the Development of Cortical Interneurons. Cerebral Cortex. 19(suppl_1). i22–i31. 30 indexed citations
12.
Thompson, Hannah, William D. Andrews, John G. Parnavelas, & Lynda Erskine. (2009). Robo2 is required for Slit-mediated intraretinal axon guidance. Developmental Biology. 335(2). 418–426. 26 indexed citations
13.
Andrews, William D., Melissa Barber, & John G. Parnavelas. (2007). Slit–Robo interactions during cortical development. Journal of Anatomy. 211(2). 188–198. 70 indexed citations
14.
Plachez, Céline, William D. Andrews, Anastasia Liapi, et al.. (2007). Robos are required for the correct targeting of retinal ganglion cell axons in the visual pathway of the brain. Molecular and Cellular Neuroscience. 37(4). 719–730. 36 indexed citations
15.
Herring, Belinda L., Flavien Bernardin, Sally Caglioti, et al.. (2007). Phylogenetic analysis of WNV in North American blood donors during the 2003–2004 epidemic seasons. Virology. 363(1). 220–228. 29 indexed citations
16.
Hammond, Rachel, Arifa Naeem, John K. Chilton, et al.. (2005). Slit-mediated repulsion is a key regulator of motor axon pathfinding in the hindbrain. Development. 132(20). 4483–4495. 64 indexed citations
17.
Sundaresan, Vasi, et al.. (2003). Dynamic expression patterns of Robo (Robo1 and Robo2) in the developing murine central nervous system. The Journal of Comparative Neurology. 468(4). 467–481. 42 indexed citations
18.
Andrews, William D., P. W. Tuke, Ammar Al‐Chalabi, et al.. (2000). Detection of reverse transcriptase activity in the serum of patients with motor neurone disease. Journal of Medical Virology. 61(4). 527–532. 54 indexed citations
19.
Andrews, William D., Ammar Al‐Chalabi, & Jeremy A. Garson. (1997). Lack of evidence for HTLV tax-rex DNA in motor neurone disease. Journal of the Neurological Sciences. 153(1). 86–90. 12 indexed citations
20.
Andrews, William D., et al.. (1969). Federal estate and gift taxation : recommendations adopted by the American Law Institute at Washington, D.C., May 23-24, 1968, and reporters' studies. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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