William C. Unrath

672 total citations
16 papers, 528 citations indexed

About

William C. Unrath is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Epidemiology. According to data from OpenAlex, William C. Unrath has authored 16 papers receiving a total of 528 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 10 papers in Cardiology and Cardiovascular Medicine and 6 papers in Epidemiology. Recurrent topics in William C. Unrath's work include Cardiomyopathy and Myosin Studies (10 papers), Muscle Physiology and Disorders (9 papers) and Pneumonia and Respiratory Infections (5 papers). William C. Unrath is often cited by papers focused on Cardiomyopathy and Myosin Studies (10 papers), Muscle Physiology and Disorders (9 papers) and Pneumonia and Respiratory Infections (5 papers). William C. Unrath collaborates with scholars based in United States, United Kingdom and Switzerland. William C. Unrath's co-authors include Christopher M. Yengo, Kevin L. Nelson, Omar A. Quintero, Arnold L. Smith, Alice L. Erwin, Bechara Kachar, Uri Manor, M’hamed Grati, Felipe T. Salles and Michael A. Apicella and has published in prestigious journals such as Journal of Biological Chemistry, Current Biology and Journal of Bacteriology.

In The Last Decade

William C. Unrath

16 papers receiving 526 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
William C. Unrath United States	13	262	173	172	142	56	16	528
Helene H. Jensen Denmark	13	325 1.2×	52 0.3×	45 0.3×	49 0.3×	31 0.6×	30	566
Martin Friis Denmark	8	128 0.5×	45 0.3×	35 0.2×	77 0.5×	59 1.1×	12	308
Nicholas J. Palmer United States	11	214 0.8×	36 0.2×	87 0.5×	59 0.4×	50 0.9×	19	402
Qiangming Sun China	12	143 0.5×	31 0.2×	59 0.3×	105 0.7×	20 0.4×	29	386
Cedar Fowler United States	11	154 0.6×	9 0.1×	18 0.1×	109 0.8×	58 1.0×	28	486
Christian M. Bruell Switzerland	8	442 1.7×	50 0.3×	27 0.2×	67 0.5×	2 0.0×	8	557
Wafa Kammouni Canada	14	200 0.8×	20 0.1×	12 0.1×	95 0.7×	24 0.4×	29	521
Tatjana Williams Germany	14	229 0.9×	6 0.0×	190 1.1×	80 0.6×	17 0.3×	30	570
P. Montanari Italy	13	148 0.6×	123 0.7×	22 0.1×	82 0.6×	16 0.3×	32	541
Jan G. Dohlman United States	7	437 1.7×	119 0.7×	29 0.2×	32 0.2×	34 0.6×	9	673

Countries citing papers authored by William C. Unrath

Since Specialization

Citations

This map shows the geographic impact of William C. Unrath's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William C. Unrath with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William C. Unrath more than expected).

Fields of papers citing papers by William C. Unrath

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William C. Unrath. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William C. Unrath. The network helps show where William C. Unrath may publish in the future.

Co-authorship network of co-authors of William C. Unrath

This figure shows the co-authorship network connecting the top 25 collaborators of William C. Unrath. A scholar is included among the top collaborators of William C. Unrath based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William C. Unrath. William C. Unrath is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

1.

Unrath, William C., et al.. (2021). Cardiomyopathy mutations impact the actin-activated power stroke of human cardiac myosin. Biophysical Journal. 120(11). 2222–2236. 16 indexed citations

2.

Unrath, William C., et al.. (2019). Dilated cardiomyopathy mutation in the converter domain of human cardiac myosin alters motor activity and response to omecamtiv mecarbil. Journal of Biological Chemistry. 294(46). 17314–17325. 17 indexed citations

3.

Rohde, John A., William C. Unrath, Darshan V. Trivedi, et al.. (2018). Converter domain mutations in myosin alter structural kinetics and motor function. Journal of Biological Chemistry. 294(5). 1554–1567. 14 indexed citations

4.

Blair, Cheavar A., et al.. (2017). Omecamtiv Mecarbil Enhances the Duty Ratio of Human β-Cardiac Myosin Resulting in Increased Calcium Sensitivity and Slowed Force Development in Cardiac Muscle. Journal of Biological Chemistry. 292(9). 3768–3778. 69 indexed citations

5.

Quintero, Omar A., William C. Unrath, James W. Gallagher, et al.. (2016). Impact of the Motor and Tail Domains of Class III Myosins on Regulating the Formation and Elongation of Actin Protrusions. Journal of Biological Chemistry. 291(43). 22781–22792. 12 indexed citations

6.

Blair, Cheavar A., et al.. (2016). Omecamtiv Mecarbil Enhances Actin Affinity and Slows Force Production in Human β-Cardiac Myosin. Biophysical Journal. 110(3). 297a–297a. 1 indexed citations

7.

Quintero, Omar A., William C. Unrath, Stanley M. Stevens, et al.. (2013). Myosin 3A Kinase Activity Is Regulated by Phosphorylation of the Kinase Domain Activation Loop. Journal of Biological Chemistry. 288(52). 37126–37137. 26 indexed citations

8.

Adikes, Rebecca C., William C. Unrath, Christopher M. Yengo, & Omar A. Quintero. (2013). Biochemical and bioinformatic analysis of the myosin‐XIX motor domain. Cytoskeleton. 70(5). 281–295. 23 indexed citations

9.

Manor, Uri, Felipe T. Salles, M’hamed Grati, et al.. (2012). Myosin IIIB Uses an Actin-Binding Motif in Its Espin-1 Cargo to Reach the Tips of Actin Protrusions. Current Biology. 22(4). 320–325. 56 indexed citations

10.

Quintero, Omar A., William C. Unrath, Uri Manor, et al.. (2010). Intermolecular Autophosphorylation Regulates Myosin IIIa Activity and Localization in Parallel Actin Bundles. Journal of Biological Chemistry. 285(46). 35770–35782. 34 indexed citations

11.

Smith, Arnold L., Alice L. Erwin, Toni Kline, et al.. (2007). Chloramphenicol Is a Substrate for a Novel Nitroreductase Pathway in Haemophilus influenzae. Antimicrobial Agents and Chemotherapy. 51(8). 2820–2829. 32 indexed citations

12.

Erwin, Alice L., Sara A. Sandstedt, Paul J. Bonthuis, et al.. (2007). Analysis of Genetic Relatedness of Haemophilus influenzae Isolates by Multilocus Sequence Typing. Journal of Bacteriology. 190(4). 1473–1483. 59 indexed citations

13.

Erwin, Alice L., Simon Allen, Derek K. Ho, et al.. (2006). Role of lgtC in Resistance of Nontypeable Haemophilus influenzae Strain R2866 to Human Serum. Infection and Immunity. 74(12). 7047–7047. 1 indexed citations

14.

Erwin, Alice L., Simon Allen, Derek K. Ho, et al.. (2006). Role of lgtC in Resistance of Nontypeable Haemophilus influenzae Strain R2866 to Human Serum. Infection and Immunity. 74(11). 6226–6235. 42 indexed citations

15.

Daines, Dayle A., Marcella Bothwell, Jason L. Furrer, et al.. (2005). Haemophilus influenzae luxS mutants form a biofilm and have increased virulence. Microbial Pathogenesis. 39(3). 87–96. 54 indexed citations

16.

Erwin, Alice L., Kevin L. Nelson, Tendai Mhlanga-Mutangadura, et al.. (2005). Characterization of Genetic and Phenotypic Diversity of Invasive Nontypeable Haemophilus influenzae. Infection and Immunity. 73(9). 5853–5863. 72 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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