Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
Impaired myocardial angiogenesis and ischemic cardiomyopathy in mice lacking the vascular endothelial growth factor isoforms VEGF164 and VEGF188
Countries citing papers authored by Willem Flameng
Since
Specialization
Citations
This map shows the geographic impact of Willem Flameng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Willem Flameng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Willem Flameng more than expected).
This network shows the impact of papers produced by Willem Flameng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Willem Flameng. The network helps show where Willem Flameng may publish in the future.
Co-authorship network of co-authors of Willem Flameng
This figure shows the co-authorship network connecting the top 25 collaborators of Willem Flameng.
A scholar is included among the top collaborators of Willem Flameng based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with Willem Flameng. Willem Flameng is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Visscher, Geofrey De, et al.. (2007). Homing, signaling and structural molecule gene expression profiling in the early phase foreign body reaction. Wound Repair and Regeneration. 15(6).2 indexed citations
3.
Visscher, Geofrey De, Eric Verbeken, Hans Van Oosterwyck, et al.. (2007). Abstract 2060: Enhanced in situ Recellularization by Fibronectin and Stromal Cell Derived Factor-1alpha Coating of Heart Valves Acquires a Quasi-Native Valve Composition of Cell Phenotypes.. Circulation. 116. 446.
4.
Dumont, Kris, Patrick Segers, Stijn Vandenberghe, et al.. (2001). New pulsatile bioreactor for the in vitro formation of tissue engineered heart valves. The International Journal of Artificial Organs. 24(8). 557.2 indexed citations
5.
Mesotten, Liesbet, Xuesong Liu, Johan Nuyts, et al.. (2001). Transition from chronic stunning to hibernation and further on to necrosis in a porcine model of chronic ischemia identified with PET. Circulation. 104(17). 455–455.1 indexed citations
6.
Meuris, Bart, et al.. (2000). Transport of vein material for coating auto-xenograft. The International Journal of Artificial Organs. 23(8). 582.1 indexed citations
7.
Flameng, Willem, et al.. (2000). Dissociation of cardiomyocyte apoptosis and cardiomyocyte dedifferentiation in infarct border zones. Circulation. 102(18). 343–343.5 indexed citations
8.
Ozaki, Shigeyuki, Eric Verbeken, Jessa Yperman, et al.. (2000). Tissue engineered auto-xenograft heart valve. Cardiovascular Surgery. 8. 36.2 indexed citations
9.
Ziętkiewicz, Mirosław, Bartłomiej Perek, Alex Maes, et al.. (1999). Reversed mismatch preceeds match patterns using PET analysis in failing myocardium with patchy necrosis. Circulation. 100(18). 88–88.4 indexed citations
10.
Štengl, Milan, Kanigula Mubagwa, Edward Carmeliet, & Willem Flameng. (1997). Phenylephrine increases sodium-calcium exchange current in rat ventricular myocytes. Biophysical Journal. 72(2).1 indexed citations
Shivalkar, Bharati, Michael A. Borger, Alex Maes, Luc Mortelmans, & Willem Flameng. (1994). Low regional function-associated with high metabolism predicts functional recovery after coronary-bypass surgery. Circulation. 90(4). 251–251.7 indexed citations
13.
Marchal, Guy, Carine Petré, Gregorius Lukito, et al.. (1994). Metalloporphyrin enhanced magnetic-resonance-imaging of acute myocardial-infarction. Circulation. 90(4). 468–468.10 indexed citations
14.
Mubagwa, Kanigula, et al.. (1994). Alpha-1a adrenergic receptor-mediated negative inotropic action in rat cardiac-muscle. Pflügers Archiv - European Journal of Physiology. 426.1 indexed citations
15.
Beck, Marcus, et al.. (1993). Cardiovascular effects of pregnanolone emulsion in chronically instrumented dogs. Anesthesia & Analgesia. 76(2).1 indexed citations
16.
Beck, Marcus, et al.. (1993). Cardiovascular effects of propofol in chronically instrumented dogs. Anesthesia & Analgesia. 76(2).1 indexed citations
17.
Flameng, Willem, et al.. (1980). Pharmacologic protection of the myocardium during one hour normothermic global-ischemia. Circulation. 62(4). 323–323.5 indexed citations
18.
Flameng, Willem, Bernd Winkler, B. Wüsten, & W. Schäper. (1977). Minimum requirements for the measurement of regional myocardial flow using tracer microspheres.. PubMed. 24–9.7 indexed citations
19.
Schwarz, Franz, et al.. (1976). Correlation between left-ventricular function and myocardial ultrastructure in patients with aortic-valve disease. Circulation. 54(4). 67–67.6 indexed citations
20.
Flameng, Willem & B. Wüsten. (1974). Postischemic reactive hyperemia in dogs with multiple chronic coronary-artery occlusion without infarction. Circulation. 50(4). 173–173.2 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.