Wen‐Wu Bai

758 total citations
23 papers, 570 citations indexed

About

Wen‐Wu Bai is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Surgery. According to data from OpenAlex, Wen‐Wu Bai has authored 23 papers receiving a total of 570 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 8 papers in Cardiology and Cardiovascular Medicine and 5 papers in Surgery. Recurrent topics in Wen‐Wu Bai's work include Cardiac Fibrosis and Remodeling (4 papers), Cardiovascular Function and Risk Factors (3 papers) and Angiogenesis and VEGF in Cancer (3 papers). Wen‐Wu Bai is often cited by papers focused on Cardiac Fibrosis and Remodeling (4 papers), Cardiovascular Function and Risk Factors (3 papers) and Angiogenesis and VEGF in Cancer (3 papers). Wen‐Wu Bai collaborates with scholars based in China, Bangladesh and Thailand. Wen‐Wu Bai's co-authors include Yifan Xing, Xiaoting Lu, Xiaoqiong Liu, Qing Yang, Bo Wang, Yuxia Zhao, Yuanyuan Sun, Tao Guo, Weidong Qin and Xiaoting Lu and has published in prestigious journals such as Nature Communications, PLoS ONE and Hypertension.

In The Last Decade

Wen‐Wu Bai

23 papers receiving 568 citations

Peers

Wen‐Wu Bai
Wen‐Wu Bai
Citations per year, relative to Wen‐Wu Bai Wen‐Wu Bai (= 1×) peers Weijing Liu

Countries citing papers authored by Wen‐Wu Bai

Since Specialization
Citations

This map shows the geographic impact of Wen‐Wu Bai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wen‐Wu Bai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wen‐Wu Bai more than expected).

Fields of papers citing papers by Wen‐Wu Bai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wen‐Wu Bai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wen‐Wu Bai. The network helps show where Wen‐Wu Bai may publish in the future.

Co-authorship network of co-authors of Wen‐Wu Bai

This figure shows the co-authorship network connecting the top 25 collaborators of Wen‐Wu Bai. A scholar is included among the top collaborators of Wen‐Wu Bai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wen‐Wu Bai. Wen‐Wu Bai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wang, Xueqing, Sen Yin, Qianwen Wang, et al.. (2025). Vitamin B6 allosterically activates AMPK to promote postischemic angiogenesis in mice. European Journal of Pharmacology. 993. 177413–177413. 1 indexed citations
2.
Song, Jiawen, Zhenshan Zhang, Qianwen Wang, et al.. (2024). Vitamin B-6 Prevents Heart Failure with Preserved Ejection Fraction Through Downstream of Kinase 3 in a Mouse Model. Journal of Nutrition. 154(10). 3031–3041. 3 indexed citations
3.
Li, Bin, Wen‐Wu Bai, Tao Guo, et al.. (2024). Statins improve cardiac endothelial function to prevent heart failure with preserved ejection fraction through upregulating circRNA-RBCK1. Nature Communications. 15(1). 2953–2953. 27 indexed citations
4.
Wang, Qianwen, et al.. (2024). Circulating monocyte adhesion repairs endothelium-denuded injury through downstream of kinase 3-mediated endothelialization. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1871(3). 167631–167631. 2 indexed citations
5.
Bai, Wen‐Wu, Tao Guo, Han Wang, et al.. (2023). S-nitrosylation of AMPKγ impairs coronary collateral circulation and disrupts VSMC reprogramming. EMBO Reports. 25(1). 128–143. 12 indexed citations
6.
Shi, Shuai, et al.. (2022). Prognostic significance of SPARC expression in non‑small cell lung cancer: A meta‑analysis and bioinformatics analysis. Oncology Letters. 24(5). 412–412. 4 indexed citations
7.
Qin, Weidong, Wen‐Wu Bai, Keyin Liu, et al.. (2021). Clinical Course and Risk Factors of Disease Deterioration in Critically Ill Patients with COVID-19. Human Gene Therapy. 32(5-6). 310–315. 23 indexed citations
8.
Wang, Bo, Haijia Xu, Jing Kong, et al.. (2021). Krüppel-Like Factor 15 Reduces Ischemia-Induced Apoptosis Involving Regulation of p38/MAPK Signaling. Human Gene Therapy. 32(23-24). 1471–1480. 13 indexed citations
9.
Bai, Wen‐Wu, Hao Wang, Fan Jiang, et al.. (2021). Continuous Infusion of Angiotensin IV Protects against Acute Myocardial Infarction via the Inhibition of Inflammation and Autophagy. Oxidative Medicine and Cellular Longevity. 2021(1). 2860488–2860488. 13 indexed citations
10.
Bai, Wen‐Wu, Min Ren, Wen Cheng, et al.. (2021). Qindan Capsule Attenuates Myocardial Hypertrophy and Fibrosis in Pressure Overload-Induced Mice Involving mTOR and TGF-β1/Smad Signaling Pathway Inhibition. Evidence-based Complementary and Alternative Medicine. 2021. 1–11. 5 indexed citations
11.
Shan, Meirong, Shengnan Zhou, Jiawen Song, et al.. (2020). Vitamin B6 inhibits macrophage activation to prevent lipopolysaccharide‐induced acute pneumonia in mice. Journal of Cellular and Molecular Medicine. 24(5). 3139–3148. 32 indexed citations
12.
Bai, Wen‐Wu, Zhengyu Tang, Peng Li, et al.. (2019). Upregulation of Paired-Related Homeobox 2 Promotes Cardiac Fibrosis in Mice Following Myocardial Infarction by Targeting of Wnt5a. SSRN Electronic Journal. 1 indexed citations
13.
Liang, Er‐shun, Wen‐Wu Bai, Hao Wang, et al.. (2018). PARP-1 (Poly[ADP-Ribose] Polymerase 1) Inhibition Protects From Ang II (Angiotensin II)–Induced Abdominal Aortic Aneurysm in Mice. Hypertension. 72(5). 1189–1199. 34 indexed citations
14.
Yin, Sen, Wen‐Wu Bai, Peng Li, et al.. (2018). Berberine suppresses the ectopic expression of miR-133a in endothelial cells to improve vascular dementia in diabetic rats. Clinical and Experimental Hypertension. 41(8). 708–716. 20 indexed citations
15.
Liang, Er‐shun, et al.. (2017). Peptidyl-prolyl isomerase Pin1 deficiency attenuates angiotensin II-induced abdominal aortic aneurysm formation in ApoE−/− mice. Journal of Molecular and Cellular Cardiology. 114. 334–344. 14 indexed citations
16.
Qin, Tao, Yuanyuan Sun, Wen‐Wu Bai, et al.. (2014). Period2 Deficiency Blunts Hypoxia-Induced Mobilization and Function of Endothelial Progenitor Cells. PLoS ONE. 9(9). e108806–e108806. 6 indexed citations
17.
Wang, Bo, Qing Yang, Wen‐Wu Bai, et al.. (2014). Tongxinluo Protects against Pressure Overload–Induced Heart Failure in Mice Involving VEGF/Akt/eNOS Pathway Activation. PLoS ONE. 9(6). e98047–e98047. 44 indexed citations
18.
Wang, Bo, Qing Yang, Yifan Xing, et al.. (2014). Resveratrol‐enhanced autophagic flux ameliorates myocardial oxidative stress injury in diabetic mice. Journal of Cellular and Molecular Medicine. 18(8). 1599–1611. 197 indexed citations
19.
Bai, Wen‐Wu, Yifan Xing, Bo Wang, et al.. (2013). Tongxinluo Improves Cardiac Function and Ameliorates Ventricular Remodeling in Mice Model of Myocardial Infarction through Enhancing Angiogenesis. Evidence-based Complementary and Alternative Medicine. 2013. 1–9. 43 indexed citations
20.
Lu, Xiaoting, Yunfang Liu, Lei Zhang, et al.. (2012). Unpredictable Chronic Mild Stress Promotes Atherosclerosis in High Cholesterol–Fed Rabbits. Psychosomatic Medicine. 74(6). 604–611. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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