Weiwen Yan

917 total citations
21 papers, 706 citations indexed

About

Weiwen Yan is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology and Cancer Research. According to data from OpenAlex, Weiwen Yan has authored 21 papers receiving a total of 706 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Pulmonary and Respiratory Medicine, 6 papers in Molecular Biology and 6 papers in Cancer Research. Recurrent topics in Weiwen Yan's work include Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (13 papers), Occupational and environmental lung diseases (8 papers) and Cancer-related molecular mechanisms research (5 papers). Weiwen Yan is often cited by papers focused on Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (13 papers), Occupational and environmental lung diseases (8 papers) and Cancer-related molecular mechanisms research (5 papers). Weiwen Yan collaborates with scholars based in China, Philippines and North Macedonia. Weiwen Yan's co-authors include Chunhui Ni, Jiali Yuan, Qiuyun Wu, Xiaoming Ji, Wenxi Yao, Lei Han, Jingjin Yang, Ruhui Han, Yi Liu and Qi Xu and has published in prestigious journals such as Scientific Reports, International Journal of Molecular Sciences and Gene.

In The Last Decade

Weiwen Yan

19 papers receiving 704 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Weiwen Yan China 14 334 316 305 73 63 21 706
Wenxi Yao China 12 260 0.8× 288 0.9× 218 0.7× 69 0.9× 48 0.8× 17 590
Yating Peng China 9 222 0.7× 304 1.0× 68 0.2× 61 0.8× 57 0.9× 24 629
Kang He China 16 263 0.8× 48 0.2× 181 0.6× 47 0.6× 35 0.6× 59 541
Xudong Shen China 13 232 0.7× 122 0.4× 138 0.5× 63 0.9× 24 0.4× 41 492
Fahd Alhamdan Germany 13 248 0.7× 124 0.4× 64 0.2× 32 0.4× 165 2.6× 21 582
Andrea C. Schamberger Germany 11 221 0.7× 295 0.9× 88 0.3× 46 0.6× 50 0.8× 18 564
Ihab Hassan United States 7 181 0.5× 87 0.3× 97 0.3× 123 1.7× 87 1.4× 8 439
Yanhui Jia China 8 253 0.8× 109 0.3× 110 0.4× 40 0.5× 61 1.0× 13 449
Na Mao China 13 127 0.4× 209 0.7× 55 0.2× 17 0.2× 46 0.7× 33 397

Countries citing papers authored by Weiwen Yan

Since Specialization
Citations

This map shows the geographic impact of Weiwen Yan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Weiwen Yan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Weiwen Yan more than expected).

Fields of papers citing papers by Weiwen Yan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Weiwen Yan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Weiwen Yan. The network helps show where Weiwen Yan may publish in the future.

Co-authorship network of co-authors of Weiwen Yan

This figure shows the co-authorship network connecting the top 25 collaborators of Weiwen Yan. A scholar is included among the top collaborators of Weiwen Yan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Weiwen Yan. Weiwen Yan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wang, Luyao, Xinyu Li, Wenjuan Wang, et al.. (2025). Bisphenol F disrupts lipophagy and lysosomal acidification via ATGL-SIRT1-PPARα signaling in NAFLD-like hepatic changes. Ecotoxicology and Environmental Safety. 303. 119000–119000.
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Yao, Yuan, Chunlai Zhang, Wei Gao, et al.. (2023). Pyruvate dehydrogenase kinase 1 protects against neuronal injury and memory loss in mouse models of diabetes. Cell Death and Disease. 14(11). 722–722. 21 indexed citations
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Yan, Weiwen, Dongyu Ma, Yi Liu, et al.. (2022). PTX3 alleviates hard metal-induced acute lung injury through potentiating efferocytosis. Ecotoxicology and Environmental Safety. 230. 113139–113139. 8 indexed citations
7.
Wu, Qiuyun, et al.. (2020). MiR‐503 suppresses fibroblast activation and myofibroblast differentiation by targeting VEGFA and FGFR1 in silica‐induced pulmonary fibrosis. Journal of Cellular and Molecular Medicine. 24(24). 14339–14348. 21 indexed citations
8.
Xu, Qi, Yi Liu, Honghong Pan, et al.. (2019). Aberrant expression of miR-125a-3p promotes fibroblast activation via Fyn/STAT3 pathway during silica-induced pulmonary fibrosis. Toxicology. 414. 57–67. 28 indexed citations
9.
Xu, Tiantian, Weiwen Yan, Qiuyun Wu, et al.. (2019). MiR-326 Inhibits Inflammation and Promotes Autophagy in Silica-Induced Pulmonary Fibrosis through Targeting TNFSF14 and PTBP1. Chemical Research in Toxicology. 32(11). 2192–2203. 52 indexed citations
10.
Shi, Xiaolei, Long Yan, Chunyan Yang, et al.. (2018). Identification of a major quantitative trait locus underlying salt tolerance in ‘Jidou 12’ soybean cultivar. BMC Research Notes. 11(1). 95–95. 19 indexed citations
11.
Han, Lei, Weiwen Yan, Shuping Wang, et al.. (2018). Quality of life and influencing factors of coal miners in Xuzhou, China. Journal of Thoracic Disease. 10(2). 835–844. 20 indexed citations
12.
Yao, Wenxi, Yan Li, Lei Han, et al.. (2018). The CDR1as/miR-7/TGFBR2 Axis Modulates EMT in Silica-Induced Pulmonary Fibrosis. Toxicological Sciences. 166(2). 465–478. 93 indexed citations
13.
Yuan, Jiali, Ruhui Han, Qiuyun Wu, et al.. (2017). Polymorphisms in autophagy related genes and the coal workers' pneumoconiosis in a Chinese population. Gene. 632. 36–42. 17 indexed citations
14.
Yan, Weiwen, Qiuyun Wu, Wenxi Yao, et al.. (2017). MiR-503 modulates epithelial-mesenchymal transition in silica-induced pulmonary fibrosis by targeting PI3K p85 and is sponged by lncRNA MALAT1. Scientific Reports. 7(1). 11313–11313. 91 indexed citations
15.
Liu, Yi, Yan Li, Qi Xu, et al.. (2017). Long non-coding RNA-ATB promotes EMT during silica-induced pulmonary fibrosis by competitively binding miR-200c. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1864(2). 420–431. 84 indexed citations
16.
Wu, Qiuyun, Tiantian Xu, Yi Liu, et al.. (2017). miR-1224-5p Mediates Mitochondrial Damage to Affect Silica-Induced Pulmonary Fibrosis by Targeting BECN1. International Journal of Molecular Sciences. 18(11). 2357–2357. 26 indexed citations
17.
Yang, Jingjin, Ting Wang, Yan Li, et al.. (2016). Earthworm extract attenuates silica-induced pulmonary fibrosis through Nrf2-dependent mechanisms. Laboratory Investigation. 96(12). 1279–1300. 33 indexed citations
18.
Wu, Qiuyun, Lei Han, Weiwen Yan, et al.. (2016). miR-489 inhibits silica-induced pulmonary fibrosis by targeting MyD88 and Smad3 and is negatively regulated by lncRNA CHRF. Scientific Reports. 6(1). 30921–30921. 87 indexed citations
19.
Han, Ruhui, Xiaoming Ji, Rong Rong, et al.. (2016). MiR-449a regulates autophagy to inhibit silica-induced pulmonary fibrosis through targeting Bcl2. Journal of Molecular Medicine. 94(11). 1267–1279. 82 indexed citations
20.
Wu, Qiuyun, Weiwen Yan, Ruhui Han, et al.. (2016). Polymorphisms in Long Noncoding RNA H19 Contribute to the Protective Effects of Coal Workers’ Pneumoconiosis in a Chinese Population. International Journal of Environmental Research and Public Health. 13(9). 903–903. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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