Weiwen Long

2.0k total citations
36 papers, 1.5k citations indexed

About

Weiwen Long is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Weiwen Long has authored 36 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Molecular Biology, 11 papers in Oncology and 7 papers in Cancer Research. Recurrent topics in Weiwen Long's work include Melanoma and MAPK Pathways (12 papers), Protein Kinase Regulation and GTPase Signaling (9 papers) and PI3K/AKT/mTOR signaling in cancer (7 papers). Weiwen Long is often cited by papers focused on Melanoma and MAPK Pathways (12 papers), Protein Kinase Regulation and GTPase Signaling (9 papers) and PI3K/AKT/mTOR signaling in cancer (7 papers). Weiwen Long collaborates with scholars based in United States, China and Japan. Weiwen Long's co-authors include Bert W. O’Malley, Frank E. Jones, David M. Lonard, Larbi Amazit, Xiaotao Li, John J. Monaco, Ming‐Jer Tsai, Sophia Y. Tsai, Lobna Elkhadragy and Lothar Hennighausen and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and The Journal of Cell Biology.

In The Last Decade

Weiwen Long

36 papers receiving 1.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Weiwen Long United States 22 1.1k 567 313 199 179 36 1.5k
Vimla Band United States 17 1.1k 1.0× 618 1.1× 351 1.1× 183 0.9× 186 1.0× 23 1.7k
Abbas Fotovati Canada 24 1.2k 1.1× 529 0.9× 320 1.0× 98 0.5× 181 1.0× 35 1.7k
Maria Letizia Motti Italy 21 1.2k 1.1× 757 1.3× 308 1.0× 118 0.6× 174 1.0× 42 1.8k
George K. Belka United States 19 1.1k 1.1× 774 1.4× 472 1.5× 291 1.5× 120 0.7× 26 1.9k
Xiao-Feng Le United States 21 910 0.8× 462 0.8× 274 0.9× 105 0.5× 99 0.6× 23 1.3k
Mei‐Ren Pan Taiwan 27 1.3k 1.2× 691 1.2× 410 1.3× 164 0.8× 116 0.6× 58 1.9k
Kevin Kwei United States 17 907 0.8× 598 1.1× 412 1.3× 165 0.8× 99 0.6× 27 1.6k
Chanel E. Smart Australia 19 988 0.9× 433 0.8× 576 1.8× 114 0.6× 116 0.6× 40 1.5k
Gretchen Argast United States 17 1.1k 1.0× 357 0.6× 223 0.7× 128 0.6× 181 1.0× 26 1.6k
Francesco Trapasso Italy 26 1.5k 1.4× 375 0.7× 312 1.0× 330 1.7× 179 1.0× 74 2.0k

Countries citing papers authored by Weiwen Long

Since Specialization
Citations

This map shows the geographic impact of Weiwen Long's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Weiwen Long with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Weiwen Long more than expected).

Fields of papers citing papers by Weiwen Long

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Weiwen Long. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Weiwen Long. The network helps show where Weiwen Long may publish in the future.

Co-authorship network of co-authors of Weiwen Long

This figure shows the co-authorship network connecting the top 25 collaborators of Weiwen Long. A scholar is included among the top collaborators of Weiwen Long based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Weiwen Long. Weiwen Long is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Elkhadragy, Lobna, et al.. (2024). Role of the Atypical MAPK ERK3 in Cancer Growth and Progression. Cancers. 16(7). 1381–1381. 3 indexed citations
2.
3.
Morel, Marion, et al.. (2023). ERK3 and DGKζ interact to modulate cell motility in lung cancer cells. Frontiers in Cell and Developmental Biology. 11. 1192221–1192221. 2 indexed citations
4.
Vallabhaneni, Sreeram, Jian Liu, Marion Morel, et al.. (2021). Conditional ERK3 overexpression cooperates with PTEN deletion to promote lung adenocarcinoma formation in mice. Molecular Oncology. 16(5). 1184–1199. 4 indexed citations
5.
Morel, Marion, et al.. (2020). The F-box protein FBXL16 up-regulates the stability of C-MYC oncoprotein by antagonizing the activity of the F-box protein FBW7. Journal of Biological Chemistry. 295(23). 7970–7980. 22 indexed citations
6.
Elkhadragy, Lobna, et al.. (2020). The C-Terminus Tail Regulates ERK3 Kinase Activity and Its Ability in Promoting Cancer Cell Migration and Invasion. International Journal of Molecular Sciences. 21(11). 4044–4044. 13 indexed citations
7.
Cooke, Mariana, et al.. (2019). ΔNp63α suppresses cells invasion by downregulating PKCγ/Rac1 signaling through miR-320a. Cell Death and Disease. 10(9). 680–680. 16 indexed citations
8.
Elkhadragy, Lobna & Weiwen Long. (2019). A Radioactive in vitro ERK3 Kinase Assay. BIO-PROTOCOL. 9(16). 3 indexed citations
9.
Elkhadragy, Lobna, et al.. (2018). Activation loop phosphorylation of ERK3 is important for its kinase activity and ability to promote lung cancer cell invasiveness. Journal of Biological Chemistry. 293(42). 16193–16205. 21 indexed citations
10.
Chen, Minyi, et al.. (2018). The atypical MAPK ERK3 potently suppresses melanoma cell growth and invasiveness. Journal of Cellular Physiology. 234(8). 13220–13232. 12 indexed citations
11.
Elkhadragy, Lobna, et al.. (2017). L290P/V mutations increase ERK3’s cytoplasmic localization and migration/invasion-promoting capability in cancer cells. Scientific Reports. 7(1). 14979–14979. 11 indexed citations
12.
Elkhadragy, Lobna, et al.. (2017). A regulatory BMI1/let‐7i/ERK3 pathway controls the motility of head and neck cancer cells. Molecular Oncology. 11(2). 194–207. 26 indexed citations
13.
Liu, Jian, Sung-Nam Cho, Bindu Akkanti, et al.. (2015). ErbB2 Pathway Activation upon Smad4 Loss Promotes Lung Tumor Growth and Metastasis. Cell Reports. 10(9). 1599–1613. 65 indexed citations
14.
Wang, Ying, Lei Li, Li Zhou, et al.. (2013). Site-specific Acetylation of the Proteasome Activator REGγ Directs Its Heptameric Structure and Functions. Journal of Biological Chemistry. 288(23). 16567–16578. 14 indexed citations
15.
Long, Weiwen, Charles E. Foulds, Jun Qin, et al.. (2012). ERK3 signals through SRC-3 coactivator to promote human lung cancer cell invasion. Journal of Clinical Investigation. 122(5). 1869–1880. 98 indexed citations
16.
Ao, Junping, Jiao Meng, Lei Zhu, et al.. (2012). Activation of androgen receptor induces ID1 and promotes hepatocellular carcinoma cell migration and invasion. Molecular Oncology. 6(5). 507–515. 37 indexed citations
17.
Long, Weiwen, Ping Yi, Larbi Amazit, et al.. (2010). SRC-3Δ4 Mediates the Interaction of EGFR with FAK to Promote Cell Migration. Molecular Cell. 37(3). 321–332. 110 indexed citations
18.
Naresh, Anjali, Weiwen Long, Gregory A. Vidal, et al.. (2006). The ERBB4/HER4 Intracellular Domain 4ICD Is a BH3-Only Protein Promoting Apoptosis of Breast Cancer Cells. Cancer Research. 66(12). 6412–6420. 172 indexed citations
19.
Williams, Christopher C., Tamika Duplessis, Weiwen Long, et al.. (2005). ERBB4/HER4 Potentiates STAT5A Transcriptional Activity by Regulating Novel STAT5A Serine Phosphorylation Events. Journal of Biological Chemistry. 280(25). 24175–24180. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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