Weiqiang Kang

428 total citations
16 papers, 358 citations indexed

About

Weiqiang Kang is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Epidemiology. According to data from OpenAlex, Weiqiang Kang has authored 16 papers receiving a total of 358 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Cardiology and Cardiovascular Medicine, 8 papers in Molecular Biology and 4 papers in Epidemiology. Recurrent topics in Weiqiang Kang's work include Cardiovascular Function and Risk Factors (4 papers), Endoplasmic Reticulum Stress and Disease (3 papers) and Renin-Angiotensin System Studies (3 papers). Weiqiang Kang is often cited by papers focused on Cardiovascular Function and Risk Factors (4 papers), Endoplasmic Reticulum Stress and Disease (3 papers) and Renin-Angiotensin System Studies (3 papers). Weiqiang Kang collaborates with scholars based in China, Thailand and Saudi Arabia. Weiqiang Kang's co-authors include Yun Zhang, Zhiming Ge, Guang‐Hui Liu, Yingying Sun, Jian Yao, Yongbin Wang, Changyong Zhou, Jing Geng, Zhuo Zhao and Tingting Wu and has published in prestigious journals such as Nature Communications, Biochemical and Biophysical Research Communications and American Heart Journal.

In The Last Decade

Weiqiang Kang

16 papers receiving 354 citations

Peers

Weiqiang Kang
Gao Xu China
Weiqiang Kang
Citations per year, relative to Weiqiang Kang Weiqiang Kang (= 1×) peers Gao Xu

Countries citing papers authored by Weiqiang Kang

Since Specialization
Citations

This map shows the geographic impact of Weiqiang Kang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Weiqiang Kang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Weiqiang Kang more than expected).

Fields of papers citing papers by Weiqiang Kang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Weiqiang Kang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Weiqiang Kang. The network helps show where Weiqiang Kang may publish in the future.

Co-authorship network of co-authors of Weiqiang Kang

This figure shows the co-authorship network connecting the top 25 collaborators of Weiqiang Kang. A scholar is included among the top collaborators of Weiqiang Kang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Weiqiang Kang. Weiqiang Kang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Zou, Zhijuan, Peng Fei Liu, Kaijun Liu, et al.. (2025). γ-Ray irradiated polyacrylamide networks enable high-performance Li||S pouch cells. Nature Communications. 16(1). 6729–6729. 1 indexed citations
2.
Ha, Mihyang, Changjin Hong, Weiqiang Kang, et al.. (2019). TEK is a novel prognostic marker for clear cell renal cell carcinoma.. European review for medical and pharmacological sciences. 23(4). 1451–1458. 20 indexed citations
3.
Liu, Xiangjuan, Qun Xu, Zhuo Zhao, et al.. (2015). Irbesartan ameliorates diabetic cardiomyopathy by regulating protein kinase D and ER stress activation in a type 2 diabetes rat model. Pharmacological Research. 93. 43–51. 38 indexed citations
4.
Wang, Yongbin, et al.. (2014). Rutin alleviates diabetic cardiomyopathy in a rat model of type 2 diabetes. Experimental and Therapeutic Medicine. 9(2). 451–455. 65 indexed citations
5.
Yang, Yi‐Qing, et al.. (2013). A novel PITX2c loss-of-function mutation underlies lone atrial fibrillation. International Journal of Molecular Medicine. 32(4). 827–834. 27 indexed citations
6.
Han, Yongsheng, et al.. (2013). Epigallocatechin Gallate Attenuated the Activation of Rat Cardiac Fibroblasts Induced by Angiotensin II via Regulating �-Arrestin1. Cellular Physiology and Biochemistry. 31(2-3). 338–346. 11 indexed citations
7.
Wu, Tingting, Zhe Dong, Jing Geng, et al.. (2011). Valsartan protects against ER stress-induced myocardial apoptosis via CHOP/Puma signaling pathway in streptozotocin-induced diabetic rats. European Journal of Pharmaceutical Sciences. 42(5). 496–502. 65 indexed citations
8.
10.
Zhao, Zhuo, Weiqiang Kang, Wei Wang, et al.. (2009). Hypertrophic response to angiotensin II is mediated by protein kinase D-extracellular signal-regulated kinase 5 pathway in human aortic smooth muscle cells. Biochemical and Biophysical Research Communications. 388(3). 517–522. 11 indexed citations
11.
Zhao, Zhuo, Jing Geng, Zhiming Ge, et al.. (2009). EFFICACY AND SAFETY OF ATORVASTATIN DURING EARLY HOSPITALIZATION IN ELDERLY PATIENTS WITH UNSTABLE ANGINA. Clinical and Experimental Pharmacology and Physiology. 36(5-6). 554–558. 8 indexed citations
12.
Zhao, Zhuo, et al.. (2008). Activation of ERK5 in angiotensin II-induced hypertrophy of human aortic smooth muscle cells. Molecular and Cellular Biochemistry. 322(1-2). 171–178. 7 indexed citations
13.
Sun, Yingying, Guang‐Hui Liu, Tengwei Song, et al.. (2008). Upregulation of GRP78 and caspase-12 in diastolic failing heart.. Acta Biochimica Polonica. 55(3). 511–516. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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