Wei‐Ping Min

6.3k total citations
121 papers, 5.1k citations indexed

About

Wei‐Ping Min is a scholar working on Immunology, Molecular Biology and Surgery. According to data from OpenAlex, Wei‐Ping Min has authored 121 papers receiving a total of 5.1k indexed citations (citations by other indexed papers that have themselves been cited), including 67 papers in Immunology, 54 papers in Molecular Biology and 18 papers in Surgery. Recurrent topics in Wei‐Ping Min's work include Immunotherapy and Immune Responses (45 papers), RNA Interference and Gene Delivery (34 papers) and T-cell and B-cell Immunology (25 papers). Wei‐Ping Min is often cited by papers focused on Immunotherapy and Immune Responses (45 papers), RNA Interference and Gene Delivery (34 papers) and T-cell and B-cell Immunology (25 papers). Wei‐Ping Min collaborates with scholars based in Canada, China and United States. Wei‐Ping Min's co-authors include Xiufen Zheng, Thomas E. Ichim, Anthony M. Jevnikar, Bertha García, Xusheng Zhang, Mu Li, Motohiko Suzuki, Robert Zhong, Costin Vladau and Xuyan Huang and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Blood.

In The Last Decade

Wei‐Ping Min

121 papers receiving 5.1k citations

Peers

Wei‐Ping Min
Comparison fields: 5 of 134
  • Immunology 2.1k
  • Molecular Biology 2.1k
  • Cancer Research 784
  • Surgery 663
  • Oncology 588
Replace Thomas E. Ichim with:
Thomas E. Ichim United States
Xiufen Zheng Canada
Maureen R. Horton United States
Makoto Naito Japan
Cécile V. Denis France
Ping Lu China
Melpo Christofidou‐Solomidou United States
Jing Jiao United States
Ludger Fink Germany
Boquan Jin China
Thomas E. Ichim United States View profile →
Citations per field, relative to Wei‐Ping Min
Wei‐Ping Min · 1×
Citations per year, relative to Wei‐Ping Min
Wei‐Ping Min · 1×

Countries citing papers authored by Wei‐Ping Min

Since Specialization
Citations

This map shows the geographic impact of Wei‐Ping Min's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei‐Ping Min with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei‐Ping Min more than expected).

Fields of papers citing papers by Wei‐Ping Min

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei‐Ping Min. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei‐Ping Min. The network helps show where Wei‐Ping Min may publish in the future.

Co-authorship network of co-authors of Wei‐Ping Min

This figure shows the co-authorship network connecting the top 25 collaborators of Wei‐Ping Min. A scholar is included among the top collaborators of Wei‐Ping Min based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei‐Ping Min. Wei‐Ping Min is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 12
2 20
3 91
4 1
5 49
6 3
7 42
8 83
9 128
10 11
11 1
12 2
13 67
14 77
15 10
16 79
17 14
18 211
19 56
20 195

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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