Wei‐Hsin Sun

1.3k total citations
36 papers, 1.1k citations indexed

About

Wei‐Hsin Sun is a scholar working on Molecular Biology, Physiology and Sensory Systems. According to data from OpenAlex, Wei‐Hsin Sun has authored 36 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 17 papers in Physiology and 13 papers in Sensory Systems. Recurrent topics in Wei‐Hsin Sun's work include Pain Mechanisms and Treatments (15 papers), Ion Channels and Receptors (13 papers) and Ion channel regulation and function (10 papers). Wei‐Hsin Sun is often cited by papers focused on Pain Mechanisms and Treatments (15 papers), Ion Channels and Receptors (13 papers) and Ion channel regulation and function (10 papers). Wei‐Hsin Sun collaborates with scholars based in Taiwan, United Kingdom and United States. Wei‐Hsin Sun's co-authors include Chih‐Cheng Chen, J. Hall, Michael Brownstein, Andreas Zimmer, Anne Zimmer, Chia-Wei Huang, Ying‐Ju Chen, Shir‐Ly Huang, Cheng‐Han Lee and Wei‐Li Wu and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and PLoS ONE.

In The Last Decade

Wei‐Hsin Sun

36 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wei‐Hsin Sun Taiwan 19 619 406 282 194 72 36 1.1k
Mircea Iftinca Canada 19 574 0.9× 362 0.9× 272 1.0× 435 2.2× 60 0.8× 30 1.2k
Zongxiang Tang China 14 467 0.8× 617 1.5× 377 1.3× 395 2.0× 59 0.8× 22 1.4k
Katharine L. Chu United States 20 349 0.6× 647 1.6× 449 1.6× 319 1.6× 94 1.3× 28 1.2k
JN Wood United Kingdom 7 640 1.0× 411 1.0× 271 1.0× 518 2.7× 44 0.6× 13 1.1k
Fenqin Xue China 14 686 1.1× 291 0.7× 610 2.2× 373 1.9× 81 1.1× 24 1.4k
Jiehong Huang China 16 419 0.7× 571 1.4× 551 2.0× 443 2.3× 101 1.4× 36 1.4k
Xiaochun Jin United States 13 347 0.6× 362 0.9× 151 0.5× 245 1.3× 33 0.5× 24 842
Una Park United States 5 489 0.8× 184 0.5× 480 1.7× 295 1.5× 32 0.4× 6 1.1k
Grant D. Nicol United States 17 618 1.0× 793 2.0× 445 1.6× 560 2.9× 71 1.0× 27 1.5k
Byung‐Chang Suh South Korea 20 1.0k 1.6× 212 0.5× 135 0.5× 490 2.5× 101 1.4× 61 1.5k

Countries citing papers authored by Wei‐Hsin Sun

Since Specialization
Citations

This map shows the geographic impact of Wei‐Hsin Sun's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei‐Hsin Sun with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei‐Hsin Sun more than expected).

Fields of papers citing papers by Wei‐Hsin Sun

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei‐Hsin Sun. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei‐Hsin Sun. The network helps show where Wei‐Hsin Sun may publish in the future.

Co-authorship network of co-authors of Wei‐Hsin Sun

This figure shows the co-authorship network connecting the top 25 collaborators of Wei‐Hsin Sun. A scholar is included among the top collaborators of Wei‐Hsin Sun based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei‐Hsin Sun. Wei‐Hsin Sun is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sun, Wei‐Hsin, et al.. (2024). T cell death-associated gene 8-mediated distinct signaling pathways modulate the early and late phases of neuropathic pain. iScience. 27(10). 110955–110955. 3 indexed citations
2.
Hung, Chih‐Hsien, et al.. (2023). Acidosis-related pain and its receptors as targets for chronic pain. Pharmacology & Therapeutics. 247. 108444–108444. 19 indexed citations
3.
Nguyễn, Ngọc Tuấn, et al.. (2022). Gut Mucosal Microbiome Is Perturbed in Rheumatoid Arthritis Mice and Partly Restored after TDAG8 Deficiency or Suppression by Salicylanilide Derivative. International Journal of Molecular Sciences. 23(7). 3527–3527. 20 indexed citations
4.
Kao, Ting‐Yu, et al.. (2020). Innovative Mind–Body Intervention Day Easy Exercise Increases Peripheral Blood CD34+ Cells in Adults. Cell Transplantation. 29. 2790874883–2790874883. 1 indexed citations
5.
Huang, Hsu‐Shan, et al.. (2020). TDAG8 deficiency reduces satellite glial number and pro-inflammatory macrophage number to relieve rheumatoid arthritis disease severity and chronic pain. Journal of Neuroinflammation. 17(1). 170–170. 25 indexed citations
6.
Chiang, Hao, et al.. (2020). Temporal expression patterns of distinct cytokines and M1/M2 macrophage polarization regulate rheumatoid arthritis progression. Molecular Biology Reports. 47(5). 3423–3437. 36 indexed citations
7.
Lee, Cheng‐Han, et al.. (2018). Involvement of advillin in somatosensory neuron subtype-specific axon regeneration and neuropathic pain. Proceedings of the National Academy of Sciences. 115(36). E8557–E8566. 29 indexed citations
8.
Sun, Wei‐Hsin, et al.. (2018). Differential expressions of plasma proteins in systemic lupus erythematosus patients identified by proteomic analysis. Journal of Microbiology Immunology and Infection. 52(5). 816–826. 4 indexed citations
9.
10.
Huang, Shir‐Ly, et al.. (2017). TDAG8, TRPV1, and ASIC3 involved in establishing hyperalgesic priming in experimental rheumatoid arthritis. Scientific Reports. 7(1). 8870–8870. 56 indexed citations
11.
Huang, Yu‐Fen, et al.. (2017). G2A as a Threshold Regulator of Inflammatory Hyperalgesia Modulates Chronic Hyperalgesia. Journal of Molecular Neuroscience. 64(1). 39–50. 6 indexed citations
12.
Sun, Wei‐Hsin, et al.. (2017). Peripheral 5-HT3 mediates mirror-image pain by a cross-talk with acid-sensing ion channel 3. Neuropharmacology. 130. 92–104. 12 indexed citations
13.
Huang, Yu‐Fen, et al.. (2017). TDAG8 involved in initiating inflammatory hyperalgesia and establishing hyperalgesic priming in mice. Scientific Reports. 7(1). 41415–41415. 25 indexed citations
14.
Steinhoff, Martin, et al.. (2016). Involvement of TRPV1 and TDAG8 in Pruriception Associated with Noxious Acidosis. Journal of Investigative Dermatology. 137(1). 170–178. 19 indexed citations
15.
Sun, Wei‐Hsin, et al.. (2015). Genetic exploration of the role of acid-sensing ion channels. Neuropharmacology. 94. 99–118. 58 indexed citations
16.
17.
Sun, Wei‐Hsin, et al.. (2011). Serotonin Receptor 5-HT2BMediates Serotonin-Induced Mechanical Hyperalgesia. Journal of Neuroscience. 31(4). 1410–1418. 55 indexed citations
18.
Wu, Wei‐Li, et al.. (2011). Targeting ASIC3 for pain, anxiety, and insulin resistance. Pharmacology & Therapeutics. 134(2). 127–138. 58 indexed citations
19.
Sun, Wei‐Hsin, et al.. (2006). RINGdb: An integrated database for G protein-coupled receptors and regulators of G protein signaling. BMC Genomics. 7(1). 317–317. 6 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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