Wei Ding

2.7k total citations
88 papers, 2.2k citations indexed

About

Wei Ding is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Physiology. According to data from OpenAlex, Wei Ding has authored 88 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 37 papers in Molecular Biology, 17 papers in Endocrinology, Diabetes and Metabolism and 16 papers in Physiology. Recurrent topics in Wei Ding's work include Hormonal Regulation and Hypertension (14 papers), Dialysis and Renal Disease Management (11 papers) and Inflammasome and immune disorders (9 papers). Wei Ding is often cited by papers focused on Hormonal Regulation and Hypertension (14 papers), Dialysis and Renal Disease Management (11 papers) and Inflammasome and immune disorders (9 papers). Wei Ding collaborates with scholars based in China, United States and Japan. Wei Ding's co-authors include Ke Jian Liu, Laurie G. Hudson, Yong Gu, Minmin Zhang, Xiao Bi, Karen L. Cooper, Honglei Guo, Bin Wang, Yuqing Liu and Feng Ding and has published in prestigious journals such as Journal of Biological Chemistry, Gastroenterology and PLoS ONE.

In The Last Decade

Wei Ding

86 papers receiving 2.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Wei Ding China 28 934 338 270 248 245 88 2.2k
Amrit Pal Singh India 24 408 0.4× 272 0.8× 65 0.2× 191 0.8× 184 0.8× 94 1.8k
Yanyan Zhao China 35 1.1k 1.2× 179 0.5× 402 1.5× 179 0.7× 97 0.4× 171 3.1k
Yuan‐Hung Wang Taiwan 26 905 1.0× 70 0.2× 177 0.7× 121 0.5× 424 1.7× 113 2.3k
Adrián M. Ramos Spain 30 1.3k 1.4× 870 2.6× 211 0.8× 277 1.1× 31 0.1× 62 3.0k
Franco Canestrari Italy 28 520 0.6× 500 1.5× 102 0.4× 106 0.4× 60 0.2× 63 2.3k
Xuemei Chen China 32 1.1k 1.2× 157 0.5× 411 1.5× 114 0.5× 32 0.1× 205 3.4k
Wei Qin China 30 1.3k 1.4× 261 0.8× 281 1.0× 203 0.8× 30 0.1× 131 3.2k
José L. Reyes Mexico 24 657 0.7× 226 0.7× 64 0.2× 145 0.6× 39 0.2× 78 2.0k
Changcheng Zhou United States 31 1.0k 1.1× 61 0.2× 295 1.1× 308 1.2× 39 0.2× 101 3.4k
Hongyan Zhao China 29 1.0k 1.1× 271 0.8× 247 0.9× 371 1.5× 12 0.0× 116 2.7k

Countries citing papers authored by Wei Ding

Since Specialization
Citations

This map shows the geographic impact of Wei Ding's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Wei Ding with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Wei Ding more than expected).

Fields of papers citing papers by Wei Ding

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Wei Ding. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Wei Ding. The network helps show where Wei Ding may publish in the future.

Co-authorship network of co-authors of Wei Ding

This figure shows the co-authorship network connecting the top 25 collaborators of Wei Ding. A scholar is included among the top collaborators of Wei Ding based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Wei Ding. Wei Ding is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Li, Xin, Qi Guo, Wei Ding, et al.. (2025). Impedance-derived phase angle as an indicator of physical function in maintenance hemodialysis patients. Nutrition. 136. 112795–112795.
2.
Xu, Heng, et al.. (2025). Cannabinoid receptor 2 facilitates the Schwann cells‐dependent peripheral nerve regeneration. Clinical and Translational Medicine. 15(1). e70184–e70184. 1 indexed citations
3.
Bi, Xiao, et al.. (2024). Gut microbiota dysbiosis and protein energy wasting in patients on hemodialysis: an observational longitudinal study. Frontiers in Nutrition. 10. 1270690–1270690. 2 indexed citations
4.
Liu, Yuqing, et al.. (2019). Mitochondrial dysfunction/NLRP3 inflammasome axis contributes to angiotensin II-induced skeletal muscle wasting via PPAR-γ. Laboratory Investigation. 100(5). 712–726. 37 indexed citations
5.
Lü, Wei, Jie Huang, Wei Ding, et al.. (2018). Predialysis fluid overload linked with quality of sleep in patients undergoing hemodialysis. Sleep Medicine. 51. 140–147. 9 indexed citations
6.
Bi, Xiao, et al.. (2018). 1,25-Dihydroxyvitamin D3 Attenuates Angiotensin II-Induced Renal Injury by Inhibiting Mitochondrial Dysfunction and Autophagy. Cellular Physiology and Biochemistry. 51(4). 1751–1762. 19 indexed citations
7.
Liu, Yuqing, et al.. (2018). Mito‐TEMPO Alleviates Renal Fibrosis by Reducing Inflammation, Mitochondrial Dysfunction, and Endoplasmic Reticulum Stress. Oxidative Medicine and Cellular Longevity. 2018(1). 5828120–5828120. 84 indexed citations
8.
Guo, Honglei, et al.. (2016). Endoplasmic Reticulum Chaperon Tauroursodeoxycholic Acid Attenuates Aldosterone-Infused Renal Injury. Mediators of Inflammation. 2016. 1–10. 7 indexed citations
9.
Ding, Wei, Bin Wang, Minmin Zhang, & Yong Gu. (2016). Involvement of Endoplasmic Reticulum Stress in Uremic Cardiomyopathy: Protective Effects of Tauroursodeoxycholic Acid. Cellular Physiology and Biochemistry. 38(1). 141–152. 21 indexed citations
10.
Wang, Bin, Wei Ding, Minmin Zhang, Hongmei Li, & Yong Gu. (2015). Rapamycin Attenuates Aldosterone-Induced Tubulointerstitial Inflammation and Fibrosis. Cellular Physiology and Biochemistry. 35(1). 116–125. 40 indexed citations
11.
Zhang, Kai, Wei Ding, Wei Sun, et al.. (2015). Beta1 integrin inhibits apoptosis induced by cyclic stretch in annulus fibrosus cells via ERK1/2 MAPK pathway. APOPTOSIS. 21(1). 13–24. 42 indexed citations
12.
Li, Wenpeng, Wei Ding, Zhongwen Zhou, et al.. (2013). Abnormal hypermethylation and clinicopathological significance of FBLN1 gene in cutaneous melanoma. Tumor Biology. 35(1). 123–127. 10 indexed citations
13.
Ding, Wei, Lei Yang, Minmin Zhang, & Yong Gu. (2012). Reactive oxygen species-mediated endoplasmic reticulum stress contributes to aldosterone-induced apoptosis in tubular epithelial cells. Biochemical and Biophysical Research Communications. 418(3). 451–456. 83 indexed citations
14.
Ma, Xi, Wei Ding, Junjun Wang, et al.. (2012). LOC66273 Isoform 2, a Novel Protein Highly Expressed in White Adipose Tissue, Induces Adipogenesis in 3T3-L1 Cells,. Journal of Nutrition. 142(3). 448–455. 23 indexed citations
15.
Ding, Wei, Lei Yang, Minmin Zhang, & Yong Gu. (2012). Chronic inhibition of nuclear factor kappa B attenuates aldosterone/salt-induced renal injury. Life Sciences. 90(15-16). 600–606. 31 indexed citations
16.
Bao, Yuanyuan, Xiliang Shang, Linuo Zhou, et al.. (2011). Relationship between N-terminal pro-B-type natriuretic peptide levels and metabolic syndrome. Archives of Medical Science. 2(2). 247–256. 28 indexed citations
17.
Ding, Wei, Wenlan Liu, Karen L. Cooper, et al.. (2008). Inhibition of Poly(ADP-ribose) Polymerase-1 by Arsenite Interferes with Repair of Oxidative DNA Damage. Journal of Biological Chemistry. 284(11). 6809–6817. 119 indexed citations
18.
Ding, Wei. (2006). Expression of Glucose Transporter 1 and Glucose Transporter 3 in Non-small Cell Lung Cancer and Its Clinical Significance. 1 indexed citations
19.
Ding, Wei, Laurie G. Hudson, & Ke Jian Liu. (2005). Inorganic arsenic compounds cause oxidative damage to DNA and protein by inducing ROS and RNS generation in human keratinocytes. Molecular and Cellular Biochemistry. 279(1-2). 105–112. 148 indexed citations
20.
Ding, Wei. (2004). Iron involved in the mechanisms of alcoholic liver disease. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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