Walter L. Binder
- Rheumatology top 5%
- Radiology, Nuclear Medicine and Imaging top 10%
- Hematology top 10%
- Immunology
- Gastroenterology top 5%
- Co-authors
- Gary L. NormanZakera ShumsErnst H. BeutnerHarry E. PrinceG. Michael IversonHenrique Luiz StaubCarlos Alberto von MühlenVincent A. DeBari
- Topics
- Systemic Lupus Erythematosus Research (9 papers)Monoclonal and Polyclonal Antibodies Research (9 papers)Celiac Disease Research and Management (6 papers)
- Partner nations
- United StatesItalySlovenia
In The Last Decade
Walter L. Binder
24 papers receiving 467 citations
Peers
Comparison fields: 5 of 52
- Rheumatology 262
- Radiology, Nuclear Medicine and Imaging 132
- Hematology 131
- Immunology 112
- Gastroenterology 101
Countries citing papers authored by Walter L. Binder
This map shows the geographic impact of Walter L. Binder's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Walter L. Binder with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Walter L. Binder more than expected).
Fields of papers citing papers by Walter L. Binder
This network shows the impact of papers produced by Walter L. Binder. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Walter L. Binder. The network helps show where Walter L. Binder may publish in the future.
Co-authorship network of co-authors of Walter L. Binder
This figure shows the co-authorship network connecting the top 25 collaborators of Walter L. Binder. A scholar is included among the top collaborators of Walter L. Binder based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Walter L. Binder. Walter L. Binder is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 31 | |
| 2 | 5 | |
| 3 | 35 | |
| 4 | 30 | |
| 5 | ANTIBODIES VERSUS DOMAIN I OF Β₂GPI AS A MARKER OF SYSTEMIC AUTOIMMUNITY | 1 |
| 6 | 1 | |
| 7 | 26 | |
| 8 | 65 | |
| 9 | 51 | |
| 10 | 33 | |
| 11 | 35 | |
| 12 | 16 | |
| 13 | 6 | |
| 14 | 3 | |
| 15 | 2 | |
| 16 | 30 | |
| 17 | 5 | |
| 18 | 2 | |
| 19 | 5 | |
| 20 | 21 |
About Walter L. Binder
Walter L. Binder is a scholar working on Gastroenterology, Rheumatology and Immunology, having authored 24 papers that have together received 483 indexed citations. Recurring topics across this work include Systemic Lupus Erythematosus Research (9 papers), Monoclonal and Polyclonal Antibodies Research (9 papers) and Celiac Disease Research and Management (6 papers). The work is most often cited by research in Gastroenterology (101 citations), Rheumatology (262 citations) and Hematology (131 citations). Walter L. Binder has collaborated with scholars based in United States, Italy and Slovenia. Frequent co-authors include Gary L. Norman, Zakera Shums, Ernst H. Beutner, Harry E. Prince, G. Michael Iverson, Henrique Luiz Staub, Carlos Alberto von Mühlen, Vincent A. DeBari, Tadeusz P. Chorzelski and Stefania Jabłońska. Their work appears in journals such as Gastroenterology, Clinical Chemistry and Annals of the Rheumatic Diseases.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.