W. Maruyama

2.3k total citations
64 papers, 1.9k citations indexed

About

W. Maruyama is a scholar working on Cellular and Molecular Neuroscience, Neurology and Molecular Biology. According to data from OpenAlex, W. Maruyama has authored 64 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Cellular and Molecular Neuroscience, 25 papers in Neurology and 20 papers in Molecular Biology. Recurrent topics in W. Maruyama's work include Parkinson's Disease Mechanisms and Treatments (21 papers), Neuroscience and Neuropharmacology Research (16 papers) and Neurotransmitter Receptor Influence on Behavior (9 papers). W. Maruyama is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (21 papers), Neuroscience and Neuropharmacology Research (16 papers) and Neurotransmitter Receptor Influence on Behavior (9 papers). W. Maruyama collaborates with scholars based in Japan, Italy and Germany. W. Maruyama's co-authors include Makoto Naoi, P. Dostert, Moussa B. H. Youdim, Hong Yi, Tsutomu Takahashi, Yukihiro Akao, Masayo Shamoto‐Nagai, M Naoi, Kazushige Mizoguchi and Takeshi Tabira and has published in prestigious journals such as Journal of Virology, Scientific Reports and Biochemical and Biophysical Research Communications.

In The Last Decade

W. Maruyama

60 papers receiving 1.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
W. Maruyama Japan 29 696 687 644 289 254 64 1.9k
N Ogawa Japan 24 448 0.6× 683 1.0× 564 0.9× 226 0.8× 204 0.8× 89 1.7k
Toshiharu Nagatsu Japan 25 425 0.6× 663 1.0× 943 1.5× 332 1.1× 137 0.5× 83 2.4k
Creed W. Abell United States 24 526 0.8× 722 1.1× 919 1.4× 160 0.6× 191 0.8× 79 2.2k
Vı́ctor Tapias United States 23 862 1.2× 650 0.9× 693 1.1× 458 1.6× 87 0.3× 34 2.2k
Nathalie Lambeng France 15 697 1.0× 1.2k 1.8× 845 1.3× 289 1.0× 210 0.8× 24 2.7k
Zheng-Hong Qin China 20 299 0.4× 646 0.9× 1.1k 1.7× 395 1.4× 207 0.8× 29 2.5k
Irmgard Paris Chile 23 972 1.4× 665 1.0× 713 1.1× 332 1.1× 172 0.7× 35 2.2k
G. Cohen United States 19 556 0.8× 715 1.0× 764 1.2× 316 1.1× 138 0.5× 37 2.0k
Andrea M. Cesura Switzerland 31 472 0.7× 1.2k 1.7× 1.4k 2.2× 446 1.5× 415 1.6× 81 3.3k
Javier Cuesta Spain 25 389 0.6× 371 0.5× 966 1.5× 454 1.6× 107 0.4× 38 2.1k

Countries citing papers authored by W. Maruyama

Since Specialization
Citations

This map shows the geographic impact of W. Maruyama's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. Maruyama with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. Maruyama more than expected).

Fields of papers citing papers by W. Maruyama

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. Maruyama. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. Maruyama. The network helps show where W. Maruyama may publish in the future.

Co-authorship network of co-authors of W. Maruyama

This figure shows the co-authorship network connecting the top 25 collaborators of W. Maruyama. A scholar is included among the top collaborators of W. Maruyama based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with W. Maruyama. W. Maruyama is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ohno, Hitoshi, et al.. (2024). Diverse B-cell tumors associated with t(14;19)(q32;q13)/<i>IGH</i>::<i>BCL3</i> identified by G-banding and fluorescence <i>in situ</i> hybridization. Journal of Clinical and Experimental Hematopathology. 64(1). 21–31. 1 indexed citations
2.
Yamamoto, Ryo, Atsushi Takeda, Satoshi Nakamura, et al.. (2023). Intravascular Large B-cell Lymphoma Presenting as Pulmonary Ground-glass Nodules That Progressed Slowly over Several Months with No Overt Symptoms. Internal Medicine. 63(4). 559–563. 1 indexed citations
3.
Yamazaki, Hiroyuki, Kotaro Shirakawa, Tadahiko Matsumoto, et al.. (2019). Endogenous APOBEC3B Overexpression Constitutively Generates DNA Substitutions and Deletions in Myeloma Cells. Scientific Reports. 9(1). 7122–7122. 25 indexed citations
4.
Fukunaga, Akiko, et al.. (2011). Successful treatment with combined chemotherapy of two adult cases of hemophagocytic lymphohistiocytosis in recipients of umbilical cord blood cell transplantation. International Journal of Hematology. 93(4). 551–554. 12 indexed citations
6.
Naoi, M, W. Maruyama, Yukihiro Akao, Hong Yi, & Y. Yamaoka. (2006). Involvement of type A monoamine oxidase in neurodegeneration: regulation of mitochondrial signaling leading to cell death or neuroprotection. PubMed. 67–77. 72 indexed citations
7.
Maruyama, W., et al.. (2006). The effect of neuromelanin on the proteasome activity in human dopaminergic SH-SY5Y cells. PubMed. 125–132. 8 indexed citations
8.
Matsumoto, Kenji, Yukihiro Akao, Hong Yi, et al.. (2005). Overexpression of amyloid precursor protein induces susceptibility to oxidative stress in human neuroblastoma SH-SY5Y cells. Journal of Neural Transmission. 113(2). 125–135. 27 indexed citations
9.
Shamoto‐Nagai, Masayo, W. Maruyama, Hong Yi, et al.. (2005). Neuromelanin induces oxidative stress in mitochondria through release of iron: mechanism behind the inhibition of 26S proteasome. Journal of Neural Transmission. 113(5). 633–644. 52 indexed citations
10.
Naoi, Makoto, W. Maruyama, Yukihiro Akao, & Hong Yi. (2002). Mitochondria determine the survival and death in apoptosis by an endogenous neurotoxin, N-methyl(R)salsolinol, and neuroprotection by propargylamines. Journal of Neural Transmission. 109(5-6). 607–621. 29 indexed citations
11.
Uezono, Takashi, W. Maruyama, Keiko Matsubara, et al.. (2001). Norharman, an indoleamine-derived β-carboline, but not Trp-P-2, a γ-carboline, induces apoptotic cell death in human neuroblastoma SH-SY5Y cells. Journal of Neural Transmission. 108(8). 943–953. 30 indexed citations
13.
Naoi, Makoto, W. Maruyama, Shinji Ohta, et al.. (1998). Effects of various tetrahydroisoquinoline derivatives on mitochondrial respiration and the electron transfer complexes. Journal of Neural Transmission. 105(6-7). 677–688. 39 indexed citations
14.
Takahashi, Tsutomu, W. Maruyama, Yulin Deng, et al.. (1997). Cytotoxicity of endogenous isoquinolines to human dopaminergic neuroblastoma SH-SY5Y cells. Journal of Neural Transmission. 104(1). 59–66. 36 indexed citations
15.
Naoi, Makoto, W. Maruyama, P. Dostert, & Yoshio Hashizume. (1997). N-Methyl-(R)salsolinol as a dopaminergic neurotoxin: From an animal model to an early marker of Parkinson’s disease. Journal of neural transmission. Supplementum. 50. 89–105. 43 indexed citations
16.
Naoi, Makoto, W. Maruyama, Yi‐Mo Deng, et al.. (1994). Inhibition of type a monoamine oxidase by 2(N)-methyl-6,7-dihydroxyisoquinolinium ions. Neurochemistry International. 25(5). 475–481. 13 indexed citations
17.
Maruyama, W., Daiichiro Nakahara, P. Dostert, Akihisa Takahashi, & Makoto Naoi. (1993). Naturally-occurring isoquinolines perturb monoamine metabolism in the brain: studied by in vivo microdialysis. Journal of Neural Transmission. 94(2). 91–102. 21 indexed citations
19.
Maruyama, W., et al.. (1992). The mechanism of perturbation in monoamine metabolism by L-DOPA therapy: in vivo and in vitro studies. Journal of Neural Transmission. 90(3). 183–197. 26 indexed citations
20.
Kasai, S, W. Maruyama, Takashi Terasawa, & Masatomi Iijima. (1977). [Study of erythroid colony formation of rabbits in vitro: the effect of spleen extract from irradiated rats on CFU-E (author's transl)].. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 39(12). 490–9. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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