W E Ollier

1.2k total citations
20 papers, 897 citations indexed

About

W E Ollier is a scholar working on Rheumatology, Immunology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, W E Ollier has authored 20 papers receiving a total of 897 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Rheumatology, 9 papers in Immunology and 6 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in W E Ollier's work include Systemic Lupus Erythematosus Research (8 papers), Monoclonal and Polyclonal Antibodies Research (6 papers) and Rheumatoid Arthritis Research and Therapies (6 papers). W E Ollier is often cited by papers focused on Systemic Lupus Erythematosus Research (8 papers), Monoclonal and Polyclonal Antibodies Research (6 papers) and Rheumatoid Arthritis Research and Therapies (6 papers). W E Ollier collaborates with scholars based in United Kingdom, United States and India. W E Ollier's co-authors include Wendy Thomson, A J Silman, Alex J. MacGregor, Damini Jawaheer, D Carthy, Hector Chinoy, Lucy R. Wedderburn, Robert G. Cooper, J. North and Harsha Gunawardena and has published in prestigious journals such as Annals of the Rheumatic Diseases, International Journal of Epidemiology and Lara D. Veeken.

In The Last Decade

W E Ollier

20 papers receiving 856 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
W E Ollier United Kingdom 15 562 380 296 167 112 20 897
Gale A. McCarty United States 13 410 0.7× 248 0.7× 120 0.4× 137 0.8× 145 1.3× 21 725
Rudolf Mierau Germany 15 250 0.4× 223 0.6× 256 0.9× 153 0.9× 236 2.1× 33 667
M. Hirakata Japan 16 318 0.6× 235 0.6× 337 1.1× 82 0.5× 178 1.6× 42 888
S Ozaki Japan 16 269 0.5× 352 0.9× 224 0.8× 120 0.7× 55 0.5× 30 813
Andrea L. Sestak United States 12 631 1.1× 557 1.5× 86 0.3× 166 1.0× 143 1.3× 17 946
Paulo Luiz Carvalho Francescantônio Brazil 9 418 0.7× 228 0.6× 181 0.6× 288 1.7× 238 2.1× 15 715
Orlando Gabriel Carballo Argentina 9 433 0.8× 216 0.6× 172 0.6× 294 1.8× 286 2.6× 19 753
J. Awad United Kingdom 18 392 0.7× 557 1.5× 69 0.2× 229 1.4× 133 1.2× 42 1.1k
Martha Mattioli United States 7 382 0.7× 275 0.7× 223 0.8× 293 1.8× 288 2.6× 7 856
Elizabeth Taylor‐Albert United States 6 393 0.7× 339 0.9× 99 0.3× 172 1.0× 118 1.1× 9 617

Countries citing papers authored by W E Ollier

Since Specialization
Citations

This map shows the geographic impact of W E Ollier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W E Ollier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W E Ollier more than expected).

Fields of papers citing papers by W E Ollier

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W E Ollier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W E Ollier. The network helps show where W E Ollier may publish in the future.

Co-authorship network of co-authors of W E Ollier

This figure shows the co-authorship network connecting the top 25 collaborators of W E Ollier. A scholar is included among the top collaborators of W E Ollier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with W E Ollier. W E Ollier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chinoy, Hector, Debbie Payne, Kay Poulton, et al.. (2009). HLA-DPB1 associations differ between DRB1*03 positive anti-Jo-1 and anti-PM-Scl antibody positive idiopathic inflammatory myopathy. Lara D. Veeken. 48(10). 1213–1217. 28 indexed citations
2.
Gunawardena, Harsha, Lucy R. Wedderburn, Hector Chinoy, et al.. (2009). Autoantibodies to a 140‐kd protein in juvenile dermatomyositis are associated with calcinosis. Arthritis & Rheumatism. 60(6). 1807–1814. 156 indexed citations
3.
Amoli, Mahsa M., D Carthy, Hazel Platt, & W E Ollier. (2008). EBV Immortalization of human B lymphocytes separated from small volumes of cryo-preserved whole blood. International Journal of Epidemiology. 37(Supplement 1). i41–i45. 38 indexed citations
4.
Wedderburn, Lucy R., N. McHugh, Hector Chinoy, et al.. (2007). HLA class II haplotype and autoantibody associations in children with juvenile dermatomyositis and juvenile dermatomyositis–scleroderma overlap. Lara D. Veeken. 46(12). 1786–1791. 80 indexed citations
5.
Greig, Carolyn, Richard Aspinwall, Mark Grant, et al.. (2006). Linkage to nodal osteoarthritis: quantitative and qualitative analyses of data from a whole-genome screen identify trait-dependent susceptibility loci. Annals of the Rheumatic Diseases. 65(9). 1131–1138. 14 indexed citations
6.
Weatherby, S. J. M., Wendy Thomson, L Pepper, et al.. (2001). HLA-DRB1 and disease outcome in multiple sclerosis. Journal of Neurology. 248(4). 304–310. 53 indexed citations
7.
Mattey, Derek L., Andrew Hassell, Michael Plant, et al.. (1999). The influence of HLA-DRB1 alleles encoding the DERAA amino acid motif on radiological outcome in rheumatoid arthritis. Lara D. Veeken. 38(12). 1221–1227. 26 indexed citations
8.
Stanworth, Simon, et al.. (1998). Absence of an association between mannose-binding lectin polymorphism and rheumatoid arthritis. Lara D. Veeken. 37(2). 186–188. 27 indexed citations
9.
Ollier, W E, et al.. (1997). Human cytomegalovirus infection and systemic lupus erythematosus.. PubMed. 15(4). 405–9. 65 indexed citations
10.
11.
Silman, Alan J., Elaine M. Hay, Jane Worthington, et al.. (1995). Lack of influence of non-inherited maternal HLA-DR alleles on susceptibility to rheumatoid arthritis.. Annals of the Rheumatic Diseases. 54(4). 311–313. 10 indexed citations
12.
MacGregor, Alex J., et al.. (1995). Rheumatoid factor isotypes in monozygotic and dizygotic twins discordant for rheumatoid arthritis.. PubMed. 22(12). 2203–7. 11 indexed citations
13.
MacGregor, Alex J., W E Ollier, Wendy Thomson, Damini Jawaheer, & A J Silman. (1995). HLA-DRB1*0401/0404 genotype and rheumatoid arthritis: increased association in men, young age at onset, and disease severity.. PubMed. 22(6). 1032–6. 160 indexed citations
14.
Hajeer, Ali H., Alex J. MacGregor, Alan S. Rigby, et al.. (1994). Influence of previous exposure to human parvovirus B19 infection in explaining susceptibility to rheumatoid arthritis: an analysis of disease discordant twin pairs.. Annals of the Rheumatic Diseases. 53(2). 137–139. 19 indexed citations
15.
Davies, E. J., Catherine J. Hutchings, M Chantal Hillarby, et al.. (1994). HLA-DP does not contribute towards susceptibility to systemic lupus erythematosus.. Annals of the Rheumatic Diseases. 53(3). 188–190. 5 indexed citations
16.
Thomson, Wendy, L Pepper, Antony Payton, et al.. (1993). Absence of an association between HLA-DRB1*04 and rheumatoid arthritis in newly diagnosed cases from the community.. Annals of the Rheumatic Diseases. 52(7). 539–541. 43 indexed citations
17.
Ollier, W E, C. A. L. Stephens, J. Awad, et al.. (1991). Is rheumatoid arthritis in Indians associated with HLA antigens sharing a DR beta 1 epitope?. Annals of the Rheumatic Diseases. 50(5). 295–297. 33 indexed citations
18.
19.
Jaraquemada, Dolores, W E Ollier, J. Awad, et al.. (1986). HLA and rheumatoid arthritis: a combined analysis of 440 British patients.. Annals of the Rheumatic Diseases. 45(8). 627–636. 98 indexed citations
20.
Alonso, A., E. J. Williams, Davinder Gill, et al.. (1985). HLA‐B15 heterogeneity in different populations. Tissue Antigens. 25(1). 33–37. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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