Volker Spindler

3.6k total citations
70 papers, 2.7k citations indexed

About

Volker Spindler is a scholar working on Pathology and Forensic Medicine, Genetics and Cell Biology. According to data from OpenAlex, Volker Spindler has authored 70 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 37 papers in Pathology and Forensic Medicine, 25 papers in Genetics and 22 papers in Cell Biology. Recurrent topics in Volker Spindler's work include Autoimmune Bullous Skin Diseases (35 papers), Coagulation, Bradykinin, Polyphosphates, and Angioedema (25 papers) and Skin and Cellular Biology Research (14 papers). Volker Spindler is often cited by papers focused on Autoimmune Bullous Skin Diseases (35 papers), Coagulation, Bradykinin, Polyphosphates, and Angioedema (25 papers) and Skin and Cellular Biology Research (14 papers). Volker Spindler collaborates with scholars based in Germany, Switzerland and United States. Volker Spindler's co-authors include Jens Waschke, Nicolas Schlegel, Franziska Vielmuth, Eva Hartlieb, Vera Rötzer, Enno Schmidt, Detlev Drenckhahn, Mariya Y. Radeva, Elias Walter and Detlef Zillikens and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Journal of Clinical Investigation.

In The Last Decade

Volker Spindler

69 papers receiving 2.6k citations

Peers

Volker Spindler

PFM

GENET

RHEUM

Martyn Chidgey United Kingdom

Noboru Asada Japan

Masato Morikawa Japan

Jean Luc Laı̈ France

Lionel Fontao Switzerland

Hiroshi Kuroda Japan

Anna Savoia Italy

Anna Brunn Germany

Marion C. Dickson United Kingdom

Katsutoshi Ozaki Japan

Martyn Chidgey United Kingdom

Volker Spindler

431 ×0.4

PFM

1.1k ×1.2

272 ×0.3

GENET

253 ×0.5

RHEUM

702 ×1.4

Citations per year, relative to Volker Spindler Volker Spindler (= 1×) peers Martyn Chidgey

Countries citing papers authored by Volker Spindler

Since Specialization

Citations

This map shows the geographic impact of Volker Spindler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Volker Spindler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Volker Spindler more than expected).

Fields of papers citing papers by Volker Spindler

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Volker Spindler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Volker Spindler. The network helps show where Volker Spindler may publish in the future.

Co-authorship network of co-authors of Volker Spindler

This figure shows the co-authorship network connecting the top 25 collaborators of Volker Spindler. A scholar is included among the top collaborators of Volker Spindler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Volker Spindler. Volker Spindler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Egu, Desalegn Tadesse, Thomas Schmitt, Nancy Ernst, et al.. (2024). EGFR Inhibition by Erlotinib Rescues Desmosome Ultrastructure and Keratin Anchorage and Protects against Pemphigus Vulgaris IgG–Induced Acantholysis in Human Epidermis. Journal of Investigative Dermatology. 144(11). 2440–2452. 9 indexed citations

Franz, Henriette, et al.. (2024). DPM1 modulates desmosomal adhesion and epidermal differentiation through SERPINB5. The Journal of Cell Biology. 223(4). 4 indexed citations

Franz, Henriette, Pauline Hanns, Dario Didona, et al.. (2024). Unbiased screening identifies regulators of cell-cell adhesion and treatment options in pemphigus. Nature Communications. 15(1). 8044–8044. 2 indexed citations

Spindler, Volker, Brenda Gerull, Kathleen J. Green, et al.. (2023). Meeting report – Desmosome dysfunction and disease: Alpine desmosome disease meeting. Journal of Cell Science. 136(1). 15 indexed citations

Radeva, Mariya Y., et al.. (2023). Cytoskeletal anchorage of different Dsg3 pools revealed by combination of hybrid STED/SMFS-AFM. Cellular and Molecular Life Sciences. 80(1). 25–25. 9 indexed citations

Schinner, Camilla, Lifen Xu, Henriette Franz, et al.. (2022). Defective Desmosomal Adhesion Causes Arrhythmogenic Cardiomyopathy by Involving an Integrin-αVβ6/TGF-β Signaling Cascade. Circulation. 146(21). 1610–1626. 28 indexed citations

Hudemann, Christoph, Robert Pollmann, Anna Zakrzewicz, et al.. (2022). IgG against the Membrane-Proximal Portion of the Desmoglein 3 Ectodomain Induces Loss of Keratinocyte Adhesion, a Hallmark in Pemphigus Vulgaris. Journal of Investigative Dermatology. 143(2). 254–263.e3. 8 indexed citations

Schinner, Camilla, et al.. (2020). Clustering of desmosomal cadherins by desmoplakin is essential for cell‐cell adhesion. Acta Physiologica. 231(4). e13609–e13609. 15 indexed citations

Egu, Desalegn Tadesse, Anna M. Sigmund, Enno Schmidt, et al.. (2019). A new ex vivo human oral mucosa model reveals that p38 MAPK inhibition is not effective in preventing autoantibody‐induced mucosal blistering in pemphigus. British Journal of Dermatology. 182(4). 987–994. 26 indexed citations

10.

Kugelmann, Daniela, Vera Rötzer, Elias Walter, et al.. (2019). Role of Src and Cortactin in Pemphigus Skin Blistering. Frontiers in Immunology. 10. 626–626. 25 indexed citations

11.

Radeva, Mariya Y., et al.. (2018). Keratin Retraction and Desmoglein3 Internalization Independently Contribute to Autoantibody-Induced Cell Dissociation in Pemphigus Vulgaris. Frontiers in Immunology. 9. 858–858. 12 indexed citations

12.

Spindler, Volker & Jens Waschke. (2018). Pemphigus—A Disease of Desmosome Dysfunction Caused by Multiple Mechanisms. Frontiers in Immunology. 9. 136–136. 71 indexed citations

13.

Walter, Elias, Franziska Vielmuth, Lukas T. Rotkopf, et al.. (2017). Different signaling patterns contribute to loss of keratinocyte cohesion dependent on autoantibody profile in pemphigus. Scientific Reports. 7(1). 3579–3579. 54 indexed citations

14.

Rötzer, Vera, Eva Hartlieb, Franziska Vielmuth, et al.. (2015). E-cadherin and Src associate with extradesmosomal Dsg3 and modulate desmosome assembly and adhesion. Cellular and Molecular Life Sciences. 72(24). 4885–4897. 55 indexed citations

15.

Monteiro, Ana Carolina, Ronen Sumagin, Franziska Vielmuth, et al.. (2014). Trans-dimerization of JAM-A regulates Rap2 and is mediated by a domain that is distinct from thecis-dimerization interface. Molecular Biology of the Cell. 25(10). 1574–1585. 28 indexed citations

16.

Spindler, Volker, Carina Dehner, Stefan Hübner, & Jens Waschke. (2014). Plakoglobin but Not Desmoplakin Regulates Keratinocyte Cohesion via Modulation of p38MAPK Signaling. Journal of Investigative Dermatology. 134(6). 1655–1664. 36 indexed citations

17.

Spindler, Volker & Jens Waschke. (2011). Role of Rho GTPases in desmosomal adhesion and pemphigus pathogenesis. Annals of Anatomy - Anatomischer Anzeiger. 193(3). 177–180. 21 indexed citations

18.

Gliem, Martin, Wolfgang-Moritz Heupel, Volker Spindler, Gregory S. Harms, & Jens Waschke. (2010). Actin reorganization contributes to loss of cell adhesion in pemphigus vulgaris. American Journal of Physiology-Cell Physiology. 299(3). C606–C613. 44 indexed citations

19.

Spindler, Volker, Wolfgang-Moritz Heupel, Athina Efthymiadis, et al.. (2009). Desmocollin 3-mediated Binding Is Crucial for Keratinocyte Cohesion and Is Impaired in Pemphigus. Journal of Biological Chemistry. 284(44). 30556–30564. 93 indexed citations

20.

Waschke, Jens, et al.. (2006). Inhibition of Rho A activity causes pemphigus skin blistering. The Journal of Cell Biology. 175(5). 721–727. 135 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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