Viktor Pastukh

810 total citations
25 papers, 637 citations indexed

About

Viktor Pastukh is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Cell Biology. According to data from OpenAlex, Viktor Pastukh has authored 25 papers receiving a total of 637 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 9 papers in Pathology and Forensic Medicine and 6 papers in Cell Biology. Recurrent topics in Viktor Pastukh's work include Mitochondrial Function and Pathology (11 papers), Cardiac Ischemia and Reperfusion (9 papers) and Aldose Reductase and Taurine (5 papers). Viktor Pastukh is often cited by papers focused on Mitochondrial Function and Pathology (11 papers), Cardiac Ischemia and Reperfusion (9 papers) and Aldose Reductase and Taurine (5 papers). Viktor Pastukh collaborates with scholars based in United States and Japan. Viktor Pastukh's co-authors include Stephen W. Schaffer, Mark N. Gillespie, Junichi Azuma, Glenn L. Wilson, Viktoriya Solodushko, David V. Schaffer, Julio F. Turrens, Joshua N. Leonard, Mykhaylo V. Ruchko and Mikhail Alexeyev and has published in prestigious journals such as The FASEB Journal, Free Radical Biology and Medicine and Annals of the New York Academy of Sciences.

In The Last Decade

Viktor Pastukh

24 papers receiving 625 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Viktor Pastukh United States 13 387 127 117 75 72 25 637
Andrea Hodgkinson United Kingdom 15 264 0.7× 91 0.7× 118 1.0× 48 0.6× 144 2.0× 28 702
Christian Fork Germany 16 347 0.9× 136 1.1× 70 0.6× 38 0.5× 94 1.3× 21 782
Tianluo Lei China 12 307 0.8× 146 1.1× 128 1.1× 38 0.5× 53 0.7× 16 622
Diana Ensenat United States 10 460 1.2× 96 0.8× 118 1.0× 73 1.0× 19 0.3× 14 662
Sylvia Y. Low United Kingdom 19 292 0.8× 247 1.9× 180 1.5× 62 0.8× 113 1.6× 27 730
Mako Yasuda‒Yamahara Japan 15 246 0.6× 175 1.4× 52 0.4× 38 0.5× 56 0.8× 24 694
Carolyn L. Buller United States 9 380 1.0× 157 1.2× 48 0.4× 39 0.5× 36 0.5× 12 744
András Frankó Germany 16 452 1.2× 292 2.3× 81 0.7× 59 0.8× 55 0.8× 27 755
Yasuhiro Horibata Japan 17 546 1.4× 124 1.0× 121 1.0× 19 0.3× 60 0.8× 46 983
Silvia Pedretti Italy 13 323 0.8× 161 1.3× 94 0.8× 31 0.4× 33 0.5× 28 719

Countries citing papers authored by Viktor Pastukh

Since Specialization
Citations

This map shows the geographic impact of Viktor Pastukh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Viktor Pastukh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Viktor Pastukh more than expected).

Fields of papers citing papers by Viktor Pastukh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Viktor Pastukh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Viktor Pastukh. The network helps show where Viktor Pastukh may publish in the future.

Co-authorship network of co-authors of Viktor Pastukh

This figure shows the co-authorship network connecting the top 25 collaborators of Viktor Pastukh. A scholar is included among the top collaborators of Viktor Pastukh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Viktor Pastukh. Viktor Pastukh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Dubey, Shubham, Zhihong Yu, Ahmed Lazrak, et al.. (2025). Oxidative damage to lung mitochondrial DNA is a key contributor to the development of chemical lung injury. Redox Biology. 82. 103624–103624.
3.
Troyanovsky, Boris, et al.. (2016). The Functionality of Minimal PiggyBac Transposons in Mammalian Cells. Molecular Therapy — Nucleic Acids. 5(10). e369–e369. 18 indexed citations
4.
Ruchko, Mykhaylo V., Sergiy Sukhanov, Olena M. Gorodnya, et al.. (2015). Involvement of Mitochondrial (mt) DNA Oxidative Damage and Circulating Fragments in Early Stages of Atherogenesis. The FASEB Journal. 29(S1). 2 indexed citations
5.
Pastukh, Viktor, Justin T. Roberts, David W. Clark, et al.. (2015). An oxidative DNA “damage” and repair mechanism localized in the VEGF promoter is important for hypoxia-induced VEGF mRNA expression. American Journal of Physiology-Lung Cellular and Molecular Physiology. 309(11). L1367–L1375. 112 indexed citations
6.
Jong, Chian Ju, et al.. (2014). Role of protein phosphorylation in excitation-contraction coupling in taurine deficient hearts. American Journal of Physiology-Heart and Circulatory Physiology. 308(3). H232–H239. 45 indexed citations
7.
Gillespie, Mark N., Viktor Pastukh, & Mykhaylo V. Ruchko. (2009). Oxidative DNA Modifications in Hypoxic Signaling. Annals of the New York Academy of Sciences. 1177(1). 140–150. 22 indexed citations
8.
Alexeyev, Mikhail, et al.. (2008). Selective elimination of mutant mitochondrial genomes as therapeutic strategy for the treatment of NARP and MILS syndromes. Gene Therapy. 15(7). 516–523. 83 indexed citations
9.
Ito, Takashi, Viktor Pastukh, Viktoriya Solodushko, Junichi Azuma, & Stephen W. Schaffer. (2008). Effect of Taurine on Protein Kinase C Isoforms: Role in Taurine’s Actions?. Advances in experimental medicine and biology. 643. 3–11. 4 indexed citations
12.
Pastukh, Viktor, Joshua N. Leonard, Julio F. Turrens, et al.. (2007). Mitochondrial DNA damage triggers mitochondrial-superoxide generation and apoptosis. American Journal of Physiology-Cell Physiology. 294(2). C413–C422. 122 indexed citations
13.
Pastukh, Viktor, Mykhaylo V. Ruchko, Olena M. Gorodnya, Glenn L. Wilson, & Mark N. Gillespie. (2007). Sequence-specific oxidative base modifications in hypoxia-inducible genes. Free Radical Biology and Medicine. 43(12). 1616–1626. 45 indexed citations
15.
Pastukh, Viktor, Songwei Wu, & Stephen Schaffer. (2006). A114. Regulation of T-type calcium channel in rat cardiomyocytes by oxidative stress. Journal of Molecular and Cellular Cardiology. 40(6). 891–892. 1 indexed citations
16.
Pastukh, Viktor, et al.. (2005). Contribution of the PI 3-kinase/Akt survival pathway toward osmotic preconditioning. Molecular and Cellular Biochemistry. 269(1). 59–67. 21 indexed citations
17.
Pastukh, Viktor, et al.. (2004). Reversal of hyperglycemic preconditioning by angiotensin II: role of calcium transport. American Journal of Physiology-Heart and Circulatory Physiology. 288(4). H1965–H1975. 14 indexed citations
18.
Schaffer, Stephen W., et al.. (2003). Possible Cause of Taurine-deficient Cardiomyopathy: Potentiation of Angiotensin II Action. Journal of Cardiovascular Pharmacology. 41(5). 751–759. 26 indexed citations
19.
Grishko, Valentina, Viktor Pastukh, Viktoriya Solodushko, et al.. (2003). Apoptotic cascade initiated by angiotensin II in neonatal cardiomyocytes: role of DNA damage. American Journal of Physiology-Heart and Circulatory Physiology. 285(6). H2364–H2372. 65 indexed citations
20.
Schaffer, Stephen W., et al.. (2002). Effect of ischemia, calcium depletion and repletion, acidosis and hypoxia on cellular taurine content.. Amino Acids. 23(4). 395–400. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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