Vijay Walia

2.3k total citations · 1 hit paper
18 papers, 1.7k citations indexed

About

Vijay Walia is a scholar working on Molecular Biology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, Vijay Walia has authored 18 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 10 papers in Oncology and 3 papers in Pathology and Forensic Medicine. Recurrent topics in Vijay Walia's work include Melanoma and MAPK Pathways (4 papers), Cancer Cells and Metastasis (4 papers) and Cancer-related Molecular Pathways (4 papers). Vijay Walia is often cited by papers focused on Melanoma and MAPK Pathways (4 papers), Cancer Cells and Metastasis (4 papers) and Cancer-related Molecular Pathways (4 papers). Vijay Walia collaborates with scholars based in United States, United Kingdom and Israel. Vijay Walia's co-authors include Randolph C. Elble, Shoumin Zhu, Mohit Sachdeva, Kounosuke Watabe, Fangting Wu, Hailong Wu, Yin‐Yuan Mo, Sumit Kumar, Yardena Samuels and Todd D. Prickett and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Genetics.

In The Last Decade

Vijay Walia

18 papers receiving 1.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

p53 represses c-Myc through induction of the tumor suppressor miR-145

2009 691 citations Mohit Sachdeva, Shoumin Zhu et al. Proceedings of the National Academy of Sciences profile →

Peers

Countries citing papers authored by Vijay Walia

Since Specialization

Citations

This map shows the geographic impact of Vijay Walia's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Vijay Walia with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Vijay Walia more than expected).

Fields of papers citing papers by Vijay Walia

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Vijay Walia. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Vijay Walia. The network helps show where Vijay Walia may publish in the future.

Co-authorship network of co-authors of Vijay Walia

This figure shows the co-authorship network connecting the top 25 collaborators of Vijay Walia. A scholar is included among the top collaborators of Vijay Walia based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Vijay Walia. Vijay Walia is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

#	Work	Indexed citations
1	Quantification of cancer driver mutations in human breast and lung DNA using targeted, error‐corrected CarcSeq 2020 ·Environmental and Molecular Mutagenesis ·Kelly L. Harris, Vijay Walia, Binsheng Gong, (unknown), Meagan B. Myers, Joshua Xu, Barbara L. Parsons	9
2	The C7orf43/TRAPPC14 component links the TRAPPII complex to Rabin8 for preciliary vesicle tethering at the mother centriole during ciliogenesis 2019 ·Journal of Biological Chemistry ·(unknown), Christine Insinna, Huijie Zhao, Peter John, (unknown), Quanlong Lü, Vijay Walia, Suzanne I. Specht, Selvambigai Manivannan, Jimmy K. Stauffer, Andrew A. Peden, Christopher J. Westlake	17
3	Akt Regulates a Rab11-Effector Switch Required for Ciliogenesis 2019 ·Developmental Cell ·Vijay Walia, (unknown), Melanie Vetter, Christine Insinna, Sumeth Perera, Quanlong Lü, Daniel A. Ritt, Elizabeth M. Semler, Suzanne I. Specht, Jimmy K. Stauffer, Deborah K. Morrison, Esben Lorentzen, Christopher J. Westlake	46
4	Analysis of Enzymatic Activity of Matrix Metalloproteinase (MMP) by Collagen Zymography in Melanoma 2018 ·Methods in molecular biology ·Vijay Walia, Yardena Samuels	4
5	Oncogenic RAS-Induced Perinuclear Signaling Complexes Requiring KSR1 Regulate Signal Transmission to Downstream Targets 2017 ·Cancer Research ·Sandip K. Basu, Sook Lee, Jacqueline Salotti, (unknown), Krisada Sakchaisri, Zhen Xiao, Vijay Walia, Christopher J. Westlake, Deborah K. Morrison, Peter F. Johnson	16
6	CLCA2 Interactor EVA1 Is Required for Mammary Epithelial Cell Differentiation 2016 ·PLoS ONE ·(unknown), Yufang Yin, Yang Yu, Vijay Walia, Randolph C. Elble	29
7	Early steps in primary cilium assembly require EHD1/EHD3-dependent ciliary vesicle formation 2015 ·Nature Cell Biology ·Quanlong Lü, Christine Insinna, Carolyn M. Ott, Jimmy K. Stauffer, Petra Pintado, Juliati Rahajeng, Ulrich Baxa, Vijay Walia, (unknown), (unknown), Ira Daar, Susana S. Lopes, Jennifer Lippincott‐Schwartz, Peter K. Jackson, Steve Caplan, Christopher J. Westlake	231
8	Mutational and Functional Analysis of the Tumor-Suppressor PTPRD in Human Melanoma 2014 ·Human Mutation ·Vijay Walia, Todd D. Prickett, Jung-Sik Kim, Jared J. Gartner, Jimmy Lin, Ming Zhou, Steven A. Rosenberg, Randolph C. Elble, David A. Solomon, Todd Waldman, Yardena Samuels	25
9	Loss of CLCA4 Promotes Epithelial-to-Mesenchymal Transition in Breast Cancer Cells 2013 ·PLoS ONE ·Yang Yu, Vijay Walia, Randolph C. Elble	53
10	Delving into somatic variation in sporadic melanoma 2012 ·Pigment Cell & Melanoma Research ·Vijay Walia, Euphemia W. Mu, Jimmy Lin, Yardena Samuels	28
11	Exon capture analysis of G protein-coupled receptors identifies activating mutations in GRM3 in melanoma 2011 ·Nature Genetics ·Todd D. Prickett, Xiaomu Wei, (unknown), Jamie K. Teer, Jimmy Lin, Vijay Walia, Jared J. Gartner, Jiji Jiang, Praveen F. Cherukuri, Alfredo A. Molinolo, Michael A. Davies, Jeffrey E. Gershenwald, Katherine Stemke‐Hale, Steven A. Rosenberg, Elliott H. Margulies, Yardena Samuels	115
12	Exome sequencing identifies GRIN2A as frequently mutated in melanoma 2011 ·Nature Genetics ·(unknown), Xiaomu Wei, Vijay Walia, Jimmy Lin, Jamie K. Teer, Todd D. Prickett, Jared J. Gartner, Sean Davis, Katherine Stemke‐Hale, Michael A. Davies, Jeffrey E. Gershenwald, William A. Robinson, Steven E. Robinson, Steven A. Rosenberg, Yardena Samuels	347
13	Loss of breast epithelial marker hCLCA2 promotes epithelial-to-mesenchymal transition and indicates higher risk of metastasis 2011 ·Oncogene ·Vijay Walia, Yiping Yu, Dong‐Yuan Cao, Miao Sun, Janel R. McLean, Brett G. Hollier, Jonathan Cheng, Sendurai A. Mani, (unknown), Louis S. Premkumar, Randolph C. Elble	3
14	Enrichment for Breast Cancer Cells with Stem/Progenitor Properties by Differential Adhesion 2010 ·Stem Cells and Development ·Vijay Walia, Randolph C. Elble	29
15	Micromanagement of the mitochondrial apoptotic pathway by p53 2010 ·Frontiers in bioscience ·Vijay Walia	18
16	hCLCA2 Is a p53-Inducible Inhibitor of Breast Cancer Cell Proliferation 2009 ·Cancer Research ·Vijay Walia, Ming Ding, Sumit Kumar, Daotai Nie, Louis S. Premkumar, Randolph C. Elble	53
17	p53 represses c-Myc through induction of the tumor suppressor miR-145 breakdown → 2009 ·Proceedings of the National Academy of Sciences ·Mohit Sachdeva, Shoumin Zhu, Fangting Wu, Hailong Wu, Vijay Walia, Sumit Kumar, Randolph C. Elble, Kounosuke Watabe, Yin‐Yuan Mo	691
18	The Putative Chloride Channel hCLCA2 Has a Single C-terminal Transmembrane Segment 2006 ·Journal of Biological Chemistry ·Randolph C. Elble, Vijay Walia, Hung-Chi Cheng, Che J. Connon, Lars Mundhenk, Achim D. Gruber, Bendicht U. Pauli	34

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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