Vannessa Davis

1.8k total citations · 1 hit paper
15 papers, 1.5k citations indexed

About

Vannessa Davis is a scholar working on Immunology and Allergy, Immunology and Molecular Biology. According to data from OpenAlex, Vannessa Davis has authored 15 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Immunology and Allergy, 8 papers in Immunology and 7 papers in Molecular Biology. Recurrent topics in Vannessa Davis's work include Cell Adhesion Molecules Research (9 papers), Angiogenesis and VEGF in Cancer (5 papers) and Atherosclerosis and Cardiovascular Diseases (4 papers). Vannessa Davis is often cited by papers focused on Cell Adhesion Molecules Research (9 papers), Angiogenesis and VEGF in Cancer (5 papers) and Atherosclerosis and Cardiovascular Diseases (4 papers). Vannessa Davis collaborates with scholars based in United States, Canada and Ukraine. Vannessa Davis's co-authors include David S. Milstone, Myron I. Cybulsky, Su-Ning Zhu, Mian Chen, Kaeko Iiyama, Hongmei Li, José-Carlos Gutierrez-Ramos, Motoi Iiyama, Philip W. Connelly and Peter O’Donnell and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and PLoS ONE.

In The Last Decade

Vannessa Davis

15 papers receiving 1.4k citations

Hit Papers

A major role for VCAM-1, but not ICAM-1, in early atheros... 2001 2026 2009 2017 2001 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Vannessa Davis United States 11 625 523 369 209 207 15 1.5k
Motoi Iiyama Japan 5 714 1.1× 459 0.9× 313 0.8× 247 1.2× 270 1.3× 6 1.5k
Su-Ning Zhu Canada 15 993 1.6× 601 1.1× 326 0.9× 339 1.6× 313 1.5× 18 2.0k
Kaeko Iiyama Japan 7 785 1.3× 521 1.0× 335 0.9× 297 1.4× 354 1.7× 9 1.9k
Mikko P.S. Ares Sweden 22 789 1.3× 511 1.0× 152 0.4× 408 2.0× 412 2.0× 32 1.8k
J. H. Qiao United States 11 695 1.1× 483 0.9× 129 0.3× 333 1.6× 425 2.1× 20 1.5k
Peter Libby United States 14 504 0.8× 395 0.8× 135 0.4× 214 1.0× 266 1.3× 22 1.7k
R Vlaicu Romania 14 828 1.3× 250 0.5× 173 0.5× 238 1.1× 179 0.9× 36 1.3k
Gerrity Rg United States 11 593 0.9× 369 0.7× 222 0.6× 242 1.2× 514 2.5× 16 1.5k
Yasushi Shikata Japan 20 295 0.5× 763 1.5× 170 0.5× 124 0.6× 124 0.6× 38 1.8k
Cody J. Diehl United States 14 872 1.4× 445 0.9× 81 0.2× 310 1.5× 207 1.0× 20 1.5k

Countries citing papers authored by Vannessa Davis

Since Specialization
Citations

This map shows the geographic impact of Vannessa Davis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Vannessa Davis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Vannessa Davis more than expected).

Fields of papers citing papers by Vannessa Davis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Vannessa Davis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Vannessa Davis. The network helps show where Vannessa Davis may publish in the future.

Co-authorship network of co-authors of Vannessa Davis

This figure shows the co-authorship network connecting the top 25 collaborators of Vannessa Davis. A scholar is included among the top collaborators of Vannessa Davis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Vannessa Davis. Vannessa Davis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
O’Donnell, Peter, et al.. (2016). Lipodystrophy, Diabetes and Normal Serum Insulin in PPARγ-Deficient Neonatal Mice. PLoS ONE. 11(8). e0160636–e0160636. 7 indexed citations
2.
Milstone, David S., Mian Chen, Peter O’Donnell, et al.. (2015). Differential Role of an NF-κB Transcriptional Response Element in Endothelial Versus Intimal Cell VCAM-1 Expression. Circulation Research. 117(2). 166–177. 37 indexed citations
3.
Parast, Mana M., et al.. (2009). PPARγ Regulates Trophoblast Proliferation and Promotes Labyrinthine Trilineage Differentiation. PLoS ONE. 4(11). e8055–e8055. 85 indexed citations
4.
Milstone, David S., Peter O’Donnell, Vannessa Davis, et al.. (2006). PPAR GAMMA IS EXPRESSED AND REGULATES PLACENTAL DEVELOPMENT AND TROPHOBLAST DIFFERENTIATION IN BOTH HUMANS AND MICE. The FASEB Journal. 20(5). 1 indexed citations
5.
Yu, Ying, Karen S. Moulton, Mohamed K. Khan, et al.. (2004). E-selectin is required for the antiangiogenic activity of endostatin. Proceedings of the National Academy of Sciences. 101(21). 8005–8010. 72 indexed citations
6.
Yu, Ying, Karen S. Moulton, Mohamed K. Khan, et al.. (2004). E-SELECTIN IS REQUIRED FOR THE ANTI-ANGIOGENIC ACTIVITY OF ENDOSTATIN. Cardiovascular Pathology. 13(3). 8–9. 4 indexed citations
7.
Cybulsky, Myron I., Kaeko Iiyama, Hongmei Li, et al.. (2001). A major role for VCAM-1, but not ICAM-1, in early atherosclerosis. Journal of Clinical Investigation. 107(10). 1255–1262. 957 indexed citations breakdown →
8.
Jain, Mohit, Chee Chew Lim, Kohzo Nagata, et al.. (2001). Targeted inactivation of Gαidoes not alter cardiac function or β-adrenergic sensitivity. American Journal of Physiology-Heart and Circulatory Physiology. 280(2). H569–H575. 28 indexed citations
9.
Milstone, David S., Raymond W. Redline, Peter O’Donnell, Vannessa Davis, & George Stavrakis. (2000). E-selectin expression and function in a unique placental trophoblast population at the fetal-maternal interface: Regulation by a trophoblast-restricted transcriptional mechanism conserved between humans and mice. Developmental Dynamics. 219(1). 63–76. 14 indexed citations
10.
Milstone, David S., Peter O’Donnell, George Stavrakis, Richard M. Mortensen, & Vannessa Davis. (2000). E-selectin Expression and Stimulation by Inflammatory Mediators are Developmentally Regulated during Embryogenesis. Laboratory Investigation. 80(6). 943–954. 16 indexed citations
11.
Milstone, David S., Dai Fukumura, Richard A. Padgett, et al.. (1998). Mice Lacking E-Selection Show Normal Numbers of Rolling Leukocytes but Reduced Leukocyte Stable Arrest on Cytokine-Activated Microvascular Endothelium. Microcirculation. 5(2). 153–171. 21 indexed citations
12.
Milstone, David S., Dai Fukumura, Richard C. Padgett, et al.. (1998). Mice Lacking E‐Selection Show Normal Numbers of Rolling Leukocytes but Reduced Leukocyte Stable Arrest on Cytokine‐Activated Microvascular Endothelium. Microcirculation. 5(2-3). 153–171. 27 indexed citations
13.
Milstone, David S., Dai Fukumura, Richard C. Padgett, et al.. (1998). Mice Lacking E-Selection Show Normal Numbers of Rolling Leukocytes but Reduced Leukocyte Stable Arrest on Cytokine-Activated Microvascular Endothelium. Microcirculation. 5(2-3). 153–171. 1 indexed citations
14.
Milstone, David S., Dai Fukumura, Richard C. Padgett, et al.. (1998). Mice lacking E-selectin show normal numbers of rolling leukocytes but reduced leukocyte stable arrest on cytokine-activated microvascular endothelium.. PubMed. 5(2-3). 153–71. 77 indexed citations
15.
Cybulsky, Myron I., Jochen W.U. Fries, Amy J. Williams, et al.. (1991). Alternative splicing of human VCAM-1 in activated vascular endothelium.. PubMed. 138(4). 815–20. 104 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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