J. H. Qiao
- Immunology top 5%
- Molecular Biology
- Surgery top 10%
- Epidemiology top 10%
- Endocrinology, Diabetes and Metabolism top 10%
- Co-authors
- Aldons J. LusisMichael C. FishbeinAlistair I. FyfeFeng LiaoThomas A. DrakeT B RajavashisthPeter LibbySteven K. Clinton
- Topics
- Atherosclerosis and Cardiovascular Diseases (6 papers)Lipoproteins and Cardiovascular Health (4 papers)Nuclear Receptors and Signaling (3 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Clinical InvestigationAnalytical Chemistry
- Partner nations
- United StatesChinaCzechia
In The Last Decade
J. H. Qiao
15 papers receiving 1.5k citations
Peers
Comparison fields: 5 of 84
- Immunology 695
- Molecular Biology 483
- Surgery 425
- Epidemiology 333
- Endocrinology, Diabetes and Metabolism 171
Countries citing papers authored by J. H. Qiao
This map shows the geographic impact of J. H. Qiao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. H. Qiao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. H. Qiao more than expected).
Fields of papers citing papers by J. H. Qiao
This network shows the impact of papers produced by J. H. Qiao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. H. Qiao. The network helps show where J. H. Qiao may publish in the future.
Co-authorship network of co-authors of J. H. Qiao
This figure shows the co-authorship network connecting the top 25 collaborators of J. H. Qiao. A scholar is included among the top collaborators of J. H. Qiao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. H. Qiao. J. H. Qiao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 0 | |
| 3 | 2 | |
| 4 | 0 | |
| 5 | 1 | |
| 6 | 0 | |
| 7 | 1 | |
| 8 | 0 | |
| 9 | 1 | |
| 10 | 104 | |
| 11 | 197 | |
| 12 | 148 | |
| 13 | Role of macrophage colony-stimulating factor in atherosclerosis: studies of osteopetrotic mice. | 257 |
| 14 | 59 | |
| 15 | 128 | |
| 16 | 198 | |
| 17 | 222 | |
| 18 | 15 | |
| 19 | 48 | |
| 20 | 111 |
About J. H. Qiao
J. H. Qiao is a scholar working on Immunology, Cancer Research and Bioengineering, having authored 20 papers that have together received 1.5k indexed citations. Recurring topics across this work include Atherosclerosis and Cardiovascular Diseases (6 papers), Lipoproteins and Cardiovascular Health (4 papers) and Nuclear Receptors and Signaling (3 papers). The work is most often cited by research in Immunology (695 citations), Immunology and Allergy (129 citations) and Biochemistry (98 citations). J. H. Qiao has collaborated with scholars based in United States, China and Czechia. Frequent co-authors include Aldons J. Lusis, Michael C. Fishbein, Alistair I. Fyfe, Feng Liao, Thomas A. Drake, T B Rajavashisth, Peter Libby, Steven K. Clinton, Lawrence W. Castellani and A.J. Lusis. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Analytical Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.