Valeria Melis
- Physiology top 10%
- Cellular and Molecular Neuroscience top 10%
- Pharmacology top 5%
- Cognitive Neuroscience
- Molecular Biology
- Co-authors
- Charles R. HarringtonClaude M. WischikDavid A. HorsleyGernot RiedelJohn M. D. StoreyMaría SchermaPaola FaddaThomas C. Baddeley
- Topics
- Alzheimer's disease research and treatments (13 papers)Neuroscience and Neuropharmacology Research (9 papers)Cholinesterase and Neurodegenerative Diseases (5 papers)
- Journals
- Journal of Biological ChemistryPLoS ONEJournal of Pharmacology and Experimental Therapeutics
- Partner nations
- United KingdomGermanyPoland
In The Last Decade
Valeria Melis
14 papers receiving 473 citations
Peers
Comparison fields: 5 of 54
- Physiology 257
- Cellular and Molecular Neuroscience 215
- Pharmacology 193
- Cognitive Neuroscience 83
- Molecular Biology 79
Countries citing papers authored by Valeria Melis
This map shows the geographic impact of Valeria Melis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Valeria Melis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Valeria Melis more than expected).
Fields of papers citing papers by Valeria Melis
This network shows the impact of papers produced by Valeria Melis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Valeria Melis. The network helps show where Valeria Melis may publish in the future.
Co-authorship network of co-authors of Valeria Melis
This figure shows the co-authorship network connecting the top 25 collaborators of Valeria Melis. A scholar is included among the top collaborators of Valeria Melis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Valeria Melis. Valeria Melis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 0 | |
| 3 | 3 | |
| 4 | 0 | |
| 5 | 2 | |
| 6 | 0 | |
| 7 | 5 | |
| 8 | 8 | |
| 9 | 9 | |
| 10 | 24 | |
| 11 | 28 | |
| 12 | 22 | |
| 13 | 72 | |
| 14 | 98 | |
| 15 | 110 | |
| 16 | 1 | |
| 17 | 95 |
About Valeria Melis
Valeria Melis is a scholar working on Cellular and Molecular Neuroscience, Physiology and Neurology, having authored 17 papers that have together received 478 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (13 papers), Neuroscience and Neuropharmacology Research (9 papers) and Cholinesterase and Neurodegenerative Diseases (5 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (215 citations), Pharmacology (193 citations) and Physiology (257 citations). Valeria Melis has collaborated with scholars based in United Kingdom, Germany and Poland. Frequent co-authors include Charles R. Harrington, Claude M. Wischik, David A. Horsley, Gernot Riedel, John M. D. Storey, María Scherma, Paola Fadda, Thomas C. Baddeley, Walter Fratta and Liana Fattore. Their work appears in journals such as Journal of Biological Chemistry, PLoS ONE and Journal of Pharmacology and Experimental Therapeutics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.