В. Л. Карпов
- Molecular Biology top 5%
- Cell Biology top 5%
- Epidemiology
- Genetics top 10%
- Immunology
- Co-authors
- O. V. PreobrazhenskayaA.D. MirzabekovА. В. МорозовHorst FeldmannI.R. VetterGertrud MannhauptKonstantin K. EbralidseElizaveta Starodubova
- Topics
- Ubiquitin and proteasome pathways (40 papers)Endoplasmic Reticulum Stress and Disease (22 papers)RNA and protein synthesis mechanisms (14 papers)
- Cited by
- Molecular BiologyAgingCell Biology
- Partner nations
- RussiaSwedenUnited Kingdom
In The Last Decade
В. Л. Карпов
152 papers receiving 2.1k citations
Peers
Comparison fields: 5 of 116
- Molecular Biology 1.6k
- Cell Biology 370
- Epidemiology 246
- Genetics 199
- Immunology 183
Countries citing papers authored by В. Л. Карпов
This map shows the geographic impact of В. Л. Карпов's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by В. Л. Карпов with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites В. Л. Карпов more than expected).
Fields of papers citing papers by В. Л. Карпов
This network shows the impact of papers produced by В. Л. Карпов. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by В. Л. Карпов. The network helps show where В. Л. Карпов may publish in the future.
Co-authorship network of co-authors of В. Л. Карпов
This figure shows the co-authorship network connecting the top 25 collaborators of В. Л. Карпов. A scholar is included among the top collaborators of В. Л. Карпов based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with В. Л. Карпов. В. Л. Карпов is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 0 | |
| 3 | 0 | |
| 4 | 4 | |
| 5 | 4 | |
| 6 | 1 | |
| 7 | 17 | |
| 8 | 8 | |
| 9 | 4 | |
| 10 | 3 | |
| 11 | 29 | |
| 12 | 10 | |
| 13 | [DNA vaccine encoding alpha-fetoprotein with fused ornithine decarboxylase degradation signal significantly suppresses hepatocellular carcinoma growth in mice]. | 5 |
| 14 | 2 | |
| 15 | 31 | |
| 16 | 5 | |
| 17 | 30 | |
| 18 | 2 | |
| 19 | 11 | |
| 20 | RADIATION MAKES BETTER WOODS AND COPOLYMERS | 2 |
About В. Л. Карпов
В. Л. Карпов is a scholar working on Virology, Molecular Biology and Cell Biology, having authored 160 papers that have together received 2.1k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (40 papers), Endoplasmic Reticulum Stress and Disease (22 papers) and RNA and protein synthesis mechanisms (14 papers). The work is most often cited by research in Molecular Biology (1.6k citations), Aging (41 citations) and Cell Biology (370 citations). В. Л. Карпов has collaborated with scholars based in Russia, Sweden and United Kingdom. Frequent co-authors include O. V. Preobrazhenskaya, A.D. Mirzabekov, А. В. Морозов, Horst Feldmann, I.R. Vetter, Gertrud Mannhaupt, Konstantin K. Ebralidse, Elizaveta Starodubova, Dmitry S. Karpov and Dmitry Guschin. Their work appears in journals such as Cell, Nucleic Acids Research and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.